Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro-inflammatory phenotype
Article first published online: 22 AUG 2013
© 2013 International Society for Neurochemistry
Journal of Neurochemistry
Volume 127, Issue 2, pages 245–258, October 2013
How to Cite
J. Neurochem.(2013) 127, 245–258.
- Issue published online: 4 OCT 2013
- Article first published online: 22 AUG 2013
- Accepted manuscript online: 6 AUG 2013 08:55PM EST
- Manuscript Accepted: 29 JUL 2013
- Manuscript Revised: 25 JUL 2013
- Manuscript Received: 1 APR 2013
- Batten Disease Support and Research Administration (BDSRA)
- UNMC Edna Ittner Pediatric Research Fund
- UNMC-Chinese Scholarship Council fellowship
Figure S1. Cytokine production is exacerbated in CLN3Δex7/8 macrophages in response to JNCL-relevant stimuli.
Figure S2. Microglial reactive oxygen species (ROS) production is not affected by CLN3 mutation.
Figure S3. Mitochondrial respiration and reactive oxygen species (ROS) production is similar between CLN3Δex7/8 and WT microglia.
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