Transient exposure to echinacoside is sufficient to activate Trk signaling and protect neuronal cells from rotenone
Article first published online: 26 DEC 2012
© 2012 International Society for Neurochemistry
Journal of Neurochemistry
Volume 124, Issue 4, pages 571–580, February 2013
How to Cite
- Issue published online: 28 JAN 2013
- Article first published online: 26 DEC 2012
- Accepted manuscript online: 28 NOV 2012 07:47PM EST
- Manuscript Accepted: 26 NOV 2012
- Manuscript Revised: 14 OCT 2012
- Manuscript Received: 30 AUG 2012
- China Postdoctoral Foundation. Grant Numbers: 20080430076, 200902205
- China Ministry of Education. Grant Number: 200802461038
- Shanghai Natural Science Foundation. Grant Number: 10ZR1406100
Figure S1. Sustained treatment with echinacoside has no effects on the survival or H2O2 production in normal cells.
Figure S2. Echinacoside transient exposure selectively protects neuronal cells and non-neuronal cells overexpressed with TrkA/TrkB against rotenone injury.
Figure S3. Transient exposure to echinacoside alone or with K252a or U0126 for 2 h had no influence on normal neuronal cell survival.
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