Urban air pollutants reduce synaptic function of CA1 neurons via an NMDA/NȮ pathway in vitro
Version of Record online: 26 AUG 2013
© 2013 International Society for Neurochemistry
Journal of Neurochemistry
Volume 127, Issue 4, pages 509–519, November 2013
How to Cite
J. Neurochem. (2013) 127, 509–519.
- Issue online: 4 NOV 2013
- Version of Record online: 26 AUG 2013
- Accepted manuscript online: 8 AUG 2013 09:31AM EST
- Manuscript Accepted: 29 JUL 2013
- Manuscript Revised: 27 JUN 2013
- Manuscript Received: 2 APR 2013
- NIH. Grant Numbers: AG-040683, AG-040753
- USC Collaborative Research Fund
- South Coast Air Quality Management District. Grant Number: 11527
- NIA training Grant. Grant Number: T32 AG000037
Figure S1. Representative westerns blots from CA1 and DG lysates of hippocampal slices incubated in nPM for 2h from a single experiment.
Figure S2. Hippocampal neuron cell culture responses to nPM.
Figure S3. Immunoprecipitation of CA1 neuron nitrosylated glutamate receptors.
Figure S4. AP5 blocked nPM mediated cell injury to hippocampal neurons.
Table S1. Antibodies
Table S2. Hippocampal slice synaptic protein response to nPM
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