Journal of Neurochemistry

Cover image for Vol. 130 Issue 3

Edited By: Jörg Schulz

Impact Factor: 3.973

ISI Journal Citation Reports © Ranking: 2012: 71/252 (Neurosciences); 83/290 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Striatal Serotonin 2C receptors decrease nigrostriatal dopamine release by increasing GABA-A receptor tone in the substantia nigra

    M.V. Burke, C. Nocjar, A.J. Sonneborn, A.C. McCreary and E.A. Pehek

    Accepted manuscript online: 29 JUL 2014 08:42AM EST | DOI: 10.1111/jnc.12842

  2. Pleiotrophin differentially regulates the rewarding and sedative effects of ethanol

    Marta Vicente-Rodríguez, Carmen Pérez-García, Marcel Ferrer-Alcón, María Uribarri, María G. Sánchez-Alonso, María P. Ramos and Gonzalo Herradón

    Accepted manuscript online: 29 JUL 2014 08:42AM EST | DOI: 10.1111/jnc.12841

  3. Cannabinoid receptor 1 modulates the autophagic flux independent of mTOR- and BECLIN1-complex

    Christof Hiebel, Tanja Kromm, Marcel Stark and Christian Behl

    Accepted manuscript online: 28 JUL 2014 04:20AM EST | DOI: 10.1111/jnc.12839

  4. A distinct subfraction of Aβ is responsible for the high-affinity Pittsburgh compound B-binding site in Alzheimer's disease brain

    Sergey V. Matveev, Hans Peter Spielmann, Brittney M. Metts, Jing Chen, Fredrick Onono, Haining Zhu, Stephen W. Scheff, Lary C. Walker and Harry LeVine III

    Article first published online: 27 JUL 2014 | DOI: 10.1111/jnc.12815

    Thumbnail image of graphical abstract

    A lipid-associated subpopulation of Aβ accounts for the high-affinity binding of Pittsburgh compound B (PIB) in Alzheimer's disease brain. Mass spectrometry of the isolated PIB-binding site from frontal cortex identified Aβ peptides and a set of plaque-associated proteins in AD but not age-matched normal brain. The PIB-binding site may represent a particularly pathogenic entity and/or report local pathologic conditions.