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HIV-1 protein Tat produces biphasic changes in NMDA-evoked increases in intracellular Ca2+ concentration via activation of Src kinase and nitric oxide signaling pathways
Article first published online: 19 APR 2014
© 2014 International Society for Neurochemistry
Journal of Neurochemistry
Volume 130, Issue 5, pages 642–656, September 2014
How to Cite
J. Neurochem. (2014) 130, 642–656.
- Issue published online: 20 AUG 2014
- Article first published online: 19 APR 2014
- Accepted manuscript online: 26 MAR 2014 01:12AM EST
- Manuscript Accepted: 11 MAR 2014
- Manuscript Revised: 5 MAR 2014
- Manuscript Received: 26 DEC 2013
- National Institutes of Health. Grant Numbers: DA07304, DA035663, DA034696, MH061933
- National Institute on Drug Abuse Training Grants. Grant Numbers: DA007097, DA007234
Figure S1. IL-1β-induced a biphasic change in NMDA-evoked [Ca2+]i responses.
Figure S2. Forty-eight-hour exposure to Tat does not affect the amplitude of NMDA-evoked [Ca2+]i responses in the presence of the GluN2B antagonist ifenprodil.
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