Gelastic seizures and the anteromesial frontal lobe: A case report and review of intracranial EEG recording and electrocortical stimulation case studies
Version of Record online: 19 MAR 2010
Wiley Periodicals, Inc. © 2010 International League Against Epilepsy
Volume 51, Issue 10, pages 2195–2198, October 2010
How to Cite
Unnwongse, K., Wehner, T., Bingaman, W. and Foldvary-Schaefer, N. (2010), Gelastic seizures and the anteromesial frontal lobe: A case report and review of intracranial EEG recording and electrocortical stimulation case studies. Epilepsia, 51: 2195–2198. doi: 10.1111/j.1528-1167.2010.02548.x
- Issue online: 19 MAR 2010
- Version of Record online: 19 MAR 2010
- Accepted January 18, 2010; Early View publication March 19, 2010.
Figure S1. (A) 3T magnetic resonance imaging (MRI) demonstrated a subtle hyperintensity on fluid attenuated inversion recovery (FLAIR), extending from the anterior pole of the right lateral ventricle to the right prefrontal cortex, suggesting a transmantle cortical dysplasia. (B) Fluorodeoxyglucose positron emission tomography (FDG-PET) showed a corresponding area of subtle hypometabolism. (C) Subtraction ictal SPECT coregistered to MRI (SISCOM) revealed an area of hyperperfusion in the right anterior frontal region. Injection was 12 seconds after the clinical seizure onset.
Figure S2. Responses characteristic for the supplementary motor area such as negative motor phenomena and tonic contractions of the left hemibody or left extremities were acquired 2.5 cm posterior to the seizure onset.
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