A.C. and G.S.-L. contributed equally to this work.
N-methyl-d-aspartate receptor- and metabotropic glutamate receptor-dependent long-term depression are differentially regulated by the ubiquitin-proteasome system
Version of Record online: 12 OCT 2009
© The Authors (2009). Journal Compilation © Federation of European Neuroscience Societies and Blackwell Publishing Ltd
European Journal of Neuroscience
Volume 30, Issue 8, pages 1443–1450, October 2009
How to Cite
Citri, A., Soler-Llavina, G., Bhattacharyya, S. and Malenka, R. C. (2009), N-methyl-d-aspartate receptor- and metabotropic glutamate receptor-dependent long-term depression are differentially regulated by the ubiquitin-proteasome system. European Journal of Neuroscience, 30: 1443–1450. doi: 10.1111/j.1460-9568.2009.06950.x
- Issue online: 19 OCT 2009
- Version of Record online: 12 OCT 2009
- Received 12 June 2009, revised 10 August 2009, accepted 21 August 2009
Fig. S1. Inhibition of proteasomal degradation by MG-132 differentially affects NMDA- and DHPG-induced AMPAR endocytosis.
Fig. S2. Basal surface levels of GluR1 are not affected by exposure to UBEI-41, lactacystin or MG-132.
Fig. S3. Basal synaptic transmission is not affected by exposure to lactacystin or UBEI-41.
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