Genes, Brain and Behavior

Cover image for Vol. 14 Issue 1

Early View (Online Version of Record published before inclusion in an issue)

Edited By: Andrew Holmes

Impact Factor: 3.505

ISI Journal Citation Reports © Ranking: 2013: 10/49 (Behavioral Sciences); 93/252 (Neurosciences)

Online ISSN: 1601-183X

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  1. 1 - 6
  1. Original Articles

    1. Hypothalamic gene expression underlying pre-hibernation satiety

      C. Schwartz, M. Hampton and M. T. Andrews

      Article first published online: 17 FEB 2015 | DOI: 10.1111/gbb.12199

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      A rapid and extreme feeding transition in pre-hibernation ground squirrels described by hypothalamic gene expression.

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      Sleep-like behavior and 24-h rhythm disruption in the Tc1 mouse model of Down syndrome

      I. Heise, S. P. Fisher, G. T. Banks, S. Wells, S. N. Peirson, R. G. Foster and P. M. Nolan

      Article first published online: 16 FEB 2015 | DOI: 10.1111/gbb.12198

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      Video analysis detects an extended period of wakefulness in Tc1 mice during early night (active phase).

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    4. Behavioral phenotypes in males with XYY and possible role of increased NLGN4Y expression in autism features

      J. L. Ross, N. Tartaglia, D. E. Merry, M. Dalva and A. R. Zinn

      Article first published online: 1 FEB 2015 | DOI: 10.1111/gbb.12200

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      Expression of the Y gene, NLGN4Y, is increased in boys with XYY, and the level of expression correlates with overall social responsiveness and autism symptoms. Fig. 1 Expression levels NLGN4Y versus SRS Autistic Mannerisms t-scores

    5. Borderline personality disorder and childhood maltreatment: a genome-wide methylation analysis

      J. Prados, L. Stenz, P. Courtet, P. Prada, R. Nicastro, W. Adouan, S. Guillaume, E. Olié, J.-M. Aubry, A. Dayer and N. Perroud

      Article first published online: 1 FEB 2015 | DOI: 10.1111/gbb.12197

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      This genome-wide methylation analysis in borderline personality disorder highlights the potentially important role played by microRNAs in the etiology of neuropsychiatric disorders.

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      A lack of functional NK1 receptors explains most, but not all, abnormal behaviours of NK1R-/- mice1

      A. J. Porter, K. Pillidge, Y. C. Tsai, J. A. Dudley, S. P. Hunt, S. N. Peirson, L. A. Brown and S. C. Stanford

      Article first published online: 1 FEB 2015 | DOI: 10.1111/gbb.12195

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      Comparison of the homozygous progeny of homozygous and heterozygous breeding-pairs reveals that certain behavioural abnormalities in NK1R-/- mice are explained by their genotype, alone, but others rest on an interaction between genotype and epigenetic/environmental factors.

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