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Fig. S1 CS-A and CS-B primary fibroblasts present increased levels of intracellular ROS as detected by EPR.

Fig. S2 Mitochondrial function declines in CS-A and CS-B cells.

Fig. S3 CS-A and CS-B primary fibroblasts have decreased mitochondrial function.

Fig. S4 CS-A and CS-B cells display higher steady-state levels of ATP.

Fig. S5 CS-A and CS-B primary fibroblasts exposed to a ROS-inducing agent present increased levels of intracellular ROS and decreased mitochondrial membrane potential.

Fig. S6 CS-A and CS-B cells are hypersensitive to the killing effects of H2O2.

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