Macroreentrant Atrial Tachycardia in Patients without Previous Atrial Surgery or Catheter Ablation: Clinical and Electrophysiological Characteristics of Scar-Related Left Atrial Anterior Wall Reentry
Article first published online: 21 DEC 2012
© 2012 Wiley Periodicals, Inc.
Journal of Cardiovascular Electrophysiology
Volume 24, Issue 4, pages 404–412, April 2013
How to Cite
FUKAMIZU, S., SAKURADA, H., HAYASHI, T., HOJO, R., KOMIYAMA, K., TANABE, Y., TEJIMA, T., NISHIZAKI, M., KOBAYASHI, Y. and HIRAOKA, M. (2013), Macroreentrant Atrial Tachycardia in Patients without Previous Atrial Surgery or Catheter Ablation: Clinical and Electrophysiological Characteristics of Scar-Related Left Atrial Anterior Wall Reentry. Journal of Cardiovascular Electrophysiology, 24: 404–412. doi: 10.1111/jce.12059
- Issue published online: 1 APR 2013
- Article first published online: 21 DEC 2012
- Accepted manuscript online: 21 NOV 2012 03:58PM EST
- Manuscript received 3 September 2012; Revised manuscript received 7 November 2012; Accepted for publication 9 November 2012.
- atrial arrhythmias;
- atrial tachycardia;
- catheter ablation;
- cardiac mapping;
- left atrial flutter;
Scar-Related Left Atrial Anterior Wall Reentry. Introduction: Macroreentrant atrial tachycardia (MRAT) has been described most frequently in patients with prior cardiac surgery. Left atrial tachycardia and flutter are common in patients who undergo atrial fibrillation ablation; however, few reports describe left atrial MRAT involving the regions of spontaneous scarring. Here, we describe left atrial MRAT in patients without prior cardiac surgery or catheter ablation (CA) and discuss the clinical and electrophysiological characteristics of tachycardia and outcome of CA.
Methods and Results: An electrophysiological study and CA were performed in 6 patients (3 men; age 76 ± 6 years) with MRAT originating from the left atrial anterior wall (LAAW). No patient had a history of cardiac surgery or CA in the left atrium. Spontaneous scars (areas with bipolar voltage ≤ 0.05 mV) were observed in all patients. The activation map showed a figure-eight circuit with loops around the mitral annulus (4 counterclockwise and 2 clockwise) and a low-voltage area with LAAW scarring. The mean tachycardia cycle length was 303 ± 49 milliseconds. The conduction velocity was significantly slower in the isthmus between the scar in the LAAW and the mitral annulus than in the lateral mitral annulus (0.17 ± 0.05 m/s vs 0.94 ± 0.35 m/s; P = 0.003). Successful ablation of the isthmus caused interruption of the tachycardia and rendered it noninducible in all patients.
Conclusion: Spontaneous LAAW scarring is an unusual cause of MRAT, showing activation patterns with a figure-eight configuration. Radiofrequency CA is a feasible and effective treatment in such cases. (J Cardiovasc Electrophysiol, Vol. 24, pp. 404-412, April 2013)