Targeting calpain-mediated proteolysis and peptide signaling as a strategy to reduce injury in multiple sclerosis

Authors

  • Thad A. Rosenberger

    Corresponding author
    1. University of North Dakota School of Medicine and Health Sciences, Department of Basic Sciences, Grand Forks, North Dakota
    • Address correspondence and reprint requests to Thad A. Rosenberger, University of North Dakota School of Medicine and Health Sciences, Department of Basic Sciences, 501 North Columbia Road, Grand Forks, North Dakota 58203. E-mail: thad.rosenberger@med.und.edu

    Search for more papers by this author

  • Read the full article ‘Effects of a novel orally administered calpain inhibitor SNJ-1945 on immunomodulation and neurodegeneration in a murine model of multiple sclerosis’ on page 268

Abstract

Read the full article ‘Effects of a novel orally administered calpain inhibitor SNJ-1945 on immunomodulation and neurodegeneration in a murine model of multiple sclerosis’ on page 268

Ancillary