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liv12047-sup-0001-FigS1.docxWord document11KFig. S1. Distribution of urinary π GST α GST and KIM1 in different cohort of patients. (a): Urinary π GST (from distal tubule) was upregulated in patients with acute deterioration of cirrhosis but not significantly different, the levels were similar between healthy volunteers and stablecirrhosis. (b): Represents urinary α GST (from proximal tubule) were similar between healthy volunteers and stable cirrhosis, though the levels were higher in patients with acute deterioration of cirrhosis did not significance. (c): shows the levels of KIM1 in different cohorts. Urinary KIM1 levels were higher in patients with acute deterioration of cirrhosis with no renal dysfunction but similar between healthy volunteers and stable cirrhosis. ‘Ψ’ represents the comparison between healthy volunteers and in patients with acute deterioration of cirrhosis with no renal dysfunction.
liv12047-sup-0002-FigS2.docxWord document10KFig. S2. Representative uTLR4 western blot in control subjects, patients with compensated cirrhosis and in patients with acute deterioration of cirrhosis with and without renal dysfunction.

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