Journal of Neurochemistry

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Edited By: Jörg Schulz

Impact Factor: 4.083

ISI Journal Citation Reports © Ranking: 2016: 65/258 (Neurosciences); 77/286 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Ischemia responsive protein 94 is a key mediator of ischemic neuronal injury-induced microglial activation

    Rajiv Tikamdas, Sarthak Singhal, Ping Zhang, Justin A. Smith, Eric G. Krause, Stanley M Stevens Jr, Sihong Song and Bin Liu

    Accepted manuscript online: 22 JUN 2017 11:10AM EST | DOI: 10.1111/jnc.14111

  2. The interaction between progranulin and prosaposin is mediated by granulins and the linker region between saposin B and C

    Xiaolai Zhou, Peter M. Sullivan, Lirong Sun and Fenghua Hu

    Accepted manuscript online: 22 JUN 2017 11:10AM EST | DOI: 10.1111/jnc.14110

  3. You have free access to this content
    Re-imagining Alzheimer's disease – the diminishing importance of amyloid and a glimpse of what lies ahead

    Kai-Hei Tse and Karl Herrup

    Version of Record online: 21 JUN 2017 | DOI: 10.1111/jnc.14079

    Thumbnail image of graphical abstract

    The Alzheimer's field can seem like the blind men of the legend trying to describe an elephant by the first part they touch. The amyloid plaque has been inevitably the first part recognized by the newcomers to the Alzheimer's field. Nevertheless, looking at the longitudinal changes of the human brain, there are other age-associated pathologies prior to amyloid depositions that can equally cause age-related dementia. Indeed, a more holistic approach is needed to understand Alzheimer's. Here we offer three possibilities, namely DNA damage, cell cycle dysregulation and myelin degradation, as the examples of how amyloid-independent mechanisms could cause age-associated dementia.

  4. Ghrelin receptors mediate ghrelin-induced excitation of agouti-related protein/neuropeptide Y but not pro-opiomelanocortin neurons

    Shao-Rui Chen, Hong Chen, Jing-Jing Zhou, Geetali Pradhan, Yuxiang Sun, Hui-Lin Pan and De-Pei Li

    Version of Record online: 21 JUN 2017 | DOI: 10.1111/jnc.14080

    Thumbnail image of graphical abstract

    Ghrelin increases food intake and body weight by stimulating orexigenic AgRP/NPY neurons and inhibiting anorexic POMC neurons in the hypothalamus. The authors showed that although ghrelin excites AgRP/NPY neurons through ghrelin receptors, it also stimulates POMC neurons independent of ghrelin receptors. Thus, ghrelin stimulates AgRP/NPY and POMC neurons through distinct signaling mechanisms.

  5. Pimozide reduces toxic forms of tau in TauC3 mice via AMPK-mediated autophagy

    YoungDoo Kim, Eun Il Jeong, Jihoon Nah, Seung-Min Yoo, WonJae Lee, Youbin Kim, Seowon Moon, Se-Hoon Hong and Yong-Keun Jung

    Accepted manuscript online: 20 JUN 2017 01:24PM EST | DOI: 10.1111/jnc.14109

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