Postanesthetic Cerebellar Dysfunction in Cats
Article first published online: 28 JUN 2008
© 2004 American College of Veterinary Internal Medicine
Journal of Veterinary Internal Medicine
Volume 18, Issue 3, pages 368–369, May 2004
How to Cite
Shamir, M., Goelman, G. and Chai, O. (2004), Postanesthetic Cerebellar Dysfunction in Cats. Journal of Veterinary Internal Medicine, 18: 368–369. doi: 10.1111/j.1939-1676.2004.tb02562.x
- Issue published online: 28 JUN 2008
- Article first published online: 28 JUN 2008
- Revised June 17 and September 19, 2003; Accepted December 15, 2003.
Eleven cats with signs of cerebellar dysfunction, developed on recovery from a brief and uneventful general anesthesia, were examined at the Koret School of Veterinary Medicine Teaching Hospital (KSVMTH) between 1998 and 2002. Neurological signs included mild to severe ataxia of all 4 limbs, intentional tremor, lack of menace response, and delayed hopping. The cats were of different ages when anesthetized and none had shown any prior signs of neural disease. They were examined 1 day to 4 years after onset of clinical signs, and the neurological deficits remained unchanged in a follow-up period of 6 months to 8 years. Medical and anesthetic records showed that all were Persian cross cats, 7 of them originating in the same city in Israel. Ketamine was the only anesthetic drug that had been used with all cats. It might be that a genetic component predisposes Persian cross cats to nonreversible cerebellar damage after exposure to an anesthetic dose of ketamine.