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T. Prell, J. Lautenschläger, O. W. Witte, M. T. Carri and J. Grosskreutz The unfolded protein response in models of human mutant G93A amyotrophic lateral sclerosis European Journal of Neuroscience 35

Article first published online: 6 MAR 2012 | DOI: 10.1111/j.1460-9568.2012.08008.x

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Recent studies indicate that endoplasmic reticulum (ER) stress is involved in the pathogenesis of familial and sporadic amyotrophic lateral sclerosis (ALS). ER stress occurs when the ER–mitochondria calcium cycle (ERMCC) is disturbed and misfolded proteins accumulate in the ER.

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