Percutaneous Stellate Ganglion Block Suppressing VT and VF in a Patient Refractory to VT Ablation

Authors

  • JUSTIN HAYASE M.D.,

    1. University of California San Diego, San Diego, California, USA
    2. Veterans Affairs San Diego Healthcare System, San Diego, California, USA
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  • JIGAR PATEL D.O.,

    1. University of California San Diego, San Diego, California, USA
    2. Veterans Affairs San Diego Healthcare System, San Diego, California, USA
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  • SANJIV M. NARAYAN M.D., Ph.D., F.A.C.C., F.H.R.S.,

    1. University of California San Diego, San Diego, California, USA
    2. Veterans Affairs San Diego Healthcare System, San Diego, California, USA
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  • DAVID E. KRUMMEN M.D., F.A.C.C., F.H.R.S.

    Corresponding author
    1. Veterans Affairs San Diego Healthcare System, San Diego, California, USA
    • University of California San Diego, San Diego, California, USA
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  • Dr. Narayan received educational support and/or honoraria from Medtronic, St. Jude Medical, Biotronik, and Boston Scientific; he holds equity in Topera. Dr. Krummen is supported by a grant from the American Heart Association 10BGIA3500045. Other authors: No disclosures.

Address for correspondence: David E. Krummen, M.D., 3350 La Jolla Village Drive, Cardiology Section 111A, San Diego, CA 92161, USA. Fax: +858-552-7490; E-mail: dkrummen@ucsd.edu

Percutaneous Stellate Ganglion Block Suppressing VT and VF

Introduction

Electrical storm is a condition characterized by multiple episodes of ventricular tachycardia (VT) or ventricular fibrillation (VF) in a short period of time.

Case Presentation

An 80-year-old male with a history of ischemic cardiomyopathy presented with multiple ICD shocks. As a last resort, he underwent percutaneous left, followed by right, stellate ganglion block under fluoroscopic guidance. Since his discharge, he has been managed with alternating, biweekly left and right stellate ganglion blocks, and he has received no ICD shocks.

Discussion

This case illustrates the potential of ongoing, temporary percutaneous stellate ganglion blockade in suppressing ventricular arrhythmogenesis.

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