Anal sphincter tears: prospective study of obstetric risk factors

Authors

  • E. Samuelsson,

    Midwife
    1. Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, Göteborg
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  • L. Ladfors,

    Consultant (Obstetrics)
    1. Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, Göteborg
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  • U. B. Wennerholm,

    Consultant (Obstetrics)
    1. Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, Göteborg
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  • B. Gåreberg,

    Midwife
    1. Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, Göteborg
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  • K. Nyberg,

    Senior lecturer
    1. Department of Nursing, College of Health and Caring Sciences, University of Göteborg, Sweden
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  • H. Hagberg

    Professor (Obstetrics and Perinatology), Corresponding author
    1. Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, Göteborg
      Correspondence: Dr H. Hagberg, Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, 41685 Göteborg, Sweden.
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Correspondence: Dr H. Hagberg, Perinatal Center, Department of Obstetrics and Gynaecology, Sahlgrenska University Hospital/Östra, 41685 Göteborg, Sweden.

Abstract

Objective To evaluate intrapartum risk factors for anal sphincter tear.

Design A prospective observational study.

Setting Delivery unit at the University Hospital in Göteborg, Sweden.

Participants 2883 consecutive women delivered vaginally during the period between 1995 and 1997. Information was obtained from patient records and from especially designed protocols which were completed during and after childbirth.

Main outcome measures Anal sphincter (third and fourth degree) tear.

Results Anal sphincter tear occurred in 95 of 2883 women (3.3%). Univariate analysis demonstrated that the risk of anal sphincter tear was increased by nulliparity, high infant weight, lack of manual perineal protection, deficient visualisation of perineum, severe perineal oedema, long duration of delivery and especially protracted second phase and bear down, use of oxytocin, episiotomy, vacuum extraction and epidural anaesthesia. After analysis with stepwise logistic regression, reported as odds ratio, 95% confidence interval, the following factors remained independently associated with anal sphincter tear: slight perineal oedema (0.40, 0.26–0.64); manual perineal protection (0.49, 0.28–0.86); short duration of bear down (0.47, 0.24–0.91); no visualisation of perineum (2.77, 1.36–5.63); parity (0.59, 0.40–0.89); and high infant weight (2.02, 1.30–3.16). Analysis of variance showed that manual perineal protection had a stronger influence on lowering the frequency, and lack of visualisation of perineum and infant weight had a stronger influence on raising the frequency, of anal sphincter tears in nulliparous compared with parous women.

Conclusions Perineal oedema, poor ocular surveillance of perineum, deficient perineal protection during delivery, protracted final phase of the second stage, parity and high infant weight all constitute independent risk factors for anal sphincter tear. Such information is essential in order to reduce perineal trauma during childbirth.

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