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Sandra E. Wiley, Alexander Y. Andreyev, Ajit S. Divakaruni, Robert Karisch, Guy Perkins, Estelle A. Wall, Peter van der Geer, Yi-Fan Chen, Ting-Fen Tsai, Melvin I. Simon, Benjamin G. Neel, Jack E. Dixon and Anne N. Murphy Wolfram Syndrome protein, Miner1, regulates sulphydryl redox status, the unfolded protein response, and Ca2+ homeostasis EMBO Molecular Medicine 5

Article first published online: 24 MAY 2013 | DOI: 10.1002/emmm.201201429

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Wolfram syndrome (DIDMOAD) is an incurable metabolic disease caused by mutations in Wolframin or Miner1 genes. This study reveals Miner1 biological role in cellular redox status and proposes antioxidant as therapeutic strategy against DIDMOAD.

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