Address given upon receipt of Young Psychophysiologist Award. Sockny for Psychophysiological Research. Vancouver, British Columbia. Canada. October 1980.
Cardiovascular Responses to Effortful Active Coping: Implications for the Role of Stress in Hypertension Development
Version of Record online: 30 JAN 2007
Volume 18, Issue 3, pages 216–225, May 1981
How to Cite
Light, K. C. (1981), Cardiovascular Responses to Effortful Active Coping: Implications for the Role of Stress in Hypertension Development. Psychophysiology, 18: 216–225. doi: 10.1111/j.1469-8986.1981.tb03021.x
investiga-The author gratefully acknowledges the advice and assist- phar-ance of Paul Obrist. Janice Hastrup. Alberto Grignolo. Michael Pollak and John Koepke who collaborated with me on these or related studies. This research was supported by US-PHS grants 18976 and 23718 as well as postdoctoral fellowships 05531 and 05671 from the Heart. Lung and Blood Institute of the National Institutes of Health.
- Issue online: 30 JAN 2007
- Version of Record online: 30 JAN 2007
- Effortful active coping;
- Heart rate;
- Blood pressure;
- Beta-adrenergic mediation;
- Individual differences;
- Family history of hypertension
Recent contributions to the literature on stress and hypertension are discussed. The significance of effortful active coping in evoking sympathetically-mediated heart rate and blood pressure increases is supported by results of studies involving both aversive and appetitive task incentives. Young healthy males who are above-average in heart rate during coping tasks show consistently higher heart rates and systolic pressures during other stresses as well, but are indistinguishable from less reactive persons when relaxed. Studies involving beta-blockade indicate that these above-average cardiovascular increases are partly due to a greater beta-adrenergic response among the high heart rate reactors. In addition, the parents of these high heart rate reactors report a greater incidence of hypertension than parents of low reactors, suggesting that high cardiovascular responses during active coping stress may reflect a high degree of susceptibility to later hypertension.