Article first published online: 15 MAR 2011
© 2011 The Association for the Publication of the Journal of Internal Medicine
Journal of Internal Medicine
Volume 269, Issue 4, page 468, April 2011
How to Cite
(2011), Corrigendum. Journal of Internal Medicine, 269: 468. doi: 10.1111/j.1365-2796.2011.02366.x
- Issue published online: 15 MAR 2011
- Article first published online: 15 MAR 2011
Vol. 268, Issue 2, 194–205, Article first published online: 28 APR 2010
J. Persson, K. Lindberg, T. P. Gustafsson, P. Eriksson, G. Paulsson-Berne, P. Lundman. Low plasma adiponectin concentration is associated with myocardial infarction in young individuals. Journal of Internal Medicine 2010; 268: 194-205.
In the above article, the metric unit was incorrectly stated in the abstract and methods sections as mg/mL. The correct metric unit is mg/L. The affected sections are shown below with the corrected units of measurement in bold type.
Abstract Section, page 194
Results. Adiponectin levels were inversely associated with myocardial infarction [odds ratio (OR) 9.3, 95% confidence interval (CI) 4.7–18.2, for the lowest quartile compared to the highest quartile]. This persisted after adjustment for history of hypertension, HDL cholesterol, smoking and body mass index (BMI) (OR 3.1, 95% CI 1.3–7.6). The rs266729 polymorphism was associated with adiponectin levels. Plasma adiponectin concentrations were lower in individuals with the rare G/G genotype (median 4.3 mg/L, interquartile range [IQR] 2.8–6.2) compared to the C/G (median 5.8 mg/L, IQR 3.9–8.0; P = 0.035) and C/C genotypes (median 5.5 mg/L, IQR 4.0–7.5; P = 0.083).
Methods Section, page 195
Patient and control groups
The Stockholm coronary atherosclerosis risk factor (SCARF) study database and biobank was used for this study . A total of 387 survivors of a first MI were recruited together with 387 sex- and age-matched control subjects. All patients and their respective controls were below 60 years of age. They were interviewed, underwent a brief medical examination, and donated blood samples at 3 months after the index event. Details of recruitment and demographics of the groups have been published previously .Case–control pairs with available data on smoking and levels of glucose, creatinine, cholesterol, high-density lipoprotein (HDL) cholesterol, haemoglobin, proinsulin and adiponectin, and with C-reactive protein (CRP) ≤5.31 mg/L (10th percentile) were used in the study, resulting in 244 well-matched pairs (Figure 1). The study was approved by the Ethics Committee of Karolinska University Hospital, and all participants gave their informed consent.
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