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Mireia Herrando-Grabulosa, Caty Casas and José Aguilera The C-terminal domain of tetanus toxin protects motoneurons against acute excitotoxic damage on spinal cord organotypic cultures Journal of Neurochemistry 124

Version of Record online: 15 NOV 2012 | DOI: 10.1111/jnc.12062

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One of the main processes that induces motoneuron (MN) cell death is the excitotocity triggered after Spinal Cord Injury (SCI). C-terminal domain of tetanus toxin (Hc-TeTx domain) protects motoneurons against this damage by activating several pro-survival signalling pathways, such as autophagy. Hc-TeTx could be useful as a neuroprotector agent to promotes MN survival.

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