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  • Allan, D., Limbrick, A.R., Thomas, P. & Westerman, M.P. (1982) Release of spectrin-free spicules on reoxygenation of sickled erythrocytes. Nature, 295, 612613.
  • Atichartakarn, V., Likittanasombat, K., Chuncharunee, S., Chandanamattha, P., Worapongpaiboon, S. & Aryurachai, K. (2003) Pulmonary arterial hypertension in previously splenectomized patients with 3-thalassemic disorders. International Journal of Hematology, 78, 139145.
  • Ballas, S.K. & Marcolina, M.J. (2006) Hyperhemolysis during the evolution of uncomplicated acute painful episodes in patients with sickle cell anemia. Transfusion, 46, 105110.
  • Bosman, G.J.C.G.M. (2004) Erythrocyte aging in sickle cell disease. Cellular and Molecular Biology, 50, 8186.
  • Bosman, G.J.C.G.M., Willekins, F.L.A. & Were, J.M. (2005) Erythrocyte aging: a more than superficial resemblance to apoptosis? Cellular and Physiology Biochemistry, 16, 18.
  • Camaschella, C. & Cappellini, M.D. (1995) Thalassemia intermedia. Haematologica, 80, 5868.
  • Cappellini, M.D., Robbiolo, L., Bottasso, B.M., Coppola, R., Fiorelli, G. & Mannucci, P.M. (2000) Venous thromboembolism and hypercoagulability in splenectomized with thalassaemia intermedia. British Journal of Haematology, 111, 467473.
  • Francis, Jr, R.B. & Hebbel, R.P. (1994) Hemostasis. In: Sickle Cell Disease: Basic Principles and Clinical Practice (ed. by S.H.Embury, R.P.Hebbel, N.Mohandas & M.H.Steinberg), pp. 299310. Lippincott-Raven, New York, NY.
  • Franck, P.F.H., Bevers, E.M., Lubin, B.H., Comfurius, P., Chiu, D.T.-Y., Op den Kamp, J.A.F., Zwaal, R.F.A., Van Deenan, L.L.M. & Roelofsen, B. (1985) Uncoupling of the membrane skeleton from the lipid bilayer. Journal of Clinical Investigation, 75, 183190.
  • Helley, D., Eldor, A., Girot, R., Ducrocq, R., Guilln, M. & Bezeaud, A. (1996) Increased procoagulant activity of red blood cells from patients with homozygous sickle cell disease and B-thalassemia. Thrombosis and Haemostasis, 76, 322327.
  • Hillery, C.A., Foster, T.D., Holzhauser, S.L., Scott, J.P., Panepinto, J.A., Mohandas, N., Mackman, N. & Wandersee, N.J. (2004) Tissue factor deficiency decreases sickle cell-induced vascular stasis in a hematopoietic stem cell transplant model of murine sickle cell disease (Abstract). Blood, 104, abstr no. 236.
  • Jackson, N., Franklin, I.M. & Hughes, M.A. (1986) Recurrent priapism following spleneectomy for thalassaemia intermedia. British Journal of Surgery, 73, 678.
  • Jeffers, A., Gladwin, M.T. & Kim-Shapiro, D.B. (2006) Computation of plasma hemoglobin nitric oxide scavenging in hemolytic anemias. Free Radical Biology and Medicine, 41, 15571565.
  • Kato, G.J., McGowan, V., Machado, R.F., Little, J.A., Taylor, VI, J., Morris, C.R., Nichols, J.S., Wang, X., Poljakovic, M., Morris, Jr, S.M. & Gladwin, M.T. (2006) Lactate dehyhrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease. Blood, 107, 22792285.
  • Knowles, D.W., Tilley, L., Mohandas, N. & Chasis, J.A. (1997) Erythrocyte membrane vesiculation: model for the molecular mechanism of protein sorting. Proceedings of the National Academy of Sciences of the United States of America, 94, 1296912974.
  • Moore, G.L., Ledford, M.E. & Merydith, A. (1981) A micromodification of the Drabkin hemoglobin assay for measuring plasma hemoglobin in the range of 5–2000 mg/dl. Biochemical Medicine, 26, 167173.
  • Naumann, H.N., Diggs, L.W., Barreras, L. & Williams, B.J. (1971) Plasma hemoglobin and hemoglobin fractions in sickle cell crisis. American Journal of Clinical Pathology, 56, 137147.
  • Nolan, V.G., Wyszynski, D.F., Farrer, L. & Steinberg, M.H. (2005) Hemolysis-associated priapism in sickle cell disease. Blood, 106, 32643267.
  • Pattanapanyasat, K., Noulsri, E., Fucharoen, S., Lerdwana, S., Lamchiagdhase, P., Siritanaratkul, N. & Webster, H.K. (2004) Flow cytometric quantitation of red blood cell vesicles in thalassemia. Cytometry Part B [Clinical Cytometry], 57B, 2331.
  • Reiter, C.D., Wang, X., Tanus-Santos, J.E., Hohh, N., Cannon, III, R.O., Schecter, A.N. & Gladwin, M.T. (2002) Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease. Nature Medicine, 8, 13831389.
  • Rodgers, G.P., Dover, G.J., Noguchi, C.T., Schecter, A.N. & Nienhuis, A.W. (1990) Hematological responses of sickle cell patients treated with hydroxyurea. New England Journal of Medicine, 322, 10371045.
  • Schrier, S.L., Rachmilewitz, E. & Mohandas, N. (1989) Cellular and membrane properties of alpha and beta thalassemic erythrocytes are different: implication for differences in clinical manifestations. Blood, 74, 21942202.
  • Setty, B.N.Y., Kulkarni, S., Rao, A.K. & Stuart, M.J. (2000) Fetal hemoglobin in sickle cell disease: relationship to erythrocyte phosphotidylserine exposure and coagulation. Blood, 96, 11191124.
  • Shet, A.S., Aras, O., Gupta, K., Hass, M.J., Rausch, D.J., Saba, N., Koopmeiners, L., Key, N.S. & Hebbel, R.P. (2003) Sickle blood contains tissue factor–positive microparticles derived from endothelial cells and monocytes. Blood, 102, 26782683.
  • Wagner, G.M., Chiu, D.T.-Y., Yee, M.C. & Lubin, B.H. (1986) Red cell vesiculation – a common membrane event. Journal of Laboratory and Clinical Medicine, 108, 315324.
  • Westerman, M.P., Cole, E.R. & Wu, K. (1984) The effect of spicules obtained from sickle red cells on clotting activity. British Journal of Haematology, 56, 557562.
  • Westerman, M.P., Pizzey, A., Wiener-Weil, Y., Ramotar, H., Lawrie, A., Purdy, G., Mackie, I., Hirschman, J. & Porter, J.B. (2002) Circulating red-cell derived vesicles expressing phosphatidylserine are increased in thalassemia as well as in sickle cell disorders (Abstract). Blood, 100, abstract no. 237a.
  • Willekens, F.L.A., Roerdinkholder-Stoelwinder, B., Groenen-Dopp, Y.A.M., Bos, H.J., Bosman, G.J.C.G.M., Van Den Bos, A.G., Verkleij, A.J. & Werre, J.M. (2003) Hemoglobin loss from erythrocytes in vivo results from spleen-facilitated vesiculation. Blood, 101, 747751.
  • Willekens, F.L.A., Were, J.M., Kruijt, K., Roerdinkholder-Stoelwinder, B., Groenen-Dopp, Y.A.M., Van Den Bos, A.G., Bosman, G.J.C.G.M. & Van Berkel, T.J.C. (2005) Liver Kupfer cells rapidly remove red blood cell-derived vesicles from the circulation by scavenger receptors. Blood, 105, 21412145.
  • Wood, B.L., Gibson, D.F. & Tait, J.F. (1996) Increased erythrocyte phosphatidylserine exposure in sickle cell disease: flow cytometric measurement and clinical associations. Blood, 88, 18731880.
  • Yasin, Z., Witting, S., Palascak, M.B., Joiner, C.H., Rucknagel, D.L. & Franco, R.S. (2003) Phosphatidylserine externalization in sickle red blood cells: associations with cell age, density, and hemoglobin F. Blood, 102, 365370.
  • Yuan, J., Kannan, R., Shinar, E., Rachmilewitz, E.A. & Low, P.S. (1992) Isolation, characterization, and immunoprecipitation studies of immune complexes from membranes of ß-thalassemic erythrocytes. Blood, 79, 30073013.