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Association study of 21 circadian genes with bipolar I disorder, schizoaffective disorder, and schizophrenia
Version of Record online: 14 OCT 2009
© 2009 The Authors. Journal compilation © 2009 John Wiley & Sons A/S
Volume 11, Issue 7, pages 701–710, November 2009
How to Cite
Mansour, H. A., Talkowski, M. E., Wood, J., Chowdari, K. V., McClain, L., Prasad, K., Montrose, D., Fagiolini, A., Friedman, E. S., Allen, M. H., Bowden, C. L., Calabrese, J., El-Mallakh, R. S., Escamilla, M., Faraone, S. V., Fossey, M. D., Gyulai, L., Loftis, J. M., Hauser, P., Ketter, T. A., Marangell, L. B., Miklowitz, D. J., Nierenberg, A. A., Patel, J., Sachs, G. S., Sklar, P., Smoller, J. W., Laird, N., Keshavan, M., Thase, M. E., Axelson, D., Birmaher, B., Lewis, D., Monk, T., Frank, E., Kupfer, D. J., Devlin, B. and Nimgaonkar, V. L. (2009), Association study of 21 circadian genes with bipolar I disorder, schizoaffective disorder, and schizophrenia. Bipolar Disorders, 11: 701–710. doi: 10.1111/j.1399-5618.2009.00756.x
- Issue online: 14 OCT 2009
- Version of Record online: 14 OCT 2009
- Received 21 December 2008, revised and accepted for publication 7 July 2009
Table S1. Gene-based association of circadian genes with bipolar I disorder (BPI).
Table S2. Gene-based association of circadian genes with schizophrenia (SZ)/schizoaffective disorder (SZA).
Table S3. Associations between single nucleotide polymorphisms and body mass index (BMI), age at onset (AAO), and presence or absence of psychosis among the bipolar I disorder (BPI) cases.
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Please note: Wiley Blackwell is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.