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Figure S1. Quartiles of LDL_C by HCV RNA level during treatment at week 2, week 4 and week 12 timepoints.

Figure S2. LDL_C (</> 130mg/dL) by HCV RNA level during treatment at week 2, week 4 and week 12 timepoints.

Table S1. Measures of linkage disequilibrium between genome wide significant SNPs and rs12980275 by race.

Table S2. Multiple Regression model for baseline LDL in Caucasians

Table S3. Top GWAS association polymorphisms in association with total cholesterol in Caucasians

Table S4. Top GWAS association polymorphisms in association with serum HDL-cholesterol in Caucasians

Table S5. Top GWAS association polymorphisms in association with serum triglycerides in Caucasisans

Table S6. Multiple Regression model for LDL at 24 weeks post treatment for non-SVR patients

Table S7. Multiple Regression model for LDL at 24 weeks post treatment for patients who attained SVR

Table S8. Comparison between Treatment Response at the Timepoint LDL was Measured

Supplemetary

Table S9. Comparison between Genotypes within Treatment Response Levels at the Timepoint LDL was Measured

Table S10. Multiple Logistic Regression model for SVR

Table S11. Multiple logistic Regression model results testing interaction between LDL and IL28B genotype (rs12980275) for SVR.

Table S12. Univariate comparison of SVR rates by each IL28B genotype (rs12980275).

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