Journal of Neurochemistry

Cover image for Vol. 140 Issue 3

Edited By: Jörg Schulz

Impact Factor: 3.842

ISI Journal Citation Reports © Ranking: 2015: 71/256 (Neurosciences); 83/289 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Multiregional analysis of global 5-methylcytosine and 5-hydroxymethylcytosine throughout the progression of Alzheimer's disease (pages 383–394)

    Elizabeth M. Ellison, Erin L. Abner and Mark A. Lovell

    Version of Record online: 20 JAN 2017 | DOI: 10.1111/jnc.13912

    Thumbnail image of graphical abstract

    Cytosine modifications can alter transcriptional states and may play a role in Alzheimer's disease (AD). DNA from AD affected brain regions was analyzed using gas chromatography mass spectrometry for normal aging, preclinical AD (PCAD), mild cognitive impairment (MCI), late onset AD (LOAD), frontotemporal lobar degeneration (FTLD), and dementia with Lewy bodies (DLB) subjects. Alterations in 5-methylcytosine (5-mC) and 5-hydroxymethylcytosine (5-hmC) were seen in early stages of AD, indicating a possible role of epigenetic modification in AD progression.

  2. Effects of a dietary ketone ester on hippocampal glycolytic and TCA cycle intermediates and amino acids in a 3xTgAD mouse model of Alzheimer's disease

    Robert J. Pawlosky, Martin F. Kemper, Yoshihero Kashiwaya, M. Todd King, Mark P. Mattson and Richard L. Veech

    Accepted manuscript online: 18 JAN 2017 06:40PM EST | DOI: 10.1111/jnc.13958

  3. Noradrenergic transmission in the central medial thalamic nucleus modulates the electroencephalographic activity and emergence from propofol anesthesia in rats

    Bao Fu, Tian Yu, Jie Yuan, Xingrui Gong and Mazhong Zhang

    Accepted manuscript online: 16 JAN 2017 06:28AM EST | DOI: 10.1111/jnc.13939

  4. The role of p38alpha in Schwann cells in regulating peripheral nerve myelination and repair

    Sheridan L. Roberts, Xin-peng Dun, Gemma Dee, Bethany Gray, Thomas Mindos and David B. Parkinson

    Version of Record online: 13 JAN 2017 | DOI: 10.1111/jnc.13929

    Thumbnail image of graphical abstract

    Using Schwann cell-specific p38α null mice, we aimed to clarify and characterise the functional role of p38α on peripheral nerve myelination in vivo. Here, we demonstrate for the first time in vivo that p38α functions as a negative regulator of myelination. Specific depletion of p38α in Schwann cells results in accelerated myelination and myelin protein expression during development, as well as early delays in Schwann cell demyelination and myelin protein degradation following nerve injury.

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