Calpain-cleaved type 1 inositol 1,4,5-trisphosphate receptor impairs ER Ca2+ buffering and causes neurodegeneration in primary cortical neurons
Version of Record online: 14 AUG 2012
© 2012 The Authors. Journal of Neurochemistry © 2012 International Society for Neurochemistry
Journal of Neurochemistry
Volume 123, Issue 1, pages 147–158, October 2012
How to Cite
Kopil, C. M., Siebert, A. P., Kevin Foskett, J. and Neumar, R. W. (2012), Calpain-cleaved type 1 inositol 1,4,5-trisphosphate receptor impairs ER Ca2+ buffering and causes neurodegeneration in primary cortical neurons. Journal of Neurochemistry, 123: 147–158. doi: 10.1111/j.1471-4159.2012.07859.x
- Issue online: 10 SEP 2012
- Version of Record online: 14 AUG 2012
- Accepted manuscript online: 4 JUL 2012 02:40PM EST
- Received April 27, 2012; revised manuscript received June 21, 2012; accepted June 26, 2012.
Figure S1. Activation of ER stress markers and proteases in transduced primary cortical neurons.
Figure S2. Neurons expressing capn-InsP3R1 have increased rises in
[Ca2+]i in response to depolarization.
Figure S3. Characterization of glutamate injury model.
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