Journal of Neurochemistry

Cover image for Vol. 131 Issue 5

Edited By: Jörg Schulz

Impact Factor: 4.244

ISI Journal Citation Reports © Ranking: 2013: 63/252 (Neurosciences); 74/291 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Identification of the role of bone morphogenetic protein (BMP) and TGF-β signaling in the trajectory of serotonergic differentiation in a rapid assay in mouse embryonic stem cells in vitro

    Atsushi Yamasaki, Atsushi Kasai, Akihiro Toi, Maki Kurita, Saki Kimoto, Atsuko Hayata-Takano, Takanobu Nakazawa, Kazuki Nagayasu, Norihito Shintani, Ryota Hashimoto, Akira Ito, Herbert Y. Meltzer, Yukio Ago, James A. Waschek, Yusuke Onaka, Toshio Matsuda, Akemichi Baba and Hitoshi Hashimoto

    Accepted manuscript online: 25 NOV 2014 01:41AM EST | DOI: 10.1111/jnc.12999

  2. Elevated spinal monoamine neurotransmitters after antenatal hypoxia-ischemia in rabbit cerebral palsy model

    Alexander Drobyshevsky, Silvia Honda Takada, Kehuan Luo, Matthew Derrick, Lei Yu, Katharina A. Quinlan, Jeannette Vasquez-Vivar, Maria Inês Nogueira and Sidhartha Tan

    Accepted manuscript online: 25 NOV 2014 01:29AM EST | DOI: 10.1111/jnc.12997

  3. Different effects of bisphenol-A on memory behavior and synaptic modification in intact and estrogen-deprived female mice

    Xiaohong Xu, Ting Gu and Qiaoqiao Shen

    Accepted manuscript online: 25 NOV 2014 01:28AM EST | DOI: 10.1111/jnc.12998

  4. Cleaning up after ICH: the role of Nrf2 in modulating microglia function and hematoma clearance

    Xiurong Zhao, Guanghua Sun, Shun-Ming Ting, Shen Song, Jie Zhang, Nancy J. Edwards and Jaroslaw Aronowski

    Article first published online: 24 NOV 2014 | DOI: 10.1111/jnc.12974

    Thumbnail image of graphical abstract

    Microglia/Macrophages (MMφ) utilize the scavenger receptor CD36 to internalize the red blood cells (RBC) during the hematoma cleanup process after intracerebral hemorrhage (ICH). During RBC catabolism in phagosomes of MMφ, reactive oxygen species (ROS) are generated causing oxidative stress and injury to the phagocyte and the neurovascular unit. We propose that the treatment of MMφ with Nrf2 agonist (sulforaphane) could:(1) speed up the phagocytosis process by up-regulating CD36 expression, and (2) improve MMΦ resistant to oxidative damage by up-regulating the expression of antioxidative enzymes such as catalase (Cat) or superoxide dismutase-1 (SOD).

  5. Low levels of methyl β-cyclodextrin disrupt GluA1-dependent synaptic potentiation but not synaptic depression

    Tae-Yong Choi, Sunmin Jung, Jihoon Nah, Hui-Yeon Ko, Su-Hyun Jo, Gehoon Chung, Kyungpyo Park, Yong-Keun Jung and Se-Young Choi

    Accepted manuscript online: 22 NOV 2014 03:21AM EST | DOI: 10.1111/jnc.12995