Crosstalk between hydrogen sulfide and nitric oxide in endothelial cells
Version of Record online: 7 JUN 2013
© 2013 The Authors. Journal of Cellular and Molecular Medicine Published by Foundation for Cellular and Molecular Medicine/Blackwell Publishing Ltd
This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.
Journal of Cellular and Molecular Medicine
Volume 17, Issue 7, pages 879–888, July 2013
How to Cite
Altaany, Z., Yang, G. and Wang, R. (2013), Crosstalk between hydrogen sulfide and nitric oxide in endothelial cells. Journal of Cellular and Molecular Medicine, 17: 879–888. doi: 10.1111/jcmm.12077
- Issue online: 24 JUL 2013
- Version of Record online: 7 JUN 2013
- Manuscript Accepted: 12 APR 2013
- Manuscript Received: 21 NOV 2012
- Canadian Institutes of Health Research
|jcmm12077-sup-0001-FigS1.tif||image/tif||7915K||Figure S1 The effect of H2S on NO production in primarily cultured mouse aortic endothelial cells. (A) The effects of CSE siRNA knockdown and NaHS treatment on NO production detected by Griess assay, n = 3, *P < 0.05 versus control, #P < 0.05 versus NaHS-treated group. (B) The effects of NaHS and L-NAME treatment on NO production detected by diaminofluorescein diacetate–based probes (DAF-FM), scale bars: 200 μm.|
|jcmm12077-sup-0002-FigS2.tif||image/tif||7915K||Figure S2 The pro-proliferative effects of H2S and NO on primarily cultured mouse aortic endothelial cells. (A) The effects of CSE siRNA, eNOS siRNA and NaHS treatments on EC proliferation assessed using BrdU proliferation assay, n = 3, *P < 0.05 versus control, #P < 0.05 versus NaHS-treated group (B) The efficiency of CSE siRNA and eNOS siRNA in ECs detected by Western blot.|
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