SEARCH

SEARCH BY CITATION

REFERENCES

  • 1
    Engel J Jr. Seizures and epilepsy. Philadelphia : F.A. Davis, 1989.
  • 2
    Schwarzkroin PA. Basic mechanisms of epileptogenesis. In: WyllieE, ed. The Treatment of epilepsy. Principles and practice. Philadelphia : Lea & Febiger, 1993:8398.
  • 3
    Dingledine R, McBain CJ, McNamara JO. Excitatory amino acid receptors in epilepsy. Trends Pharmacol Sci 1990; 11:3348.
  • 4
    Meldrum BS. Excitatory amino acid neurotransmission in epilepsy and anticonvulsant therapy. In: MeldrumBS, MoroniF, SimonRP, WoodsJH, eds. Excitatory amino acids. New York : Raven Press, 1991;65570.
  • 5
    Meldrum BS. Excitatory amino acids in epilepsy and potential novel therapies. Epilepsy Res 1992 12:189189.
  • 6
    SchwarczR, Ben-AriY, eds. Excitatory amino acids and epilepsy. Advances in experimental medicine and biology, vol. 203. New York : Plenum Press, 1986.
  • 7
    Albala BJ, Mosh SL, Okada R. Kainic acid-induced seizures: a developmental study. Dev Brain Res 1984;13:13948.
  • 8
    Ben-Ari Y, Tremblay E, Berger M, Nitecka L. Kainic acid seizure syndrome and binding sites in developing rats. Dev Brain Res 1984; 14:2848.
  • 9
    Velišková J, Velišek L, Mareš P. Epileptic phenomena produced by kainic acid in laboratory rats during ontogenesis. Physiol Bohemoslov 1988;37:395405.
  • 10
    Schoepp DD, Gamble AY, Salhoff CR, Johnson BG, Ornstein PL. Excitatory amino acid-induced convulsions in neonatal rats mediated by distinct receptor subtypes. Eur J Pharmacol 1990;182: 4217.
  • 11
    Mareš P, Velíšek L. N -Methyl-d-aspartate (NMDA)-induced seizures in developing rats. Dev Brain Res 1992;65:1859.
  • 12
    McDonald JW, Fix AS, Tizzano JP, Schoepp DD. Seizures and brain injury in neonatal rats induced by 1S,3R-ACPD, a metabotropic glutamate receptor agonist. J Neurosci 1993;13:444555.
  • 13
    Young RSK, Petroff OAC, Aquila WJ, Yates J. Effects of glutamate, quisqualate, and N-methyl-d-aspartate in neonatal brain. Exp Neurol 1991;111:3628.
  • 14
    Do KQ, Grandes P, Hansel C, et al. Sulphur-containing excitatory amino-acids: release, activity and localisation. Mol Neuropharmacol 1992;2:3942.
  • 15
    Do KQ, Mattenberger M, Streit P, CuCnod M. In vitro release of endogenous excitatory sulphur-containing amino acids from various rat brain regions. J Neurochem 1986;46:77986.
  • 16
    Klancnik JM, Cuenod M, Gähwiler BH, Jiang ZP, Do KQ. Release of endogenous amino acids, including homocysteic acid and cysteine sulphinic acid, from rat hippocampal slices evoked by electrical stimulation of Schaffer collateral-commissural fibres. Neuroscience 1992;49:55770.
  • 17
    Grieve A, Dunlop J, Schousboe A, Griffiths R. Kinetic characterization of sulphur-containing excitatory amino acid uptake in primary cultures of neurons and astrocytes. Neurochem Int 1991;19: 46774.
  • 18
    Curtis DR, Watkins JC. Acidic amino acids with strong excitatory actions on mammalian neurones. J Physiol (Lond) 1963;66:114.
  • 19
    Watkins JC, Evans RH. Excitatory amino acid transmitters. Annu Rev Pharmacol Toxicol 1981;21:165204.
  • 20
    Meweitt KN, Oakes DJ, Olvermann HJ, Smith DAS, Watkins JC. Pharmacology of the excitatory actions of sulphonic and sulphinic amino acids. In: MandelP, DeFeudisFV, eds. CNS receptors-form molecular pharmacology to behavior. New York : Raven Press, 1983;16374. (Advances in biochemical psychopharmacology; vol. 37.).
  • 21
    Turski WA. Homocysteic acid convulsant action of stereoisomers in mice. Brain Res 1989;479:3713.
  • 22
    Johnston GAR. Convulsions induced in 10–day-old rats by intra-peritoneal injection of monosodium glutamate and related excitant amino acids. Biochem Pharmacol 1973;22:13740.
  • 23
    McDonald JW, Johnston MV. Physiological and pathological roles of excitatory amino acids during central nervous system development. Brain Res Rev 1990;15:4170.
  • 24
    McDonald JW, Trescher WH, Johnston MV. Susceptibility of brain to AMPA induced excitotoxicity transiently peaks during early postnatal development. Brain Res 1992;583:5470.
  • 25
    Trescher WH, McDonald JW, Johnston MV. Quinolinateinduced injury is enhanced in developing rat brain. Develop Brain Res 1994;83:22432.
  • 26
    Sperber EF, Stanton PK, Haas K, Ackermann RF, MoshC SL. Developmental differences in the neurobiology of epileptic brain damage. Epilepsy Res 1992;suppl 9:6781.
  • 27
    Babb TL, Leite JP, Mathern GW, Pretorius JK. Kainic acid induced hippocampal seizures in rats: comparisons of acute and chronic seizures using intrahippocampal versus systemic injections. Ital J Neurol Sci 1995;16:3944.
  • 28
    Kubová H, Folbergrová J, Mareš P. Seizures induced by homocysteine in rats during ontogenesis. Epilepsia 1995;36:7506.
  • 29
    Finney DJ. Probit analysis. 2nd ed. Cambridge England : Cambridge University Press, 1952.
  • 30
    Saunders NR, Mollgard K. Development of the blood-brain barrier. J Dev Physiol 1984;6:4557.
  • 31
    Joo F. Current aspects or the development of the blood-brain barrier. Int J Dev Neurosci 1987;5:36972.
  • 32
    Folbergrová J. NMDA and not non-NMDA receptor antagonists are protective against seizures induced by homocysteine in neonatal rats. Exp Neurol 1994;130:34450.
  • 33
    Wuerthele SE, Yasuda RP, Freed WJ, Hoffer BJ. The effect of local application of homocysteine on neuronal activity in the central nervous system of the rat. Life Sci 1982;31;268391.
  • 34
    Cuénod M, Grandes P, Zängerle L, Streit P, Do KQ. Sulphur-containing excitatory amino acids in intercellular communication. Biochem Soc Trans 1993;21:727.
  • 35
    Bisaga A, Krzascik P, Jankowska E, Palejko W, Kostowski W, Danysz W. Effect of glutamate receptor antagonists on N-methyl-D-aspartate-and (S)-a-amino-3-hydroxy-5-methyl-4-isoxazole-proprionic acid-induced convulsant effects in mice and rats. Eur J Pharmacol 1993;242:21320.
  • 36
    Thurber SJ, Mikati MA, Stafstrom CE, Jensen FE, Holmes GL. Quisqualic acid-induced seizures during development: a behavioral and EEG study. Epilepsia 1994;35:86875.
  • 37
    Olney JW, Price MT, Salles KS, et al. El-Homocysteic acid: an endogenous excitotoxic ligand of the NMDA receptor. Brain Res Bull 1987;19:597602.
  • 38
    Pullan LM, Olney JW, Price MT, et al. Excitatory amino acid receptor potency and subclass specificity of sulphur-containing amino acids. J Neurochem 1987;49:13017.
  • 39
    Lehmann J, Tsai C, Wood PL. Homocysteic acid as a putative excitatory amino acid neurotransmitter: I. Postsynaptic characteristics at N-methyl-D-aspartate-type receptors on striatal cholinergic interneurons. J Neurochem 1988;51:176570.
  • 40
    Porter RHP, Roberts PJ. Glutamate metabotropic receptor activation in neonatal rat cerebral cortex by sulphur-containing excitatory amino acids. Neurosci Lett 1993;154:7880.
  • 41
    Nevander G, Ingvar M, Auer R, Siesjo BK. Irreversible neuronal damage after short periods of status epilepticus. Acta Physiol Scand 1984;120:1557.
  • 42
    Gruenthal M, Armstrong DR, Ault B, Nadler JV. Comparison of seizures and brain lesions produced by intracerebroventricular kainic acid and bicuculline methiodide. Exp Neurol 1986;93:62130.
  • 43
    Ingvar M, Morgan PF, Auer RN. The nature and timing of excitotoxic neuronal necrosis in the cerebral cortex, hippocampus and thalamus due to fluorothyl-induced status epilepticus. Acta Neuropathol (Berl) 1988;75:3629.
  • 44
    Wolf G, Keilhoff G. Kainate and glutamate neurotoxicity in dependence on the postnatal development with special reference to hippocampal neurons. Brain Res 1984; 14:1521.
  • 45
    Söderfeldt B, Kalimo H, Olsson Y, Siesjo BK. Bicuculline-induced epileptic brain injury. Transient and persistent cell changes in rat cerebral cortex in the early recovery period. Acta Neuropathol (Berl) 1983;62:8795.