Journal of Neurochemistry

Cover image for Vol. 131 Issue 5

Edited By: Jörg Schulz

Impact Factor: 4.244

ISI Journal Citation Reports © Ranking: 2013: 63/252 (Neurosciences); 74/291 (Biochemistry & Molecular Biology)

Online ISSN: 1471-4159

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Recently Published Articles

  1. Low levels of methyl β-cyclodextrin disrupt GluA1-dependent synaptic potentiation but not synaptic depression

    Tae-Yong Choi, Sunmin Jung, Jihoon Nah, Hui-Yeon Ko, Su-Hyun Jo, Gehoon Chung, Kyungpyo Park, Yong-Keun Jung and Se-Young Choi

    Accepted manuscript online: 22 NOV 2014 03:21AM EST | DOI: 10.1111/jnc.12995

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    Zheng Chen, Bin Hu, Fuzhou Wang, Lingling Du, Baosheng Huang, Lixin Li, Jia Qi and Xiang Wang

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  4. α-synuclein-induced mitochondrial dysfunction in isolated preparation and intact cells: Implications in the pathogenesis of Parkinson's disease

    Aritri Bir, Oishimaya Sen, Shruti Anand, Vineet Kumar Khemka, Priyanjalee Banerjee, Roberto Cappai, Arghyadip Sahoo and Sasanka Chakrabarti

    Article first published online: 18 NOV 2014 | DOI: 10.1111/jnc.12966

    Thumbnail image of graphical abstract

    α-Synuclein is shown to cause mitochondrial impairment through interaction with permeability transition pore complex in isolated preparations. Intracellular accumulation of α-synuclein in SHSY5Y cells following proteasomal inhibition leads to mitochondrial impairment and cell death which could be prevented by knocking down α-synuclein gene. The results link mitochondrial dysfunction and α-synuclein accumulation, two key pathogenic mechanisms of Parkinson's disease, in a common damage pathway.

  5. The effects of nesfatin-1 in the paraventricular nucleus on gastric motility and its potential regulation by the lateral hypothalamic area in rats

    Fei-fei Guo, Luo Xu, Sheng-li Gao, Xiang-rong Sun, Zhi-ling Li and Yan-ling Gong

    Article first published online: 18 NOV 2014 | DOI: 10.1111/jnc.12973

    Thumbnail image of graphical abstract

    Nesfatin-1 regulated the gastric distension (GD)-responsive neurons and reduced gastric motility in the paraventricular nucleus (PVN), which were partly blocked by H4928. Electrical stimulation of the lateral hypothalamic area (LHA) increased the firing activities of GD-responsive neurons in the PVN and promoted the gastric motility. NUCB2/nesfatin-1/fluorogold double-labeled neurons were identified in the LHA, indicating that nesfatin-1 in the PVN could play a pivotal role in the central control of gastric motility and the LHA may participate in the regulatory process. NUCB2 = nucleobindin-2

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