Regulation of the ischemia-induced autophagy–lysosome processes by nitrosative stress in endothelial cells
Article first published online: 11 MAR 2011
© 2011 John Wiley & Sons A/S
Journal of Pineal Research
Volume 51, Issue 1, pages 124–135, August 2011
How to Cite
Han, F., Chen, Y.-x., Lu, Y.-m., Huang, J.-y., Zhang, G.-s., Tao, R.-r., Ji, Y.-l., Liao, M.-h., Fukunaga, K. and Qin, Z.-h. (2011), Regulation of the ischemia-induced autophagy–lysosome processes by nitrosative stress in endothelial cells. Journal of Pineal Research, 51: 124–135. doi: 10.1111/j.1600-079X.2011.00869.x
- Issue published online: 13 JUL 2011
- Article first published online: 11 MAR 2011
- Accepted manuscript online: 1 FEB 2011 09:39AM EST
- Received December 31, 2010; accepted January 20, 2011.
Figure S1. A representative image showing autophagy activation by MDC staining with or without OGD treatment in bEnd.3 cells. Scale bar = 25 μm.
Figure S2. The co-localization of cathepsin B (red fluorescence) with vWF (green fluorescence) in microvessels of ME rats is shown in a series of XZ reconstructions. DAPI counterstaining indicates cell nuclei (blue). Scale bar = 100 μm.
Figure S3. Proteins were separated by SDS-PAGE and were analyzed by Western blotting using anti-Beclin 1, -LC3, -Lamp2 and -cathepsin B antibodies. A representative image showing temporal expression of the indicated proteins following ME. β-actin antibody was used to equalize the immunodetection signals for protein loading.
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