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imm12045-sup-0001-FigureS1.pptapplication/PPT316KFigure S1. Morphology and expression of cell surface markers in macrophages differentiated with macrophage colony-stimulating factor and interferon-γ (Mγ-Mϕs) and macrophages differentiated with macrophage colony-stimulating factor only (M-Mϕs).
imm12045-sup-0002-FigureS2.pptapplication/PPT176KFigure S2. The tumour necrosis factor-α assay is not influenced by solvents.
imm12045-sup-0003-FigureS3.pptapplication/PPT245KFigure S3. The TGR5 agonist cannot inhibit tumour necrosis factor-α production from macrophages differentiated with macrophage colony-stimulating factor alone (M-Mϕs), dendritic cells (DCs) and macrophages differentiated with granulocyte–macrophage colony-stimulating factor (GM-Mϕs.
imm12045-sup-0004-FigureS4.pptapplication/PPT186KFigure S4. The TGR5 agonist cannot inhibit interleukin-10 production from macrophages differentiated with macrophage colony-stimulating factor and interferon-γ (Mγ-Mϕs).
imm12045-sup-0005-FigureS5.pptapplication/PPT302KFigure S5. Surface phenotypes on the macrophages differentiated with macrophage colony-stimulating factor and interferon-γ (Mγ-Mϕs) modified with or without the TGR5 agonist.
imm12045-sup-0006-FigureS6.pptapplication/PPT175KFigure S6. Tumour necrosis factor-α levels in peripheral CD14+ monocytes culture supernatants were analysed after 24 hr stimulation with lipopolysaccharide (LPS; 100 ng/ml) with or without the TGR5 agonist (1, 3·3 and 10 μm).
imm12045-sup-0007-FigureS7.pptapplication/PPT163KFigure S7. Inflamed Crohn's disease lamina propria mononuclear cells (CD LPMCs) had higher tumour necrosis factor-α level than non-inflamed.
imm12045-sup-0008-FigureS8.pptapplication/PPT278KFigure S8. Effects of the TGR5 agonist on tumour necrosis factor-α production from several subsets of Crohn's disease lamina propria mononuclear cells (CD LPMCs) stimulated with Enterococcus faecalis.

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