Funding sources This work was supported in part by grants to S.S. in the form of Grants-in-Aid for Scientific Research from JSPS (23300278, 23659126) and the Fuji Foundation for Protein Research (2010), the Iijima Memorial Foundation for the Promotion of Food Science and Technology (2011) and a High-Tech Research Centre project for Waseda University. A matching fund subsidy was received from MEXT, Japan.
Biological clock dysfunction exacerbates contact hypersensitivity in mice
Article first published online: 2 NOV 2012
© 2012 The Authors. BJD © 2012 British Association of Dermatologists
British Journal of Dermatology
Volume 168, Issue 1, pages 39–46, January 2013
How to Cite
Takita, E., Yokota, S., Tahara, Y., Hirao, A., Aoki, N., Nakamura, Y., Nakao, A. and Shibata, S. (2013), Biological clock dysfunction exacerbates contact hypersensitivity in mice. British Journal of Dermatology, 168: 39–46. doi: 10.1111/j.1365-2133.2012.11176.x
Conflicts of interest None declared.
E.T. and S.Y. contributed equally to this study.
- Issue published online: 21 DEC 2012
- Article first published online: 2 NOV 2012
- Accepted manuscript online: 26 JUL 2012 11:40PM EST
- Accepted for publication 20 July 2012
Figure S1. Differentiation, maturation and function of bone marrow-derived dendritic cells (BMDCs) were comparable between wild-type (WT) mice and CLOCK mutant mice.
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