SEARCH

SEARCH BY CITATION

References

  • 1
    Lisbonne M, Leite-de-Moraes MC. Invariant Valpha14 NKT lymphocytes: a double-edged immuno-regulatory T cell population. Eur Cytokine Netw 2003; 14: 414.
  • 2
    Askenase PW. Delayed-type hypersensitivity and cellular immunity. Effector/regulatory molecules and mechanisms. In: MiddletonEJr, ed. Middleton's Allergy, Principles and Practice. St. Louis, MO: Mosby-Year Book, Inc., 2003: 42551.
  • 3
    Dieli F, Sireci G, Scire E, Salerno A, Bellavia A. Impaired contact hypersensitivity to trinitrochlorobenzene in interleukin-4-deficient mice. Immunology 1999; 98: 719.
  • 4
    Nagai H, Ueda Y, Ochi T, Hirano Y, Tanaka H, Inagaki N, Kawada K. Different role of IL-4 in the onset of hapten-induced contact hypersensitivity in BALB/c and C57BL/6 mice. Br J Pharmacol 2000; 129: 29906.
  • 5
    Weigmann B, Schwing J, Huber H, Ross R, Mossmann H, Knop J, Reske-Kunz AB. Diminished contact hypersensitivity response in IL-4 deficient mice at a late phase of the elicitation reaction. Scand J Immunol 1997; 45: 30814.
  • 6
    Xu H, DiIulio N, Fairchild R. T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (Il) 4/Il-10-producing (Th2) negative regulatory CD4+ T cells. J Exp Med 1996; 183: 100112.
  • 7
    Biedermann T, Mailhammer R, Mai A et al. Reversal of established delayed type hypersensitivity reactions following therapy with IL-4 or antigen-specific Th2 cells. Eur J Immunol 2001; 31: 158291.
  • 8
    Berg DJ, Leach MW, Kuhn R, Rajewsky K, Muller W, Davidson NJ, Rennick D. Interleukin 10 but not interleukin 4 is a natural suppressant of cutaneous inflammatory responses. J Exp Med 1995; 182: 99108.
  • 9
    Van Loveren H, Askenase PW. Delayed-type hypersensitivity is mediated by a sequence of two different T cell activities. J Immunol 1984; 133: 2397401.
  • 10
    Tsuji RF, Szczepanik M, Kawikova I et al. B cell-dependent T cell responses: IgM antibodies are required to elicit contact sensitivity. J Immunol 2002; 196: 127790.
  • 11
    Tsuji RF, Kikuchi M, Askenase PW. Possible involvement of C5/C5a in the efferent and elicitation phases of contact sensitivity. J Immunol 1996; 156: 444450.
  • 12
    Tsuji RF, Kawikova I, Ramabhadran R, Akahira-Azuma M, Taub D, Hugli TE, Gerard C, Askenase PW. Early local generation of C5a initiates the elicitation of contact sensitivity by leading to early T cell recruitment. J Immunol 2000; 165: 158898.
  • 13
    Geba GP, Ptak W, Anderson GM, Paliwal V, Ratzlaff RE, Levin J, Askenase PW. Delayed-type hypersensitivity in mast cell-deficient mice: dependence on platelets for expression of contact sensitivity. J Immunol 1996; 157: 55765.
  • 14
    McHale JF, Harari OA, Marshall D, Haskard DO. Vascular endothelial cell expression of ICAM-1 and VCAM-1 at the onset of eliciting contact hypersensitivity in mice: evidence for a dominant role of TNF-alpha. J Immunol 1999; 162: 164855.
  • 15
    Askenase PW, Bursztajn S, Gershon MD, Gershon RK. T cell-dependent mast cell degranulation and release of serotonin in murine delayed-type hypersensitivity. J Exp Med 1980; 152: 135874.
  • 16
    Askenase PW, Geba GP, Levin J, Ratzlaff RE, Anderson GM, Ushio H, Ptak W, Matsuda H. A role for platelet release of serotonin in the initiation of contact sensitivity. Int Arch Allergy Immunol 1995; 107: 1457.
  • 17
    Hwang JM, Yamanouchi J, Santamaria P, Kubes P. A critical temporal window for selectin-dependent CD4+ lymphocyte homing and initiation of late-phase inflammation in contact sensitivity. J Exp Med 2004; 199: 122334.
  • 18
    Ptak W, Herzog WR, Askenase PW. Delayed-type hypersensitivity initiation by early-acting cells that are antigen mismatched or MHC incompatible with late-acting, delayed-type hypersensitivity effector T cells. J Immunol 1991; 146: 46975.
  • 19
    Campos RA, Szczepanik M, Itakura A, Akahira-Azuma MSSMK, Askenase PW. Cutaneous immunization rapidly activates liver invariant Va14 NKT cells stimulating B-1 B cells to initiate T cell recruitment for elicitation of contact sensitivity. J Exp Med 2003; 198: 112.
  • 20
    Niewenhius E, Gilessen S, Scheper R, Exley M, Taniguchi M, Dranoff G, Blumberg R, Wilson S. CD1d and CD1d restricted iNKT cells play a pivotal role in contact hypersensitivity. Exp Dermatol 2005; 14: 2508.
  • 21
    Erickson LD, Foy TM, Waldschmidt TJ. Murine B1 B cells require IL-5 for optimal T cell-dependent activation. J Immunol 2001; 166: 15319.
  • 22
    Chen J, Trounstine M, Alt FW, Young F, Kurahara C, Loring JF, Huszar D. Immunoglobulin gene rearrangement in B cell deficient mice generated by targeted deletion of the JH locus. Int Immunol 1993; 5: 64756.
  • 23
    Morita M, Motoki K, Akimoto K et al. Structure-activity relationship of alpha-galactosylceramides against B16-bearing mice. J Med Chem 1995; 38: 217687.
  • 24
    Lisbonne M, Diem S, Castro-Keller A et al. Invariant Va14 NKT cells are required for allergen-induced airway inflammation and hyperreactivity in an experimental asthma model. J Immunol 2003; 171: 163741.
  • 25
    Takeda K, Kishimoto T, Akira S. STAT6: its role in interleukin 4-mediated biological functions. J Mol Med 1997; 75: 31726.
  • 26
    Yokozeki H, Ghoreishi M, Takagawa S et al. Signal transducer and activator of transcription 6 is essential in the induction of contact hypersensitivity. J Exp Med 2000; 191: 9951004.
  • 27
    Burdin N, Brossay L, Kronenberg M. Immunization with a-galactosylceramide polarizes CD1-reactive NKT cells towards Th2 cytokine synthesis. Eur J Immunol 1999; 29: 201425.
  • 28
    Redegeld FA, Nijkamp FP. Immunoglobulin free light chains and mast cells: pivotal role in T-cell-mediated immune reactions? Trends Immunol 2003; 4: 1815.
  • 29
    Brigl M, Bry L, Kent SC, Gumperz JE, Brenner MB. Mechanism of CD1d-restricted natural killer T cell activation during microbial infection. Nat Immunol 2003; 4: 12307.
  • 30
    Leite-de-Moraes MC, Hameg A, Arnould A, Machavoine F, Koezuka Y, Schneider E, Herbelin A, Dy M. A distinct IL-18-induced pathway to fully activate NK T lymphocytes independently from TCR engagement. J Immunol 1999; 163: 58716.
  • 31
    Yoshimoto T, Min B, Sugimoto T et al. Nonredundant roles for CD1d-restricted natural killer T cells and conventional CD4+ T cells in the induction of immunoglobulin E antibodies in response to interleukin 18 treatment of mice. J Exp Med 2003; 197: 9971005.
  • 32
    Leite-de-Moraes MC, Hameg A, Pacilio M et al. IL-18 enhances IL-4 production by ligand-activated NKT lymphocytes: a pro-Th2 effect of IL-18 exerted through NKT cells. J Immunol 2001; 166: 94551.
  • 33
    Berland R, Wortis HH. Origins and functions of B-1 cells with notes on the role of CD5. Annu Rev Immunol 2002; 20: 253300.
  • 34
    Prussin C, Metcalfe DD. IgE, mast cells, basophils, and eosinophils. J Allergy Clin Immunol 2003; 111: S48694.
  • 35
    Askenase PW, Van Loveren H, Kraeuter-Kops S et al. Defective elicitation of delayed-type hypersensitivity in W/Wv and SI/SId mast cell-deficient mice. J Immunol 1983; 131: 268794.
  • 36
    Biedermann T, Kneilling M, Mailhammer R et al. Mast cells control neutrophil recruitment during T cell-mediated delayed-type hypersensitivity reactions through tumor necrosis factor and macrophage inflammatory protein 2. J Exp Med 2000; 192: 144152.
  • 37
    Bryce PJ, Miller ML, Miyajima I, Tsai M, Galli SJ, Oettgen HC. Immune sensitization in the skin is enhanced by antigen-independent effects of IgE. Immunity 2004; 20: 38192.
  • 38
    Yokozeki H, Wu MH, Sumi K et al. Th2 cytokines, IgE and mast cells play a crucial role in the induction of para-phenylenediamine-induced contact hypersensitivity in mice. Clin Exp Immunol 2003; 132: 38592.
  • 39
    Orinska Z, Osiak A, Lohler J, Bulanova E, Budagian V, Horak I, Bulfone-Paus S. Novel B cell population producing functional IgG in the absence of membrane IgM expression. Eur J Immunol 2002; 32: 347280.
  • 40
    Varinou L, Ramsauer K, Karaghiosoff M, Kolbe T, Pfeffer K, Muller M, Decker T. Phosphorylation of the Stat1 transactivation domain is required for full-fledged IFN-gamma-dependent innate immunity. Immunity 2003; 19: 793802.
  • 41
    Reiner SL, Locksley RM. The regulation of immunity to Leishmania major. Annu Rev Immunol 1995; 13: 15177.
  • 42
    Balmer P, Devaney E. NK T cells are a source of early interleukin-4 following infection with third-stage larvae of the filarial nematode Brugia pahangi. Infect Immun 2002; 70: 22159.
  • 43
    Faveeuw C, Angeli V, Fontaine J et al. Antigen presentation by CD1d contributes to the amplification of Th2 responses to Schistosoma mansoni glycoconjugates in mice. J Immunol 2002; 169: 90612.
  • 44
    Ronet C, Darche S, Leite-de-Moraes MC, Miyake S, Yamamura T, Louis JA, Kasper LH, Buzoni-Gatel D. NKT cells are critical for the initiation of an inflammatory bowel response against Toxoplasma gondii. J Immunol 2005; 175: 899908.
  • 45
    Mencacci A, Del Sero G, Cenci E, D'Ostiani CF, Bacci A, Montagnoli C, Kopf M, Romani L. Endogenous interleukin 4 is required for development of protective CD4+ T helper type 1 cell responses to Candida albicans. J Exp Med 1998; 187: 30717.
  • 46
    Blackstock R, Murphy JW. Role of interleukin-4 in resistance to Cryptococcus neoformans infection. Am J Respir Cell Mol Biol 2004; 30: 10917.
  • 47
    Akbari O, Stock P, Meyer E et al. Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity. Nat Med 2003; 189: 55388.
  • 48
    Bilenki L, Yang J, Fan Y, Wang S, Yang X. Natural killer T cells contribute to airway eosinophilic inflammation induced by ragweed through enhanced IL-4 and eotaxin production. Eur J Immunol 2004; 34: 34554.
  • 49
    Zhou D, Mattner J, Cantu CR et al. Lysosomal glycosphingolipid recognition by NKT cells. Science 2004; 306: 17869.