Glia

Cover image for Vol. 65 Issue 10

Edited By: Bruce R. Ransom and Helmut Kettenmann

Impact Factor: 6.2

ISI Journal Citation Reports © Ranking: 2016: 23/258 (Neurosciences)

Online ISSN: 1098-1136

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2016 Impact Factor:
GLIA is pleased to announce its new Impact Factor is 6.2!

2017 Release of Journal Citation Reports®. Source: Thomson Reuters 2016 Citation Data







Recently Published Articles

  1. Schwann cell dedifferentiation-associated demyelination leads to exocytotic myelin clearance in inflammatory segmental demyelination

    So Young Jang, Byeol-A Yoon, Yoon Kyung Shin, Seoug Hoon Yun, Young Rae Jo, Yun Young Choi, Meejung Ahn, Taekyun Shin, Joo In Park, Jong Kuk Kim and Hwan Tae Park

    Version of Record online: 10 AUG 2017 | DOI: 10.1002/glia.23200

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    Main Points

    • Schwann cells exhibit stereotypic profiles of dedifferentiation in inflammatory segmental demyelination
    • Autophagolysosome activation in Schwann cell dedifferentiation-associated demyelination (SAD) leads to exocytotic myelin clearance
  2. You have full text access to this OnlineOpen article
    G protein-coupled receptor 37-like 1 modulates astrocyte glutamate transporters and neuronal NMDA receptors and is neuroprotective in ischemia

    Sarah Jolly, Narges Bazargani, Alejandra C. Quiroga, Nigel P. Pringle, David Attwell, William D. Richardson and Huiliang Li

    Version of Record online: 10 AUG 2017 | DOI: 10.1002/glia.23198

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    Main Points

    • GPR37L1 is expressed by astrocytes and some oligodendrocyte precursors in adult mice.
    • GPR37L1 activation by its ligand prosaptide inhibits astrocyte glutamate transporters and neuronal NMDA receptors, reducing glutamate neurotoxicity in ischemia.
  3. A specific GABAergic synapse onto oligodendrocyte precursors does not regulate cortical oligodendrogenesis

    Maddalena Balia, Najate Benamer and María Cecilia Angulo

    Version of Record online: 10 AUG 2017 | DOI: 10.1002/glia.23197

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    Main Points

    • Inactivation of γ2-mediated synapses of OPCs does not affect OPC proliferation, differentiation or myelination of presynaptic interneurons.
    • This inactivation reduces OPC density, suggesting a synaptic role in regulating OPC cellular homeostasis.
  4. Subcellular reorganization and altered phosphorylation of the astrocytic gap junction protein connexin43 in human and experimental temporal lobe epilepsy

    Tushar Deshpande, Tingsong Li, Michel K. Herde, Albert Becker, Hartmut Vatter, Martin K. Schwarz, Christian Henneberger, Christian Steinhäuser and Peter Bedner

    Version of Record online: 10 AUG 2017 | DOI: 10.1002/glia.23196

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    Main Points

    • We investigated expression of Cx30 and Cx43 in chronic human and experimental temporal lobe epilepsy
    • In sclerosis, Cx43 undergoes changes in phosphorylation and accumulates in perivascular endfeet, which may account for loss of astrocyte coupling
  5. The α2β2 isoform combination dominates the astrocytic Na+/K+-ATPase activity and is rendered nonfunctional by the α2.G301R familial hemiplegic migraine type 2-associated mutation

    Anca Stoica, Brian Roland Larsen, Mette Assentoft, Rikke Holm, Leanne Melissa Holt, Frederik Vilhardt, Bente Vilsen, Karin Lykke-Hartmann, Michelle Lynne Olsen and Nanna MacAulay

    Version of Record online: 8 AUG 2017 | DOI: 10.1002/glia.23194

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    Main Points

    • The astrocytic Na+/K+-ATPase activity is dominated by the [K+]o-sensitive α2β2 isozyme with a smaller contribution from the [Na+]i-sensitive α2β1
    • The FHM2-associated mutation α2.G301R renders the Na+/K+-ATPase nonfunctional

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