Birth Defects Research Part A: Clinical and Molecular Teratology
© Wiley Periodicals, Inc.
Edited By: Michel Vekemans
Impact Factor: 2.089
ISI Journal Citation Reports © Ranking: 2014: 32/41 (Developmental Biology); 53/87 (Toxicology)
Online ISSN: 1542-0760
Call for Papers
The journal Birth Defects Research Part A: Clinical and Molecular Teratology (BDRA) welcomes your submissions.
BDRA is concerned with the explanation of the causative factors and mechanisms leading to adverse pregnancy outcomes in the human population, including structural birth defects, pregnancy loss and postnatal functional defects and also to the identification of prenatal factors and biological mechanisms that reduce risk of adverse pregnancy outcomes
The journal welcomes research articles related to the following areas:
Clinical Dysmorphology/Genetics - identify mutant genes and polymorphisms or epigenetic alterations contributing to risk for specific birth defects, and their interactions with specific environmental factors; describe developmental mechanisms leading to specific isolated birth defects or birth defect syndromes; describe pregnancy outcomes after in utero exposure to potential developmental toxicants.
Epidemiology/Risk Assessment - identify human developmental toxicants, adverse and protective maternal factors, genetic risk factors and gene-environment interactions using epidemiologic approaches; develop and critically evaluate approaches to the prevention of birth defects including risk assessment and clinical counseling.
Experimental/Molecular Teratology - elucidate mechanisms and processes of developmental toxicants or adverse maternal factors; analyze critical events and critical molecular targets, e.g., signal transduction pathways, molecular stress and checkpoint pathways, and drug metabolizing enzymes.
Genomics and Proteomics - identify or predict mechanisms of adverse developmental outcomes or predict preventative mechanisms through use of genomic/proteomic analyses and databases, computer simulations and modeling.
Animal and in vitro Models - identify critical factors and pathways at the genetic, epigenetic, molecular, cellular, and tissue levels that play causal or preventative roles in the liability to induction of congenital defects in any strain, mutant, multifactorial animal model or in vitro tissue model.
Please submit your papers online at ScholarOne Manuscripts.
Address all editorial correspondence to:
Michel Vekemans, Editor-in-Chief
Genetics and INSERM U781
Université Paris Descarte
Hôpital Necker-Enfants Malades
75743 Paris, France