THIS ARTICLE HAS BEEN RETRACTED
Retracted: Ethanol disrupts axon outgrowth stimulated by netrin-1, GDNF, and L1 by blocking their convergent activation of Src family kinase signaling
Version of Record online: 28 SEP 2012
© VA Boston Healthcare Journal of Neurochemistry © 2012 International Society for Neurochemistry
Journal of Neurochemistry
Volume 123, Issue 4, pages 602–612, November 2012
How to Cite
J. Neurochem. (2012) 123, 602–612.
- Issue online: 12 OCT 2012
- Version of Record online: 28 SEP 2012
- Accepted manuscript online: 28 AUG 2012 02:45AM EST
- Manuscript Revised: 23 AUG 2012
- Manuscript Accepted: 23 AUG 2012
- Manuscript Received: 25 JUL 2012
- National Institute on Alcohol Abuse and Alcoholism. Grant Number: 2R01AA012974
- Medical Research Service, Department of Veterans Affairs
Vol. 132, Issue 6, 756, Version of Record online: 6 FEB 2015
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Table S1. Summary of the effects of PP2 and ethanol (EtOH) on axon elongation and axon initiation stimulated in CGNs by netrin-1, GDNF, L1 and BDNF.
Table S2. Summary of the effects of PP2 and ethanol (EtOH) on the activation of SFK, Cas, and ERK1/2 induced in CGNs by netrin-1, GDNF, L1 and BDNF.
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