Journal of Neurochemistry

Cover image for Vol. 131 Issue 1

Edited By: Jörg Schulz

Impact Factor: 4.244

ISI Journal Citation Reports © Ranking: 2013: 63/251 (Neurosciences); 74/291 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Positive feedback of NR2B-containing NMDA receptor activity is the initial step towards visual imprinting, a model for juvenile learning

    Tomoharu Nakamori, Katsushige Sato, Masae Kinoshita, Tomoyuki Kanamatsu, Hiroyuki Sakagami, Kohichi Tanaka and Hiroko Ohki-Hamazaki

    Accepted manuscript online: 1 OCT 2014 01:37AM EST | DOI: 10.1111/jnc.12954

  2. Astrocytes mediate HIV-1 Tat-induced neuronal damage via ligand-gated ion channel P2X7R

    Manju Tewari, . Monika, Rebu K Varghese, Malini Menon and Pankaj Seth

    Accepted manuscript online: 1 OCT 2014 01:37AM EST | DOI: 10.1111/jnc.12953

  3. Ceftriaxone modulates uptake activity of glial glutamate transporter-1 against global brain ischemia in rats

    Yu-Yan Hu, Jing Xu, Min Zhang, Dan Wang, Li Li and Wen-Bin Li

    Accepted manuscript online: 30 SEP 2014 11:10AM EST | DOI: 10.1111/jnc.12958

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    High-Affinity Glutamate Transporters in Chronic Pain: An Emerging Therapeutic Target

    Georgi Gegelashvili and Ole Jannik Bjerrum

    Accepted manuscript online: 30 SEP 2014 11:08AM EST | DOI: 10.1111/jnc.12957

  5. Hyperammonemia enhances the function and expression of P-glycoprotein and Mrp2 at the blood–brain barrier through NF-κB

    Ji Zhang, Mian Zhang, Binbin Sun, Ying Li, Ping Xu, Can Liu, Li Liu and Xiaodong Liu

    Article first published online: 26 SEP 2014 | DOI: 10.1111/jnc.12944

    Thumbnail image of graphical abstract

    Hyperammonemia, a proverbial main factor responsible for neurocognitive disorder and blood–brain barrier (BBB) dysfunction resulting from liver failure, could increase the expression and activity of P-glycoprotein and multidrug resistance-associated protein 2 (Mrp2) at the BBB both in vivo and in vitro. Furthermore, the NF-κB activation stimulated by hyperammonemia may be the potential mechanism underlying such abnormalities induced by hyperammonemia.