Opposite function of dopamine D1 and N-methyl-D-aspartate receptors in striatal cannabinoid-mediated signaling
Version of Record online: 31 OCT 2011
Published 2011. This article is a US Government work and is in the public domain in the USA
European Journal of Neuroscience
Volume 34, Issue 9, pages 1378–1389, November 2011
How to Cite
Daigle, T. L., Wetsel, W. C. and Caron, M. G. (2011), Opposite function of dopamine D1 and N-methyl-D-aspartate receptors in striatal cannabinoid-mediated signaling. European Journal of Neuroscience, 34: 1378–1389. doi: 10.1111/j.1460-9568.2011.07874.x
- Issue online: 31 OCT 2011
- Version of Record online: 31 OCT 2011
- Received 16 May 2011, revised 12 August 2011, accepted 18 August 2011
Fig. S1. Basal pERK1/2 levels are unaltered in several genetic mouse models. Basal levels of striatal pERK1/2 were compared from drug-naive wild-type (WT) littermates and either D2 receptor (D2R) KO (A), DAT KO (B), β-arrestin (βarr)-2 KO (C) or β-arrestin-1 KO (D) mice. Representative western blots and the corresponding densitometric analyses are shown. Data are mean + SEM; n = 5 mice for each treatment group. No significant differences were found between genotypes by unpaired t-test.
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