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Tim1 and Tim3 are not essential for experimental allergic asthma
Version of Record online: 7 APR 2011
© 2011 Blackwell Publishing Ltd
Clinical & Experimental Allergy
Volume 41, Issue 7, pages 1012–1021, July 2011
How to Cite
Barlow, J. L., Wong, S. H., Ballantyne, S. J., Jolin, H. E. and McKenzie, A. N. J. (2011), Tim1 and Tim3 are not essential for experimental allergic asthma. Clinical & Experimental Allergy, 41: 1012–1021. doi: 10.1111/j.1365-2222.2011.03728.x
- Issue online: 14 JUN 2011
- Version of Record online: 7 APR 2011
- Submitted 26 November 2010; revised 7 January 2011; accepted 28 January 2011.
Fig. S1. Inactivation of Havcr2 (Tim3) by homologous recombination (A) The structure of the Tim3 locus, the targeting vector, and predicted homologous recombination event. (B) Genotyping of genomic DNA from Tim3 gene-targeted mice by PCR analysis. (C) RT-PCR analysis of Tim3 expression, assessed using exon-specific primer sets in wild type and Tim3−/− Th1 and Th2 cells. Con, vector containing full length Tim3 transcript; Neg, negative control. (D) Flow cytometric analysis of Tim3 expression on lung cell suspensions from OVA sensitised wild type and Tim3−/− mice.
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