DVM, PhD, Department of Veterinary Physiology and Pharmacology, College of Veterinary Medicine, Texas A&M University, College Station, TX 77843.
The Role of Vascular Mechanisms in the Development of Acute Equine Laminitis
Version of Record online: 5 FEB 2008
Journal of Veterinary Internal Medicine
Volume 7, Issue 4, pages 228–234, July 1993
How to Cite
Hood, D. M., Grosenbaugh, D. A., Mostafa, M. B., Morgan, S. J. and Thomas, B. C. (1993), The Role of Vascular Mechanisms in the Development of Acute Equine Laminitis. Journal of Veterinary Internal Medicine, 7: 228–234. doi: 10.1111/j.1939-1676.1993.tb01012.x
- Issue online: 5 FEB 2008
- Version of Record online: 5 FEB 2008
- January 27, 1992.
ACUTE LAMINITIS has long been attributed to factors or events that precede the onset of laminitis. Between 1759 and 1907 the overconsumption of grain, inflammation of the feet, suppression of perspiration (anhydrosis), excessive rest, excessive bleeding, road concussion, poor shoeing, unilateral weight bearing, sudden environmental temperature changes, prolonged standing (in the cold and aboard ships), diarrhea, and postpartum complications were all designated as causes. Today, commonly listed etiologic factors include ingestion of large amounts of grain, cold water, lush grass, or black walnut shavings, repeated concussion, endometritis or other severe infections, colic, exhaustion, stress, drug toxicities, and endocrine dysfunctions. At Texas A&M University (Table 1) the factors recorded as the cause presume a causal relationship between some preceding event and the acute laminitis. Logically, any event that precedes laminitis might be a cause, but etiologic validity depends on the definition of “cause” and the role that coincidence might have in the appearance of the disease.