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References

  • 1
    Li H, Du S, Yang L, et al. Rapid pulmonary fibrosis induced by acute lung injury via a lipopolysaccharide three-hit regimen. Innate Immun. 2009; 15: 14354.
  • 2
    Cesta MF, Ryman-Rasmussen JP, Wallace DG, et al. Bacterial lipopolysaccharide enhances PDGF signaling and pulmonary fibrosis in rats exposed to carbon nanotubes. Am J Respir Cell Mol Biol. 2010; 43: 14251.
  • 3
    Tian J, Wang Y, He Z, et al. Hydroxyethyl Starch (130 kD) Inhibits Toll-Like Receptor 4 Signaling Pathways in Rat Lungs Challenged with Lipopolysaccharide. Anesth Analg. 2011; 113: 1129.
  • 4
    He Z, Zhu Y, Jiang H. Inhibiting toll-like receptor 4 signaling ameliorates pulmonary fibrosis during acute lung injury induced by lipopolysaccharide: an experimental study. Respir Res. 2009; 10: 126.
  • 5
    Xiao-jun C, Min F, Liang C, et al. Expression and role of adiponectin receptor 1 in lipopolysaccharide-induced proliferation of cultured rat adventitial fibroblasts. Cell Biol Int. 2010; 34: 1639.
  • 6
    Cai XJ, Chen L, Li L, et al. Adiponectin inhibits lipopolysaccharide-induced adventitial fibroblast migration and transition to myofibroblasts via AdipoR1-AMPK-iNOS pathway. Mol Endocrinol. 2010; 24: 21828.
  • 7
    Gutierrez-Venegas G, Kawasaki-Cardenas P, Arroyo-Cruz SR, et al. Luteolin inhibits lipopolysaccharide actions on human gingival fibroblasts. Eur J Pharmacol. 2006; 541: 95105.
  • 8
    Wu WK, Law PT, Wong HP, et al. Shift of homeostasis from parenchymal regeneration to fibroblast proliferation induced by lipopolysaccharide-activated macrophages in gastric mucosal healing in vitro. Wound Repair Regen. 2007; 15: 2216.
  • 9
    Takeuchi R, Matsumoto H, Akimoto Y, et al. Reduction in lipopolysaccharide-induced apoptosis of fibroblasts obtained from a patient with gingival overgrowth during nifedipine-treatment. Arch Oral Biol. 2011; 56: 107380.
  • 10
    Susilowati H, Santoso AL, Barid I, et al. Rat periodontal fibroblast responses to bacterial lipopolysaccharide in vitro. J Microbiol Immunol Infect. 2002; 35: 2036.
  • 11
    Hagood JS, Prabhakaran P, Kumbla P, et al. Loss of fibroblast Thy-1 expression correlates with lung fibrogenesis. Am J Pathol. 2005; 167: 36579.
  • 12
    Sanders YY, Pardo A, Selman M, et al. Thy-1 promoter hypermethylation: a novel epigenetic pathogenic mechanism in pulmonary fibrosis. Am J Respir Cell Mol Biol. 2008; 39: 6108.
  • 13
    Ramirez G, Hagood JS, Sanders Y, et al. Absence of Thy-1 results in TGF-beta induced MMP-9 expression and confers a profibrotic phenotype to human lung fibroblasts. Lab Invest. 2011; 91: 120618.
  • 14
    Zhou Y, Hagood JS, Lu B, et al. Thy-1-integrin alphav beta5 interactions inhibit lung fibroblast contraction-induced latent transforming growth factor-beta1 activation and myofibroblast differentiation. J Biol Chem. 2010; 285: 2238293.
  • 15
    Sanders YY, Kumbla P, Hagood JS. Enhanced myofibroblastic differentiation and survival in Thy-1(-) lung fibroblasts. Am J Respir Cell Mol Biol. 2007; 36: 22635.
  • 16
    Sanders YY, Tollefsbol TO, Varisco BM, et al. Epigenetic regulation of thy-1 by histone deacetylase inhibitor in rat lung fibroblasts. Am J Respir Cell Mol Biol. 2011; 45: 1623.
  • 17
    Aung HT, Schroder K, Himes SR, et al. LPS regulates proinflammatory gene expression in macrophages by altering histone deacetylase expression. FASEB J. 2006; 20: 131527.
  • 18
    Angrisano T, Pero R, Peluso S, et al. LPS-induced IL-8 activation in human intestinal epithelial cells is accompanied by specific histone H3 acetylation and methylation changes. BMC Microbiol. 2010; 10: 172.
  • 19
    He ZY, Zhu YS, Jiang H. Toll-like receptor 4 mediates lipopolysaccharide-induced collagen secretion by phosphoinositide3-kinase-akt pathway in fibroblasts during acute lung injury. J Recept Signal Transduct. 2009; 29: 11925.
  • 20
    Chen B, Polunovsky V, White J, et al. Mesenchymal cells isolated after acute lung injury manifest an enhanced proliferative phenotype. J Clin Invest. 1992; 90: 177885.
  • 21
    Xia H, Diebold D, Nho R, et al. Pathological integrin signaling enhances proliferation of primary lung fibroblasts from patients with idiopathic pulmonary fibrosis. J Exp Med. 2008; 205: 165972.
  • 22
    Meijerink J, Mandigers C, van de Locht L, et al. A novel method to compensate for different amplification efficiencies between patient DNA samples in quantitative real-time PCR. J Mol Diagn. 2001; 3: 5561.
  • 23
    Berger SL. The complex language of chromatin regulation during transcription. Nature. 2007; 447: 40712.
  • 24
    Chakravortty D, Kumar KS. Induction of cell proliferation and collagen synthesis in human small intestinal lamina propria fibroblasts by lipopolysaccharide: possible involvement of nitric oxide. Biochem Biophys Res Commun. 1997; 240: 45863.
  • 25
    Subramoniam A, Padh H, Aleo JJ. Factors influencing calcium influx in endotoxin-challenged fibroblasts. Proc Soc Exp Biol Med. 1985; 178: 509.
  • 26
    Takemura A, Matsuda N, Kimura S, et al. Porphyromonas gingivalis lipopolysaccharide modulates the responsiveness of human periodontal ligament fibroblasts to platelet-derived growth factor. J Periodontal Res. 1998; 33: 4007.
  • 27
    Chakravortty D, Kumar KS. Interaction of lipopolysaccharide with human small intestinal lamina propria fibroblasts favors neutrophil migration and peripheral blood mononuclear cell adhesion by the production of proinflammatory mediators and adhesion molecules. Biochim Biophys Acta. 1999; 1453: 26172.
  • 28
    Layman DL, Diedrich DL. Growth inhibitory effects of endotoxins from Bacteroides gingivalis and intermedius on human gingival fibroblasts in vitro. J Periodontol. 1987; 58: 38792.
  • 29
    Zhang J, Wu L, Qu JM. Inhibited proliferation of human lung fibroblasts by LPS is through IL-6 and IL-8 release. Cytokine. 2011; 54: 28995.
  • 30
    Yang H, Kaneko M, He C, et al. Effect of a lipopolysaccharide from E. coli on the proliferation of fibroblasts and keratinocytes in vitro. Phytother Res. 2002; 16: 437.
  • 31
    Shan B, Hagood JS, Zhuo Y, et al. Thy-1 attenuates TNF-alpha-activated gene expression in mouse embryonic fibroblasts via Src family kinase. PLoS ONE. 2010; 5: e11662.
  • 32
    Zhou Y, Hagood JS, Murphy-Ullrich JE. Thy-1 expression regulates the ability of rat lung fibroblasts to activate transforming growth factor-beta in response to fibrogenic stimuli. Am J Pathol. 2004; 165: 65969.
  • 33
    Zhu H, Shan L, Schiller PW, et al. Histone deacetylase-3 activation promotes tumor necrosis factor-alpha (TNF-alpha) expression in cardiomyocytes during lipopolysaccharide stimulation. J Biol Chem. 2010; 285: 942936.
  • 34
    Ni YF, Wang J, Yan XL, et al. Histone deacetylase inhibitor, butyrate, attenuates lipopolysaccharide-induced acute lung injury in mice. Respir Res. 2010; 11: 33.
  • 35
    Choo QY, Ho PC, Tanaka Y, et al. Histone deacetylase inhibitors MS-275 and SAHA induced growth arrest and suppressed lipopolysaccharide-stimulated NF-kappaB p65 nuclear accumulation in human rheumatoid arthritis synovial fibroblastic E11 cells. Rheumatology. 2010; 49: 144760.
  • 36
    Roger T, Lugrin J, Le Roy D, et al. Histone deacetylase inhibitors impair innate immune responses to Toll-like receptor agonists and to infection. Blood. 2011; 117: 120517.
  • 37
    Brogdon JL, Xu Y, Szabo SJ, et al. Histone deacetylase activities are required for innate immune cell control of Th1 but not Th2 effector cell function. Blood. 2007; 109: 112330.
  • 38
    Suh HS, Choi S, Khattar P, et al. Histone deacetylase inhibitors suppress the expression of inflammatory and innate immune response genes in human microglia and astrocytes. J Neuroimmune Pharmacol. 2010; 5: 52132.