Premature expression of KCC2 in embryonic mice perturbs neural development by an ion transport-independent mechanism
Version of Record online: 1 JUN 2010
© The Authors (2010). Journal Compilation © Federation of European Neuroscience Societies and Blackwell Publishing Ltd
European Journal of Neuroscience
Special Issue: On the occasion of the 7th FENS Forum, Amsterdam 2010
Volume 31, Issue 12, pages 2142–2155, June 2010
How to Cite
Horn, Z., Ringstedt, T., Blaesse, P., Kaila, K. and Herlenius, E. (2010), Premature expression of KCC2 in embryonic mice perturbs neural development by an ion transport-independent mechanism. European Journal of Neuroscience, 31: 2142–2155. doi: 10.1111/j.1460-9568.2010.07258.x
- Issue online: 21 JUN 2010
- Version of Record online: 1 JUN 2010
- Received 15 February 2010, revised 1 April 2010, accepted 2 April 2010
Fig. S1. Representative AP-2α and SOX-10 stainings corresponding to the neural crest scores.
Fig. S2. KCC2-C568A mice survive postnatally.
Fig. S3. Proliferating and apoptotic cells were not different in transgenic embryos.
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