Forward signaling by EphB1/EphB2 interacting with ephrin-B ligands at the optic chiasm is required to form the ipsilateral projection
Version of Record online: 21 NOV 2011
© 2011 The Authors. European Journal of Neuroscience © 2011 Federation of European Neuroscience Societies and Blackwell Publishing Ltd
European Journal of Neuroscience
Special Issue: Molecular Mechanisms of Neuronal Differentiation
Volume 34, Issue 10, pages 1620–1633, November 2011
How to Cite
Chenaux, G. and Henkemeyer, M. (2011), Forward signaling by EphB1/EphB2 interacting with ephrin-B ligands at the optic chiasm is required to form the ipsilateral projection. European Journal of Neuroscience, 34: 1620–1633. doi: 10.1111/j.1460-9568.2011.07845.x
- Issue online: 21 NOV 2011
- Version of Record online: 21 NOV 2011
- Received 12 April 2011, revised 18 July 2011, accepted 21 July 2011
Fig. S1. Detailed schematic of generation of EphB1T-lacZ mutant mouse with southern blot results and whole embryo X-gal staining at three stages of development: E10.5, E11.5, and E12.5 (scale bar = 2 mm). Expression of the EphB1-β-gal fusion protein increases in the developing nervous system from E10.5 to E12.5 (arrows).
Fig. S2. Comparison of the percentage of ipsilaterally projecting RGC axons of pigmented wild-type and EphB2lacZ/lacZ mutant mice.
Fig. S3. Comparison of the percentage of ipsilaterally projecting RGC axons of albino wild-type and ephrin-B3−/− mutant mice.
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