These authors contributed equally to this work.
An integrative approach to identify YB-1-interacting proteins required for cisplatin resistance in MCF7 and MDA-MB-231 breast cancer cells
Article first published online: 5 MAY 2011
© 2011 Japanese Cancer Association
Volume 102, Issue 7, pages 1410–1417, July 2011
How to Cite
Garand, C., Guay, D., Sereduk, C., Chow, D., Tsofack, S. P., Langlois, M., Perreault, È., Yin, H. H. and Lebel, M. (2011), An integrative approach to identify YB-1-interacting proteins required for cisplatin resistance in MCF7 and MDA-MB-231 breast cancer cells. Cancer Science, 102: 1410–1417. doi: 10.1111/j.1349-7006.2011.01948.x
- Issue published online: 15 JUN 2011
- Article first published online: 5 MAY 2011
- Accepted manuscript online: 5 APR 2011 08:18AM EST
- (Received October 29, 2010/Revised March 25, 2011/Accepted March 28, 2011/Accepted manuscript online April 5, 2011/Article first published online May 5, 2011)
Fig. S1. Cisplatin resistance of TAP and TAP-YB-1 clones.
Fig. S2. Depletion of PABPC1, MRPS7, and RB1CC1 with specific siRNA molecules in MCF7 cells.
Fig. S3. Response of transfected MCF7 cells to cisplatin.
Fig. S4. Response of transfected MDA-MB-231 cells to cisplatin.
Fig. S5. Examples of FACS cell cycle analysis of MDA-MB-231 cells transfected with an empty vector (left) and a YB-1 expression vector (right).
Table S1. List of proteins potentially interacting with YB-1, their chromosomal localization, and expression levels in breast cancer.
Table S2. Sequence of each siRNA used to establish dose–response curves.
Data S1.Materials and methods.
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