SEARCH

SEARCH BY CITATION

References

  • 1
    Racusen LC, Solez K, Colvin RB et al. The Banff 97 working classification of renal allograft pathology. Kidney Int 1999; 55: 713723.
  • 2
    Solez K, Racusen LC, Marcussen N et al. Morphology of ischemic acute renal failure, normal function, and cyclosporine toxicity in cyclosporine-treated renal allograft recipients. Kidney Int 1993; 43: 10581067.
  • 3
    Solez K, Axelsen RA, Benediktsson H et al. International standardization of criteria for the histologic diagnosis of renal allograft rejection: The Banff working classification of kidney transplant pathology. Kidney Int 1993; 44: 411422.
  • 4
    Einecke G, Fairhead T, Hidalgo LG et al. Tubulitis and Epithelial Cell Alterations in Mouse Kidney Transplant Rejection Are Independent of CD103, Perforin or Granzymes A/B. Am J Transplant 2006; 6: 21092120.
  • 5
    Jabs WJ, Sedlmeyer A, Ramassar V et al. Heterogeneity in the evolution and mechanisms of the lesions of kidney allograft rejection in mice. Am J Transplant 2003; 3: 15011509.
  • 6
    Halloran PF, Miller LW, Urmson J et al. IFN-gamma alters the pathology of graft rejection: Protection from early necrosis. J Immunol 2001; 166: 70727081.
  • 7
    Einecke G, Melk A, Ramassar V et al. Expression of CTL associated transcripts precedes the development of tubulitis in T-cell mediated kidney graft rejection. Am J Transplant 2005; 5: 18271836.
  • 8
    Goes N, Urmson J, Ramassar V, Halloran PF. Ischemic acute tubular necrosis induces an extensive local cytokine response: Evidence for induction of interferon-γ, transforming growth factorβ-1, granulocyte-macrophage colony-stimulating factor, interleukin-2 and interleukin-10. Transplant 1995; 59: 565572.
  • 9
    Gentleman R, Carey VJ, Huber W, Irizarry R, Dudoit S. Bioinformatics and Computational Biology Solutions Using R and Bioconductor. XII ed. 2005.
  • 10
    Smyth G. Linear models and empirical bayes methods for assessing differential expression in microarray experiments. Statistical Application in Genetics and Molecular Biology 21204; 3:Article 3.
  • 11
    Halloran PF, Urmson J, Ramassar V et al. Lesions of T-cell-mediated kidney allograft rejection in mice do not require perforin or granzymes A and B. Am J Transplant 2004; 4: 705712.
  • 12
    Einecke G, Mueller TF, Famulski KS, Ramassar V, Sis B, Halloran PF. Cytotoxic T cells, Interferon gamma and the renal response: Pathogenesis-based transcript sets have high diagnostic value in human kidney allograft rejection. Am J Transplant 2006; 6: 465472.
  • 13
    Adams AB, Williams MA, Jones TR et al. Heterologous immunity provides a potent barrier to transplantation tolerance. J Clin Invest 2003; 111: 18871895.
  • 14
    Sinha S, Heagerty AM, Shuttleworth CA, Kielty CM. Expression of latent TGF-beta binding proteins and association with TGF-beta 1 and fibrillin-1 following arterial injury. Cardiovasc Res 2002; 53: 971983.
  • 15
    Eickelberg O, Pansky A, Koehler E et al. Molecular mechanisms of TGF-(beta) antagonism by interferon (gamma) and cyclosporine A in lung fibroblasts. FASEB J 2001; 15: 797806.
  • 16
    Fairweather D, Frisancho-Kiss S, Yusung SA et al. Interferon-gamma protects against chronic viral myocarditis by reducing mast cell degranulation, fibrosis, and the profibrotic cytokines transforming growth factor-beta 1, interleukin-1 beta, and interleukin-4 in the heart. Am J Pathol 2004; 165: 18831894.
  • 17
    Robertson H, Ali S, McDonnell BJ, Burt AD, Kirby JA. Chronic renal allograft dysfunction: The role of T cell-mediated tubular epithelial to mesenchymal cell transition. J Am Soc Nephrol 2004; 15: 390397.
  • 18
    Du C, Jiang J, Guan Q et al. Renal tubular epithelial cell self-injury through Fas/Fas ligand interaction promotes renal allograft injury. Am J Transplant 2004; 4: 15831594.
  • 19
    Schnermann J. Homer W. Smith Award lecture. The juxtaglomerular apparatus: from anatomical peculiarity to physiological relevance. J Am Soc Nephrol 2003; 14: 16811694.