SEARCH

SEARCH BY CITATION

The following article from Journal of Neurochemistry, “Diminished glutathione levels cause spontaneous and mitochondria-mediated cell death in neurons from trisomy 16 mice: a model of Down’s syndrome” by S. Schuchmann and U. Heinemann, published in Volume 74, Issue 3, 2000, pages 1205–1214 (available through www.onlinelibrary.wiley.com) has been retracted by agreement between the authors, journal Editors in Chief, Seán Murphy and Jörg Schulz, and Blackwell Publishing Ltd. Method images and data in the above paper are identical with the following articles “Altered Ca2+ signaling and mitochondrial deficiencies in hippocampal neurons of trisomy 16 mice: a model of Down’s syndrome” (Schuchmann S. and Heinemann U. J. Neurosci. 1998; 18: 7216–7231) and “Increased mitochondrial superoxide generation in neurons from trisomy 16 mice: A model of Down’s syndrome” (Schuchmann S. and Heinemann U. Free Radic. Biol. Med. 2000; 28: 235–250).

In detail: Method images from Fig. 1 and Fig. 2b from the article Schuchmann et al., J. Neurosci. (1998) were used for the Fig. 1a, b, e in Schuchmann & Heinemann, J. Neurochem. (2000) and Fig. 1a, b in Schuchmann & Heinemann, Free Radic. Biol. Med. (2000) without permission of The Journal of Neuroscience. Data from Fig. 3a, b, e, f from the article Schuchmann et al., J. Neurosci. (1998) were identical with Fig. 5a, b, d, f in Schuchmann & Heinemann, J. Neurochem. (2000) and Fig. 7a in Schuchmann & Heinemann, Free Radic. Biol. Med. (2000).

References

  1. Top of page
  2. References
  • Schuchmann S. and Heinemann U. (2000) Diminished glutathione levels cause spontaneous and mitochondria-mediated cell death in neurons from trisomy 16 mice: a model of Down’s syndrome. J. Neurochem. 74, 12051214.
  • Schuchmann S. and Heinemann U. (1998) Altered Ca2+ signaling and mitochondrial deficiencies in hippocampal neurons of trisomy 16 mice: a model of Down’s syndrome. J. Neurosci. 18, 72167231.
  • Schuchmann S. and Heinemann U. (2000) Increased mitochondrial superoxide generation in neurons from trisomy 16 mice: a model of Down’s syndrome. Free Radic. Biol. Med. 28, 235250.