Diurnal variation of uterine contractility

  1. V. LUNDSTRÖM1,
  2. P. ENEROTH1,
  3. M.-L. SWAHN1,
  4. E. GRANSTRÖM2

Article first published online: 23 AUG 2005

DOI: 10.1111/j.1471-0528.1984.tb05900.x

Issue

BJOG: An International Journal of Obstetrics & Gynaecology

BJOG: An International Journal of Obstetrics & Gynaecology

Volume 91, Issue 2, pages 155–159, February 1984

Additional Information

How to Cite

LUNDSTRÖM, V., ENEROTH, P., SWAHN, M.-L. and GRANSTRÖM, E. (1984), Diurnal variation of uterine contractility. BJOG: An International Journal of Obstetrics & Gynaecology, 91: 155–159. doi: 10.1111/j.1471-0528.1984.tb05900.x

Author Information

  1. 1

    Department of Obstetrics and Gynaecology Karolinska Hospital, S-104 01 Stockholm, Sweden

  2. 2

    Department of Physiological Chemistry, Karolinska Institute, S-104 01 Stockholm, Sweden

Publication History

  1. Issue published online: 23 AUG 2005
  2. Article first published online: 23 AUG 2005
  3. Received 28 March 1983; Accepted 15 July 1983

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summary

The diurnal uterine activity in four normal women in the secretory phase of their menstrual cycles and one woman suffering from dysmenorrhea were studied in relation to concomitant hormone levels in blood (progesterone, hGH, prolactin, cortisol, vasopressin, and 15-keto-13, 14-dihydro-PGF2α). In the four normal women uterine activity decreased after midnight, unrelated to circulating levels of 15-keto-13,14-dihydro-PGF2α. But during a dysmenorrheic episode the uterine hyper-contractility pattern correlated well with levels of the PGF2α-metabolite, indicating a role of endogenous-produced PGF2α in this condition. The results demonstrate a diurnal rhythm, possibly related to the wake-sleep cycle. No simple associations were seen between vasopressin, cortisol, prolactin, hGH, the PGF2α-metabolite, and uterine activity.

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