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  • Abdallah, M., Abdel-Naser, M.B., Moussa, M.H., Assaf, C., and Orfanos, C.E. (2003). Sequential immunohistochemical study of depigmenting and repigmenting minigrafts in vitiligo. Eur. J. Dermatol. 13, 548552.
  • Agarwal, U.S., Gulati, R., Shukla, S.R., and Mathur, N. (2004). Satellite repigmentation after punch grafting. Int. J. Dermatol. 43, 273274.
  • Al’abadie, M.S., Senior, H.J., Bleehen, S.S., and Gawkrodger, D.J. (1994). Neuropeptide and neuronal marker studies in vitiligo. Br. J. Dermatol. 131, 160165.
  • al Badri, A.M., Foulis, A.K., Todd, P.M., Gariouch, J.J., Gudgeon, J.E., Stewart, D.G., Gracie, J.A., and Goudie, R.B. (1993). Abnormal expression of MHC class II and ICAM-1 by melanocytes in vitiligo. J. Pathol. 169, 203206.
  • Baker, B.S., Powles, A.V., Lambert, S., Valdimarsson, H., and Fry, L. (1988). A prospective study of the Koebner reaction and T lymphocytes in uninvolved psoriatic skin. Acta Derm. Venereol. 68, 430434.
  • Barona, M.I., Arrunátegui, A., Falabella, R., and Alzate, A. (1995). An epidemiologic case-control study in a population with vitiligo. J. Am. Acad. Dermatol. 33, 621625.
  • Basak, P.Y., Adiloglu, A.K., Ceyhan, A.M., Tas, T., and Akkaya, V.B. (2009). The role of helper and regulatory T cells in the pathogenesis of vitiligo. J. Am. Acad. Dermatol. 60, 256260.
  • Boersma, B.R., Westerhof, W., and Bos, J.D. (1995). Repigmentation in vitiligo vulgaris by autogous minigrafting: results in nineteen patients. J. Am. Acad. Dermatol. 33, 990995.
  • van den Boorn, J.G., Konijnenberg, D., Dellemijn, T.A., van der Veen, J.P., Bos, J.D., Melief, C.J., Vyth-Dreese, F.A., and Luiten, R.M. (2009). Autoimmune destruction of skin melanocytes by perilesional T cells from vitiligo patients. J. Invest. Dermatol. 129, 22202232.
  • Boyd, A.S., and Neldner, K.H. (1990). The isomorphic response of Koebner. Int. J. Dermatol. 29, 401410.
  • Brzoska, T., Luger, T.A., Maaser, C., Abels, C., and Böhm, M. (2008). Alpha-melanocyte-stimulating hormone and related tripeptides: biochemistry, antiinflammatory and protective effects in vitro and in vivo, and future perspectives for the treatment of immune-mediated inflammatory diseases. Endocr. Rev. 29, 581602.
  • Cario-André, M., Pain, C., Gauthier, Y., and Taieb, A. (2007). The melanocytorrhagic hypothesis of vitiligo tested on pigmented, stressed, reconstructed epidermis. Pigment Cell Res. 20, 385393.
  • Conrad, C., Meller, S., and Gilliet, M. (2009). Plasmacytoid dendritic cells in the skin: to sense or not to sense nucleic acids. Semin. Immunol. 21, 101109.
  • Eddy, D.D., Aschheim, E., and Farber, E.M. (1964). Experimental analysis of isomorphic (Koebner) response in psoriasis. Arch. Dermatol. 89, 579588.
  • Eyre, R.W., and Krueger, G.G. (1982). Response to injury of skin involved and uninvolved with psoriasis, and its relation to disease activity: Koebner and ‘reverse’ Koebner reactions. Br. J. Dermatol. 106, 153159.
  • Ezzedine, K., Gauthier, Y., Léauté-Labrèze, C., Marquez, S., Bouchtnei, S., Jouary, T., and Taieb, A. (2010). Segmental vitiligo associated with generalized vitiligo (mixed vitiligo): a retrospective case series of 19 patients. J. Am. Acad. Dermatol. doi: 10.1016/j.jaad.2010.08.031.
  • Falabella, R., Arrunategui, A., Barona, M.I., and Alzate, A. (1995). The minigrafting test for vitiligo: detection of stable lesions for melanocyte transplantation. J. Am. Acad. Dermatol. 32, 228232.
  • Garbelli, S., Mantovani, S., and Palermo Giachino, C. (2005). Melanocyte-specific, cytotoxic T cell responses in vitiligo: the effective variant of melanoma immunity? Pigment Cell Res. 18, 234242.
  • Gauthier, Y. (1995). The importance of Koebner’s phenomenon in the induction of vitiligo vulgaris lesions. Eur. J. Dermatol. 5, 704708.
  • Gauthier, Y., and Benzekri, L.. (2010). The koebner’s phenomenon. In Vitiligo, M., Picardo, and A., Taïeb, eds. (Berlin Heidelberg: Springer-Verlag), pp. 167173.
  • Gauthier, Y., Cario-Andre, M., Lepreux, S., Pain, C., and Taieb, A. (2003a). Melanocyte detachment after skin friction in non lesional skin of patients with generalized vitiligo. Br. J. Dermatol. 148, 95101.
  • Gauthier, Y., Cario Andre, M., and Taieb, A. (2003b). A critical appraisal of vitiligo etiologic theories. Is melanocyte loss a melanocytorrhagy? Pigment Cell Res. 16, 322332.
  • van Geel, N., Ongenae, K., De Mil, M., Haeghen, Y.V., Vervaet, C., and Naeyaert, J.M. (2004a). Double-blind placebo-controlled study of autologous transplanted epidermal cell suspensions for repigmenting vitiligo. Arch. Dermatol. 140, 12031208.
  • van Geel, N.A., Ongenae, K., Vander Haeghen, Y.M., and Naeyaert, J.M. (2004b). Autologous transplantation techniques for vitiligo: how to evaluate treatment outcome. Eur. J. Dermatol. 14, 4651.
  • van Geel, N., Vandenhaute, S., Speeckaert, R., Brochez, L., Mollet, I., De Cooman, L., and Lambert, J. (2010a). Prognostic value and clinical significance of halo nevi regarding vitiligo. Br. J. Dermatol. doi: 10.1111/j.1365-2133.2010.10154.x.
  • van Geel, N., De Lille, S., Vandenhaute, S., Gauthier, Y., Mollet, I., Brochez, L., and Lambert, J. (2010b). Different phenotypes of segmental vitiligo based on a clinical observational study. J. Eur. Acad. Dermatol. Venereol. doi: 10.1111/j.1468-3083.2010.03847.x
  • Gopinathan, T. (1965). A study of the lesion of vitiligo. Arch. Dermatol. 91, 397404.
  • Hann, S.K., Park, Y.K., and Chun, W.H. (1997). Clinical features of vitiligo. Clin. Dermatol. 15, 891897.
  • Jin, Y., Birlea, S.A., Fain, P.R. et al. (2010). Variant of TYR and autoimmunity susceptibility loci in generalized vitiligo. N. Engl. J. Med. 362, 16861697.
  • Kalayciyan, A., Aydemir, E.H., and Kotogyan, A. (2007). Experimental Koebner phenomenon in patients with psoriasis. Dermatology 215, 114117.
  • Kippenberger, S., Bernd, A., Loitsch, S., Muller, J., Guschel, M., and Kaufmann, R. (1999). Cyclic stretch up-regulates proliferation and heat shock protein 90 expression in human melanocytes. Pigment Cell Res. 12, 246251.
  • Köbner, H. (1877). Zur aetiologie der psoriasis. Vjschr Dermatol Syphil 8, 559561.
  • Kostyuk, V.A., Potpovich, A.I., Cesareo, E. et al. (2010). Dysfunction of glutathione s-transferase leads to excess 4-hydroxy-2-nonenal and H2O2 and impaired cytokine pattern in cultured keratinocytes and blood of vitiligo patients. Antioxid. Redox Signal. 13, 607620.
  • Kroll, T.M., Bommiasamy, H., Boissy, R.E., Hernandez, C., Nickoloff, B.J., Mestril, R., and Caroline Le Poole, I. (2005). 4-Tertiary butyl phenol exposure sensitizes human melanocytes to dendritic cell-mediated killing: relevance to vitiligo. J. Invest. Dermatol. 124, 798806.
  • Lande, R., Gregorio, J., Facchinetti, V. et al. (2007). Plasmacytoid dendritic cells sense self-DNA coupled with antimicrobial peptide. Nature 449, 564569.
  • Le Poole, I.C., Das, P.K., van den Wijngaard, R.M., Bos, J.D., and Westerhof, W. (1993). Review of the etiopathomechanism of vitiligo: a convergence theory. Exp. Dermatol. 2, 145153.
  • Lee, D.J., and Modlin, R.L. (2005). Breaking tolerance–another piece added to the vitiligo puzzle. J. Invest. Dermatol. 124, xiiixv.
  • Lee, A.Y., Kim, N.H., Choi, W.I., and Youm, Y.H. (2005). Less keratinocyte-derived factors related to more keratinocyte apoptosis in depigmented than normally pigmented suction-blistered epidermis may cause passive melanocyte death in vitiligo. J. Invest. Dermatol. 124, 976983.
  • Malakar, S., and Dhar, S. (1998). Spontaneous repigmentation of vitiligo patches distant from the autologous skin graft sites: a remote reverse Koebner’s phenomenon? Dermatology 197, 274.
  • Malakar, S., Lahiri, K., and Malakar, R.S. (2000). How unstable is the concept of stability in surgical repigmentation of vitiligo? Dermatology 201, 182183.
  • Mazereeuw-Hautier, J., Bezio, S., Mahe, E. et al. (2010). Segmental and nonsegmental childhood vitiligo has distinct clinical characteristics: a prospective observational study. J. Am. Acad. Dermatol. 62, 945949.
  • Melski, J.W., Bernhard, J.D., and Stern, R.S. (1983). The Koebner (isomorphic) response in psoriasis. Associations with early age at onset and multiple previous therapies. Arch. Dermatol. 119, 655659.
  • Miller, R.A. (1982a). The Koebner phenomenon. Int. J. Dermatol. 21, 192197.
  • Miller, R.A., and Griffiths, W.A. (1982b). Pressure inhibition of the Koebner reaction by capillary occlusion. Acta Derm. Venereol. 62, 331333.
  • Mottaz, J.H., Thorne, E.G., and Zelickson, A.S. (1971). Response of the epidermal melanocyte to minor trauma. Arch. Dermatol. 104, 611618.
  • Mulekar, S.V., Asaad, M., Ghwish, B., Al Issa, A., and Al Eisa, A. (2007). Koebner phenomenon in vitiligo: not always an indication of surgical failure. Arch. Dermatol. 143, 801802.
  • Njoo, M.D., Das, P.K., Bos, J.D., and Westerhof, W. (1999). Association of the Kobner phenomenon with disease activity and therapeutic responsiveness in vitiligo vulgaris. Arch. Dermatol. 135, 407413.
  • Noessner, E., Gastpar, R., Milani, V. et al. (2002). Tumor-derived heat shock protein 70 peptide complexes are cross-presented by human dendritic cells. J. Immunol. 169, 54245432.
  • Ongenae, K., van Geel, N., and Naeyaert, J.M. (2003). Evidence for an autoimmune pathogenesis of vitiligo. Pigment Cell Res. 16, 90100.
  • Pedace, F.J., Muller, S.A., and Winkelmann, R.K. (1969). The biology of psoriasis. An experimental study of the Koebner phenomenon. Acta Derm. Venereol. 49, 390400.
  • Powles, A.V., Baker, B.S., Rutman, A.J., Mcfadden, J.P., Valdimarsson, H., and Fry, L. (1990). Epidermal rupture is the initiating factor for the Koebner response in psoriasis. Acta Derm. Venereol. 70, 3538.
  • Rubin, A.I., and Stiller, M.J. (2002). A listing of skin conditions exhibiting the koebner and pseudo-koebner phenomena with eliciting stimuli. J. Cutan. Med. Surg. 6, 2934.
  • Ruiz-Maldonado, R., and Orozco-Covarrubias, M.L. (1997). Postinflammatory hypopigmentation and hyperpigmentation. Semin. Cutan. Med. Surg. 16, 1643.
  • Salem, M.M., Shalbaf, M., Gibbons, N.C., Chavan, B., Thornton, J.M., and Schallreuter, K.U. (2009). Enhanced DNA binding capacity on up-regulated epidermal wild-type p53 in vitiligo by H2O2-mediated oxidation: a possible repair mechanism for DNA damage. FASEB J. 23, 37903807.
  • Schallreuter, K.U., Moore, J., Wood, J.M. et al. (1999). In vivo and in vitro evidence for hydrogen peroxide (H2O2) accumulation in the epidermis of patients with vitiligo and its successful removal by a UVB-activated pseudocatalase. J. Invest. Dermatol. Symp. Proc. 4, 9196.
  • Schallreuter, K.U., Hasse, S., Rokos, H., Chavan, B., Shalbaf, M., Spencer, J.D., and Wood, J.M. (2009). Cholesterol regulates melanogenesis in human epidermal melanocytes and melanoma cells. Exp. Dermatol. 18, 680688.
  • Slominski, A., Wortsman, J., Luger, T., Paus, R., and Solomon, S. (2000). Corticotropin releasing hormone and proopiomelanocortin involvement in the cutaneous response to stress. Physiol. Rev. 80, 9791020.
  • Spencer, J.D., Gibbons, N.C., Rokos, H., Peters, E.M., Wood, J.M., and Schallreuter, K.U. (2007). Oxidative stress via hydrogen peroxide affects proopiomelanocortin peptides directly in the epidermis of patients with vitiligo. J. Invest. Dermatol. 127, 411420.
  • Steitz, J., Wenzel, J., Gaffal, E., and Tüting, T. (2004). Initiation and regulation of CD8 +  T cells recognizing melanocytic antigens in the epidermis: implications for the pathophysiology of vitiligo. Eur. J. Cell Biol. 83, 797803.
  • Taïeb, A., Picardo, M.; VETF Members. (2007). The definition and assessment of vitiligo: a consensus report of the Vitiligo European Task Force. Pigment Cell Res. 20, 2735.
  • Ueki, H. (2005). Koebner phenomenon in lupus erythematosus with special consideration of clinical findings. Autoimmun. Rev. 4, 219223.
  • Weiss, G., Shemer, A., and Trau, H. (2002). The Koebner phenomenon: review of the literature. J. Eur. Acad. Dermatol. Venereol. 16, 241248.
  • Westerhof, W., and d’Ischia, M. (2007). Vitiligo puzzle: the pieces fall in place. Pigment Cell Res. 20, 345359.
  • Westerhof, W., Manini, P., Napolitano, A., and d’Ischia, M. (2011). The haptenation theory of vitiligo: a close-up. Exp. Dermatol. 20, 9296.