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This article corrects:

  1. Endoscopic, Biopsy, and Histopathologic Guidelines for the Evaluation of Gastrointestinal Inflammation in Companion Animals Volume 24, Issue 1, 10–26, Article first published online: 4 January 2010

The below article,1 published in the January/February 2010 issue of the Journal of Veterinary Internal Medicine, contained a typographical error on page 18, in the second paragraph under “Distinguishing Helicobacter- Associated Gastritis from Gastritis Associated with IBD.” In conclusion (2), “causal” was misprinted as “casual.” The correct paragraph is printed below.

The association of Helicobacter spp. infection with chronic gastritis in dogs and cats has been the subject of considerable investigation and debate (summarized in Neiger and Simpson84). The 2000 ACVIM Consensus Statement85 on Helicobacter-associated gastritis concluded that (1) Helicobacter spp. are highly prevalent in healthy and sick dogs and cats, (2) a direct causal relationship among Helicobacter spp., gastritis, and clinical signs has not been firmly established, and (3) Helicobacter spp.-associated gastritis is variable in its severity and characterized by a lymphoplasmacytic infiltrate in the lamina propria, lymphoid follicular hyperplasia, and Helicobacter organisms colonized within gastric glands. The dual presence of biopsy-proven gastric inflammation accompanied by mucosal invasion with Helicobacter organisms, diagnosed by special stains (Warthin-Starry), PCR, or fluorescence in situ hybridization, may serve to differentiate Helicobacter gastritis from IBD involving the gastric mucosa.86 Because of the gastric carriage of Helicobacter spp. in both health and disease, the GI Standardization Group has concluded that it may not be possible to differentiate IBD-associated gastritis from Helicobacter-associated gastritis by histopathologic examination of HE-stained sections.

The authors apologize for this error.