This article is accompanied by commentary, Abfraction, Abrasion, Biocorrosion, and the Enigma of Noncarious Cervical Lesions: A 20-Year Perspective, W. Stephan Eakle, DDS, DOI 10.1111/j.1708-8240.2011.00488.x
Abfraction, Abrasion, Biocorrosion, and the Enigma of Noncarious Cervical Lesions: A 20-Year Perspective
Version of Record online: 17 NOV 2011
© 2011 Wiley Periodicals, Inc.
Journal of Esthetic and Restorative Dentistry
Volume 24, Issue 1, pages 10–23, February 2012
How to Cite
GRIPPO, J. O., SIMRING, M. and COLEMAN, T. A. (2012), Abfraction, Abrasion, Biocorrosion, and the Enigma of Noncarious Cervical Lesions: A 20-Year Perspective. Journal of Esthetic and Restorative Dentistry, 24: 10–23. doi: 10.1111/j.1708-8240.2011.00487.x
- Issue online: 1 FEB 2012
- Version of Record online: 17 NOV 2011
Hitherto, noncarious cervical lesions (NCCLs) of teeth have been generally ascribed to either toothbrush—dentifrice abrasion or acid “erosion.”
The last two decades have provided a plethora of new studies concerning such lesions. The most significant studies are reviewed and integrated into a practical approach to the understanding and designation of these lesions. A paradigm shift is suggested regarding use of the term “biocorrosion” to supplant “erosion” as it continues to be misused in the United States and many other countries of the world. Biocorrosion embraces the chemical, biochemical, and electrochemical degradation of tooth substance caused by endogenous and exogenous acids, proteolytic agents, as well as the piezoelectric effects only on dentin. Abfraction, representing the microstructural loss of tooth substance in areas of stress concentration, should not be used to designate all NCCLs because these lesions are commonly multifactorial in origin. Appropriate designation of a particular NCCL depends upon the interplay of the specific combination of three major mechanisms: stress, friction, and biocorrosion, unique to that individual case. Modifying factors, such as saliva, tongue action, and tooth form, composition, microstructure, mobility, and positional prominence are elucidated.
By performing a comprehensive medical and dental history, using precise terms and concepts, and utilizing the Revised Schema of Pathodynamic Mechanisms, the dentist may successfully identify and treat the etiology of root surface lesions. Preventive measures may be instituted if the causative factors are detected and their modifying factors are considered.
(J Esthet Restor Dent 24:10–25, 2012)