The role of Ifng in alterations in liver gene expression in a mouse model of fulminant autoimmune hepatitis
Version of Record online: 20 APR 2009
© 2009 John Wiley & Sons A/S. NIH Publishing Agreement
Volume 29, Issue 9, pages 1307–1315, October 2009
How to Cite
Milks, M. W., Cripps, J. G., Lin, H., Wang, J., Robinson, R. T., Sargent, J. L., Whitfield, M. L. and Gorham, J. D. (2009), The role of Ifng in alterations in liver gene expression in a mouse model of fulminant autoimmune hepatitis. Liver International, 29: 1307–1315. doi: 10.1111/j.1478-3231.2009.02028.x
- Issue online: 3 SEP 2009
- Version of Record online: 20 APR 2009
- Received 9 December 2008Accepted 14 February 2009
Figure S1. Quantitative RT-PCR verification of microarray expression data. Selected genes were reanalyzed by quantitative real-time RT-PCR using an additional 4–6 eleven-day old mice per genotype. (A, B) Transcripts for (A) two up-regulated genes, encoding H2-Eα and CCL3, and (B) three down-regulated genes, encoding Keg, SerpinA6, and Ces, were measured by QRT-PCR. QRT-PCR data are normalized to a β-actin control QRT-PCR. Results from microarray and QRT-PCR are shown side-by-side. (C) β-actin expression levels from the microarrays are shown. (A, B, C) Units are arbitrary. The y-axes are in log10 format.
Table S1. Primer sequences used in Real-time RT-PCR.
Table S2. Gene ontology grouping of genes suppressed >10-fold.
|LIV_2028_sm_figS1.pdf||28K||Supporting info item|
|LIV_2028_sm_tables.pdf||105K||Supporting info item|
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