This work was supported by Grant HL-13629 (RHA), 1UL1RR031973 (AS), and AHA 11GRNT7690032 (JAP).
Low-frequency human platelet antigens as triggers for neonatal alloimmune thrombocytopenia
Article first published online: 16 OCT 2013
© 2013 American Association of Blood Banks
Volume 54, Issue 5, pages 1286–1293, May 2014
How to Cite
Peterson, J. A., Gitter, M., Bougie, D. W., Pechauer, S., Hopp, K. A., Pietz, B., Szabo, A., Curtis, B. R., McFarland, J. and Aster, R. H. (2014), Low-frequency human platelet antigens as triggers for neonatal alloimmune thrombocytopenia. Transfusion, 54: 1286–1293. doi: 10.1111/trf.12450
- Issue published online: 12 MAY 2014
- Article first published online: 16 OCT 2013
- Manuscript Accepted: 27 AUG 2013
- Manuscript Revised: 5 AUG 2013
- Manuscript Received: 19 JUN 2013
- AHA 11GRNT7690032
- Howard Hughes Medical Institute
- National Institutes of Health, National Human Genome Research Institute. Grant Numbers: R01-HG00257, P50-HG00098
Fig. S1. Allelic discrimination assays. Typical HPA-21 (A) and HPA-12 (B) results shown for 50 fathers of unresolved NAIT cases. Signal intensities of Cy5 (HPA-21bw or HPA-12bw) versus FAM (HPA-21a or HPA-12bw) fluorophores are plotted. No template controls are denoted by grey diamonds. Homozygous and heterozygous samples are denoted with black diamonds and triangles respectively. Unknowns that tested positive in the assay are denoted with grey circles. Each unknown that typed at HPA-21a/bw or HPA-12a/bw was verified with sequence analysis.
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