<?xml version="1.0" encoding="UTF-8"?>
<rdf:RDF xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#"><channel rdf:about="http://onlinelibrary.wiley.com/rss/journal/10.1111/(ISSN)1834-7819" xmlns="http://purl.org/rss/1.0/"><title>Australian Dental Journal</title><description> Wiley Online Library : Australian Dental Journal</description><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2F%28ISSN%291834-7819</link><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc</dc:publisher><dc:language xmlns:dc="http://purl.org/dc/elements/1.1/">en</dc:language><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/">© Australian Dental Association</dc:rights><prism:issn xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">0045-0421</prism:issn><prism:eIssn xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">1834-7819</prism:eIssn><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-03-01T00:00:00-05:00</dc:date><prism:coverDisplayDate xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">March 2013</prism:coverDisplayDate><prism:volume xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">58</prism:volume><prism:number xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">1</prism:number><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">1</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">129</prism:endingPage><image rdf:resource="http://onlinelibrary.wiley.com/store/10.1111/adj.2013.58.issue-1/asset/cover.gif?v=1&amp;s=ad22448c0ded95e369f5a55b369cbbbbbc9399a9"/><items><rdf:Seq><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12038"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12060"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12056"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12055"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12059"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12058"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12039"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12022"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12037"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12020"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12023"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12031"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12024"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12019"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12021"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12033"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12032"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12018"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12017"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12030"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12029"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12028"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12027"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12016"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12026"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12025"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12034"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12035"/><rdf:li rdf:resource="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12036"/></rdf:Seq></items></channel><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12038" xmlns="http://purl.org/rss/1.0/"><title>Effects of chlorhexidine on bonding durability of different adhesive systems using a novel thermocycling method</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12038</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Effects of chlorhexidine on bonding durability of different adhesive systems using a novel thermocycling method</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">D Deng, X Huang, C Huang, T Yang, X Du, Y Wang, X Ouyang, D Pei</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:24:49.011491-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12038</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12038</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12038</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12038-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The purpose of this study was to evaluate the influence of chlorhexidine on the bonding durability of etch-and-rinse and self-etch adhesive systems using the polymerase chain reaction (PCR) thermocycling method.</p></div></div>
<div class="section" id="adj12038-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Twenty freshly extracted intact human third molars were ground and bonded with either an etch-and-rinse adhesive (Single-Bond) or a self-etch adhesive (G-Bond). Specimens were either left untouched or placed in PCR tubes filled with three thermocycling mediums: water, chlorhexidine or silicone oil. Thermocycling (5000 cycles) was done using the PCR programme at temperatures of 5 °C and 55 °C. The microtensile bond strength (μTBS) was evaluated and interfacial nanoleakage was assessed by scanning electron microscopy before and after thermocycling.</p></div></div>
<div class="section" id="adj12038-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Significant differences were detected among groups kept in different media after thermocycling. For Single-Bond, both the chlorhexidine and silicone oil groups could preserve the μTBS (p &lt; 0.001). For G-Bond, μTBS of the chlorhexidine and water groups were significantly decreased (p &lt; 0.05). No obvious increase in silver deposition was observed in specimens incubated in water after thermocycling, less silver penetration was found in specimens incubated in chlorhexidine.</p></div></div>
<div class="section" id="adj12038-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>In this experimental model, chlorhexidine was found to preserve bonding durability in Single-Bond but have no significant effects on G-Bond.</p></div></div>
]]></content:encoded><description>


Background
The purpose of this study was to evaluate the influence of chlorhexidine on the bonding durability of etch-and-rinse and self-etch adhesive systems using the polymerase chain reaction (PCR) thermocycling method.


Methods
Twenty freshly extracted intact human third molars were ground and bonded with either an etch-and-rinse adhesive (Single-Bond) or a self-etch adhesive (G-Bond). Specimens were either left untouched or placed in PCR tubes filled with three thermocycling mediums: water, chlorhexidine or silicone oil. Thermocycling (5000 cycles) was done using the PCR programme at temperatures of 5 °C and 55 °C. The microtensile bond strength (μTBS) was evaluated and interfacial nanoleakage was assessed by scanning electron microscopy before and after thermocycling.


Results
Significant differences were detected among groups kept in different media after thermocycling. For Single-Bond, both the chlorhexidine and silicone oil groups could preserve the μTBS (p &lt; 0.001). For G-Bond, μTBS of the chlorhexidine and water groups were significantly decreased (p &lt; 0.05). No obvious increase in silver deposition was observed in specimens incubated in water after thermocycling, less silver penetration was found in specimens incubated in chlorhexidine.


Conclusions
In this experimental model, chlorhexidine was found to preserve bonding durability in Single-Bond but have no significant effects on G-Bond.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12060" xmlns="http://purl.org/rss/1.0/"><title>Will improving access to dental care improve oral health-related quality of life?</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12060</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Will improving access to dental care improve oral health-related quality of life?</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">LA Crocombe, GD Mahoney, AJ Spencer, M Waller</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:24:42.118397-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12060</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12060</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12060</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12060-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The aim of this study was to determine if Australian Defence Force (ADF) members had better oral health-related quality of life (OHRQoL) than the general Australian population and whether the difference was due to better access to dental care.</p></div></div>
<div class="section" id="adj12060-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>The OHRQoL, as measured by OHIP-14 summary indicators, of participants from the Defence Deployed Solomon Islands (SI) Health Study and the National Survey of Adult Oral Health 2004–06 (NSAOH) were compared. The SI sample was age/gender status-adjusted to match that of the NSAOH sample which was age/gender/regional location weighted to that of the Australian population.</p></div></div>
<div class="section" id="adj12060-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>NSAOH respondents with good access to dental care had lower OHIP-14 summary measures [frequency of impacts 8.5% (95% CI = 5.4, 11.6), extent mean = 0.16 (0.11, 0.22), severity mean = 5.0 (4.4, 5.6)] than the total NSAOH sample [frequency 18.6 (16.6, 20.7); extent 0.52 (0.44, 0.59); severity 7.6 (7.1, 8.1)]. The NSAOH respondents with both good access to dental care and self-reported good general health did not have as low OHIP-14 summary scores as in the SI sample [frequency 2.6 (1.2, 5.4), extent 0.05 (0.01, 0.10); severity 2.6 (1.9, 3.4)].</p></div></div>
<div class="section" id="adj12060-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>ADF members had better OHRQoL than the general Australian population, even those with good access to dental care and self-reported good general health.</p></div></div>
]]></content:encoded><description>


Background
The aim of this study was to determine if Australian Defence Force (ADF) members had better oral health-related quality of life (OHRQoL) than the general Australian population and whether the difference was due to better access to dental care.


Methods
The OHRQoL, as measured by OHIP-14 summary indicators, of participants from the Defence Deployed Solomon Islands (SI) Health Study and the National Survey of Adult Oral Health 2004–06 (NSAOH) were compared. The SI sample was age/gender status-adjusted to match that of the NSAOH sample which was age/gender/regional location weighted to that of the Australian population.


Results
NSAOH respondents with good access to dental care had lower OHIP-14 summary measures [frequency of impacts 8.5% (95% CI = 5.4, 11.6), extent mean = 0.16 (0.11, 0.22), severity mean = 5.0 (4.4, 5.6)] than the total NSAOH sample [frequency 18.6 (16.6, 20.7); extent 0.52 (0.44, 0.59); severity 7.6 (7.1, 8.1)]. The NSAOH respondents with both good access to dental care and self-reported good general health did not have as low OHIP-14 summary scores as in the SI sample [frequency 2.6 (1.2, 5.4), extent 0.05 (0.01, 0.10); severity 2.6 (1.9, 3.4)].


Conclusions
ADF members had better OHRQoL than the general Australian population, even those with good access to dental care and self-reported good general health.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12056" xmlns="http://purl.org/rss/1.0/"><title>A case of primary diffuse large B-cell non-Hodgkin's lymphoma misdiagnosed as chronic periapical periodontitis</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12056</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">A case of primary diffuse large B-cell non-Hodgkin's lymphoma misdiagnosed as chronic periapical periodontitis</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">M Jessri, AA Abdul Majeed, MA Matias, CS Farah</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:24:34.345054-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12056</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12056</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12056</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>Lymphoma is a malignant neoplasm of component cells of the lymphoid system which is very rare in the jaws. Here we report a case of primary diffuse large B-cell lymphoma located in the periapical region of a mandibular molar which was misdiagnosed as chronic periapical periodontitis. The present case was diagnosed at an early stage and effectively managed by chemotherapy. Although lymphoma of the mandible is rare, it must be considered in the differential diagnosis of radiolucent lesions in this region. Lack of knowledge of this rare presentation may lead to delays in diagnosis and poor prognosis.</p></div>
]]></content:encoded><description>

Lymphoma is a malignant neoplasm of component cells of the lymphoid system which is very rare in the jaws. Here we report a case of primary diffuse large B-cell lymphoma located in the periapical region of a mandibular molar which was misdiagnosed as chronic periapical periodontitis. The present case was diagnosed at an early stage and effectively managed by chemotherapy. Although lymphoma of the mandible is rare, it must be considered in the differential diagnosis of radiolucent lesions in this region. Lack of knowledge of this rare presentation may lead to delays in diagnosis and poor prognosis.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12055" xmlns="http://purl.org/rss/1.0/"><title>Tooth wear and associated risk factors in a sample of Australian primary school children</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12055</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Tooth wear and associated risk factors in a sample of Australian primary school children</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">A Fung, L Brearley Messer</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:24:24.71299-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12055</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12055</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12055</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12055-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Anecdotally, tooth wear is increasing, perhaps attributable to diet. The irreversible surface loss may result in sensitivity and loss of form and function. Little data exist on the prevalence of tooth wear in Australian children. This study investigated consumption of potentially erosive foods and drinks, examining the prevalence, distribution of tooth wear and associations in a sample of children.</p></div></div>
<div class="section" id="adj12055-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Parents of 350, 6–12-year-olds reported their child's oral hygiene, dietary intake, medical and dental histories; 154 children (subsample) were examined. Associations were studied with single and multivariable analyses.</p></div></div>
<div class="section" id="adj12055-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Tooth wear was parentally reported for 17% (59 children of the study population) and observed in 66% (102 of the subsample), particularly affecting primary teeth. Significant risk factors for parentally reported tooth wear were: consuming 2–4 cups soft drink/day (OR = 9.52), citrus flavoured sweets/gums ≥1/day (5.10), citrus fruits 1–2/wk (4.28); tooth grinding (5.32); medical condition present (2.48); male gender (2.80). Drinking 2–4 cups fruit juice/day was a significant risk factor for both parentally reported (3.23) and observed tooth wear (3.97).</p></div></div>
<div class="section" id="adj12055-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Tooth wear appeared under-reported as some parents were unaware their child's teeth were affected. Significant risk factors for tooth wear were identifiable from children's histories. Risk factors should be addressed early so that tooth wear in the primary dentition does not affect permanent teeth.</p></div></div>
]]></content:encoded><description>


Background
Anecdotally, tooth wear is increasing, perhaps attributable to diet. The irreversible surface loss may result in sensitivity and loss of form and function. Little data exist on the prevalence of tooth wear in Australian children. This study investigated consumption of potentially erosive foods and drinks, examining the prevalence, distribution of tooth wear and associations in a sample of children.


Methods
Parents of 350, 6–12-year-olds reported their child's oral hygiene, dietary intake, medical and dental histories; 154 children (subsample) were examined. Associations were studied with single and multivariable analyses.


Results
Tooth wear was parentally reported for 17% (59 children of the study population) and observed in 66% (102 of the subsample), particularly affecting primary teeth. Significant risk factors for parentally reported tooth wear were: consuming 2–4 cups soft drink/day (OR = 9.52), citrus flavoured sweets/gums ≥1/day (5.10), citrus fruits 1–2/wk (4.28); tooth grinding (5.32); medical condition present (2.48); male gender (2.80). Drinking 2–4 cups fruit juice/day was a significant risk factor for both parentally reported (3.23) and observed tooth wear (3.97).


Conclusions
Tooth wear appeared under-reported as some parents were unaware their child's teeth were affected. Significant risk factors for tooth wear were identifiable from children's histories. Risk factors should be addressed early so that tooth wear in the primary dentition does not affect permanent teeth.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12059" xmlns="http://purl.org/rss/1.0/"><title>Association of dental caries and salivary sIgA with tobacco smoking</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12059</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Association of dental caries and salivary sIgA with tobacco smoking</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">L Golpasand Hagh, F Zakavi, S Ansarifar, O Ghasemzadeh, G Solgi</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:24:16.692117-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12059</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12059</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12059</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12059-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Salivary secretory IgA (sIgA) is said to play an important role in the immune response against dental caries. This study aimed to determine the salivary sIgA levels in healthy smokers and non-smokers, and its correlation with dental caries.</p></div></div>
<div class="section" id="adj12059-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>A total of 70 healthy subjects were selected and classified into four groups according to dental caries and tobacco smoking habits: smoking with caries (Group 1, n = 15); smoking without caries (Group 2, n = 15); non-smoking with caries (Group 3, n = 15); and non-smoking without caries (Group 4, n = 25). Salivary sIgA was measured using ELISA. The fissure and proximal caries were examined clinically and radiographically. Caries status was determined according to the decay surface index.</p></div></div>
<div class="section" id="adj12059-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Smokers showed a higher number of caries and the lowest concentration of sIgA. The highest levels of sIgA were observed in non-smoking and caries-free subjects compared to caries-active smokers (123.2 ± 19.9 vs. 13.3 ± 4.1 μg/ml respectively, p &lt; 0.001). Also, the mean level of sIgA in Group 4 was significantly higher than Group 3 (p = 0.009). More importantly, higher and significant levels of sIgA were found in Group 3 versus Group 1 (p &lt; 0.0001) and Group 2 (p = 0.0004).</p></div></div>
<div class="section" id="adj12059-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Our findings indicate that low concentrations of salivary sIgA are correlated with a higher prevalence of dental caries in smokers.</p></div></div>
]]></content:encoded><description>


Background
Salivary secretory IgA (sIgA) is said to play an important role in the immune response against dental caries. This study aimed to determine the salivary sIgA levels in healthy smokers and non-smokers, and its correlation with dental caries.


Methods
A total of 70 healthy subjects were selected and classified into four groups according to dental caries and tobacco smoking habits: smoking with caries (Group 1, n = 15); smoking without caries (Group 2, n = 15); non-smoking with caries (Group 3, n = 15); and non-smoking without caries (Group 4, n = 25). Salivary sIgA was measured using ELISA. The fissure and proximal caries were examined clinically and radiographically. Caries status was determined according to the decay surface index.


Results
Smokers showed a higher number of caries and the lowest concentration of sIgA. The highest levels of sIgA were observed in non-smoking and caries-free subjects compared to caries-active smokers (123.2 ± 19.9 vs. 13.3 ± 4.1 μg/ml respectively, p &lt; 0.001). Also, the mean level of sIgA in Group 4 was significantly higher than Group 3 (p = 0.009). More importantly, higher and significant levels of sIgA were found in Group 3 versus Group 1 (p &lt; 0.0001) and Group 2 (p = 0.0004).


Conclusions
Our findings indicate that low concentrations of salivary sIgA are correlated with a higher prevalence of dental caries in smokers.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12058" xmlns="http://purl.org/rss/1.0/"><title>Oral health and oral diseases in pregnancy: a multicentre survey of Italian postpartum women</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12058</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Oral health and oral diseases in pregnancy: a multicentre survey of Italian postpartum women</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">A Villa, S Abati, P Pileri, S Calabrese, G Capobianco, L Strohmenger, L Ottolenghi, I Cetin, GG Campus</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:22:43.316787-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12058</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12058</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12058</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12058-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The aim of this study was to explore the oral hygiene practices and oral health status of Italian postpartum women.</p></div></div>
<div class="section" id="adj12058-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>A self-administered questionnaire assessed socio-demographic information, oral hygiene habits and frequency of dental visits. All women received a thorough oral examination within five days after delivery. Logistic regression models were used to estimate odds ratios and 95% confidence intervals for exposures of interest and the presence of ‘severe’ periodontitis.</p></div></div>
<div class="section" id="adj12058-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Seven hundred and fifty women participated in the study; 99.1% brushed their teeth everyday and 59.9% visited the dentist annually. The mean frequency of sites with bleeding on probing was 16.1% and the median clinical attachment level was 2.1 mm. The mean caries experience score (DMFT) was 8. Severe periodontal disease was present in 21.9% of individuals. Patients who reported visiting a dentist only when in pain and women with three dental caries or more were significantly more likely to have periodontitis (OR: 1.6; 95% CI: 1.1–2.2; p &lt; 0.05 and OR: 2.3; 95% CI: 1.5–3.5; p &lt; 0.01, respectively).</p></div></div>
<div class="section" id="adj12058-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Given the possible association between maternal and infant oral health, and between periodontal infection and general health, antenatal care providers should collaborate with dentists to encourage all pregnant women to comply with the oral health professionals' recommendations regarding appropriate dental brushing techniques and the importance of dental visits.</p></div></div>
]]></content:encoded><description>


Background
The aim of this study was to explore the oral hygiene practices and oral health status of Italian postpartum women.


Methods
A self-administered questionnaire assessed socio-demographic information, oral hygiene habits and frequency of dental visits. All women received a thorough oral examination within five days after delivery. Logistic regression models were used to estimate odds ratios and 95% confidence intervals for exposures of interest and the presence of ‘severe’ periodontitis.


Results
Seven hundred and fifty women participated in the study; 99.1% brushed their teeth everyday and 59.9% visited the dentist annually. The mean frequency of sites with bleeding on probing was 16.1% and the median clinical attachment level was 2.1 mm. The mean caries experience score (DMFT) was 8. Severe periodontal disease was present in 21.9% of individuals. Patients who reported visiting a dentist only when in pain and women with three dental caries or more were significantly more likely to have periodontitis (OR: 1.6; 95% CI: 1.1–2.2; p &lt; 0.05 and OR: 2.3; 95% CI: 1.5–3.5; p &lt; 0.01, respectively).


Conclusions
Given the possible association between maternal and infant oral health, and between periodontal infection and general health, antenatal care providers should collaborate with dentists to encourage all pregnant women to comply with the oral health professionals' recommendations regarding appropriate dental brushing techniques and the importance of dental visits.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12039" xmlns="http://purl.org/rss/1.0/"><title>Developmental enamel defects in the primary dentition: aetiology and clinical management</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12039</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Developmental enamel defects in the primary dentition: aetiology and clinical management</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">S Salanitri, WK Seow</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-05-05T22:22:39.706939-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12039</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12039</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12039</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Original Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>Developmental enamel defects, presenting as enamel hypoplasia or opacities are caused by damage or disruption to the developing enamel organ as a result of inherited and acquired systemic conditions. The high prevalence of these defects in the primary dentition demonstrates the vulnerability of the teeth to changes in the pre- and postnatal environment. The presence of enamel hypoplasia increases the risk of primary teeth to early childhood caries and tooth wear as the defective enamel is thinner, more plaque retentive and less resistant to dissolution in acid compared to normal enamel. The purpose of this paper was to critically review the aetiology and clinical complications of developmental enamel defects in the primary dentition and propose recommendations for the clinical management of affected teeth.</p></div>
]]></content:encoded><description>

Developmental enamel defects, presenting as enamel hypoplasia or opacities are caused by damage or disruption to the developing enamel organ as a result of inherited and acquired systemic conditions. The high prevalence of these defects in the primary dentition demonstrates the vulnerability of the teeth to changes in the pre- and postnatal environment. The presence of enamel hypoplasia increases the risk of primary teeth to early childhood caries and tooth wear as the defective enamel is thinner, more plaque retentive and less resistant to dissolution in acid compared to normal enamel. The purpose of this paper was to critically review the aetiology and clinical complications of developmental enamel defects in the primary dentition and propose recommendations for the clinical management of affected teeth.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12022" xmlns="http://purl.org/rss/1.0/"><title>Evaluation of beta-2 microglobulin and alpha-2 macroglobulin levels in patients with different periodontal diseases</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12022</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Evaluation of beta-2 microglobulin and alpha-2 macroglobulin levels in patients with different periodontal diseases</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">AS Ertugrul, H Sahin, A Dikilitas, N Alpaslan, A Bozoglan</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-03-01T04:55:57.749516-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12022</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12022</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12022</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">n/a</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12022-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Beta-2 microglobulin (B2M) and alpha-2 macroglobulin (A2M) play key roles in the immune system. The aim of this study was to compare B2M and A2M levels in patients with different periodontal diseases.</p></div></div>
<div class="section" id="adj12022-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Eighty patients (20 periodontally healthy, 20 with gingivitis, 20 with chronic periodontitis and 20 with generalized aggressive periodontitis) were enrolled in the study. The analysis of B2M and A2M was performed on gingival crevicular fluid (GCF) using an enzyme-linked immunosorbent assay in GCF.</p></div></div>
<div class="section" id="adj12022-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The total levels of B2M and A2M were statistically lower in the periodontally healthy group than in the other groups (p &lt; 0.05) and significantly higher in the generalized aggressive periodontitis group compared to the other groups (p &lt; 0.05).</p></div></div>
<div class="section" id="adj12022-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>B2M and A2M play key roles in the balance between periodontal health and disease. It is proposed that tissues release B2M and A2M to stop inflammation and inhibit the proliferation of microorganisms and this may be the reason for the high levels of B2M and A2M in the generalized aggressive periodontitis and chronic periodontitis groups. B2M and A2M are assumed to be user-friendly and cost-effective markers for periodontal disease to identify asymptomatic diseases.</p></div></div>
]]></content:encoded><description>


Background
Beta-2 microglobulin (B2M) and alpha-2 macroglobulin (A2M) play key roles in the immune system. The aim of this study was to compare B2M and A2M levels in patients with different periodontal diseases.


Methods
Eighty patients (20 periodontally healthy, 20 with gingivitis, 20 with chronic periodontitis and 20 with generalized aggressive periodontitis) were enrolled in the study. The analysis of B2M and A2M was performed on gingival crevicular fluid (GCF) using an enzyme-linked immunosorbent assay in GCF.


Results
The total levels of B2M and A2M were statistically lower in the periodontally healthy group than in the other groups (p &lt; 0.05) and significantly higher in the generalized aggressive periodontitis group compared to the other groups (p &lt; 0.05).


Conclusions
B2M and A2M play key roles in the balance between periodontal health and disease. It is proposed that tissues release B2M and A2M to stop inflammation and inhibit the proliferation of microorganisms and this may be the reason for the high levels of B2M and A2M in the generalized aggressive periodontitis and chronic periodontitis groups. B2M and A2M are assumed to be user-friendly and cost-effective markers for periodontal disease to identify asymptomatic diseases.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12037" xmlns="http://purl.org/rss/1.0/"><title>Where is Dentistry Heading?</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12037</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Where is Dentistry Heading?</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">P Mark Bartold</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12037</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12037</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12037</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Editorial</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">1</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">1</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[]]></content:encoded><description/></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12020" xmlns="http://purl.org/rss/1.0/"><title>Human papillomavirus and oral disease – emerging evidence: a review</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12020</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Human papillomavirus and oral disease – emerging evidence: a review</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">SR Prabhu, DF Wilson</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-31T00:12:06.98214-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12020</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12020</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12020</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Review</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">2</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>Human papillomavirus (HPV) infections have received considerable attention in recent years. Of the 120 or so known types of the virus, some cause a variety of benign wart-like lesions of the skin and genital and oral mucosae, whilst others are aetiologically associated with cervical and anogenital cancers. Recent epidemiologic evidence suggests that HPV may also be an independent risk factor for oropharyngeal cancer. In this context it has been suggested that HPV virus may modulate the process of carcinogenesis in some tobacco and alcohol induced oropharyngeal cancers and act as the primary oncogenic agent for inducing carcinogenesis among non-smokers. Dental practitioners have a major role in detecting all lesions of the oral mucosa caused, or possibly caused, by HPV. This paper briefly reviews the current state of knowledge of molecular and clinical aspects of HPV infections of the oral mucosa.</p></div>
]]></content:encoded><description>

Human papillomavirus (HPV) infections have received considerable attention in recent years. Of the 120 or so known types of the virus, some cause a variety of benign wart-like lesions of the skin and genital and oral mucosae, whilst others are aetiologically associated with cervical and anogenital cancers. Recent epidemiologic evidence suggests that HPV may also be an independent risk factor for oropharyngeal cancer. In this context it has been suggested that HPV virus may modulate the process of carcinogenesis in some tobacco and alcohol induced oropharyngeal cancers and act as the primary oncogenic agent for inducing carcinogenesis among non-smokers. Dental practitioners have a major role in detecting all lesions of the oral mucosa caused, or possibly caused, by HPV. This paper briefly reviews the current state of knowledge of molecular and clinical aspects of HPV infections of the oral mucosa.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12023" xmlns="http://purl.org/rss/1.0/"><title>Stimulating the discussion on saliva substitutes: a clinical perspective</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12023</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Stimulating the discussion on saliva substitutes: a clinical perspective</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">F Dost, CS Farah</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T04:05:12.206575-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12023</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12023</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12023</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Review</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">11</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">17</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>Xerostomia is a significant problem commonly faced by patients and oral health practitioners. There is no cure for this condition, which commonly manifests as a side effect of medications, head and neck irradiation and other systemic conditions, such as Sjögren's syndrome and type 2 diabetes. It may also arise idiopathically. Therefore, treatment is palliative and takes the form of oral lubricants and saliva substitutes which aim to reduce symptoms associated with xerostomia as well as prevent oral disease secondary to it. Recently there has been an expansion of the number and range of products available in Australia for the palliative management of xerostomia. It is imperative then that oral health professionals have a sound understanding of the advantages and disadvantages of using such products as patients tend to be well informed about new products which are commercially available. This article discusses some of the most commonly available products used for the symptomatic relief and preventive management of xerostomia. Amongst the plethora of products available to the patient suffering from xerostomia, no single product or product range adequately reproduces the properties of natural saliva and therefore consideration of patients' concerns, needs and oral health state should be taken into account when formulating a home care regime. With Australia's ageing population and its heavier reliance on medications and treatments which may induce xerostomia, oral health professionals are likely to encounter this condition more than ever before and therefore an understanding of xerostomia and its management is essential to patient care.</p></div>
]]></content:encoded><description>

Xerostomia is a significant problem commonly faced by patients and oral health practitioners. There is no cure for this condition, which commonly manifests as a side effect of medications, head and neck irradiation and other systemic conditions, such as Sjögren's syndrome and type 2 diabetes. It may also arise idiopathically. Therefore, treatment is palliative and takes the form of oral lubricants and saliva substitutes which aim to reduce symptoms associated with xerostomia as well as prevent oral disease secondary to it. Recently there has been an expansion of the number and range of products available in Australia for the palliative management of xerostomia. It is imperative then that oral health professionals have a sound understanding of the advantages and disadvantages of using such products as patients tend to be well informed about new products which are commercially available. This article discusses some of the most commonly available products used for the symptomatic relief and preventive management of xerostomia. Amongst the plethora of products available to the patient suffering from xerostomia, no single product or product range adequately reproduces the properties of natural saliva and therefore consideration of patients' concerns, needs and oral health state should be taken into account when formulating a home care regime. With Australia's ageing population and its heavier reliance on medications and treatments which may induce xerostomia, oral health professionals are likely to encounter this condition more than ever before and therefore an understanding of xerostomia and its management is essential to patient care.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12031" xmlns="http://purl.org/rss/1.0/"><title>Oral conditions and their social impact among HIV dental patients, 18 years on</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12031</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Oral conditions and their social impact among HIV dental patients, 18 years on</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">SA Liberali, EA Coates, AD Freeman, RM Logan, L Jamieson, G Mejia</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T04:03:57.692826-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12031</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12031</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12031</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">ADRF Research Report</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">18</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">25</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12031-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>A study undertaken in 1992–1993 identified that HIV-infected dental patients were substantially disadvantaged with regard to the social impact of their oral disease. The oral pain experienced by HIV-positive patients prior to the introduction of combination antiretroviral therapy (cART) was attributable to specific features of HIV-related periodontal disease and other oral manifestations of HIV such as candida infections and xerostomia. A repeat of this study in 2009–2010 provided additional information in the post-cART era.</p></div></div>
<div class="section" id="adj12031-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Data were collected from three sources: the 2009–2010 HIV-positive sample, the National Survey of Adult Oral Health (NSAOH) and the original 1992–1993 study. Collation of data was by clinical and radiographic oral examination. Information about the social impact of oral conditions was obtained from the Oral Health Impact Profile.</p></div></div>
<div class="section" id="adj12031-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The caries experience of the 2009–2010 HIV-positive sample was improved with statistical significance for both mean DMFT and mean DT, while the presence of HIV-related periodontal disease still occurs. Statistically significant improvements were achieved for prevalence and severity of oral health related quality of life.</p></div></div>
<div class="section" id="adj12031-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>The need for timely access to oral health care with a focus on prevention is essential for HIV-positive individuals whose health is impacted by chronic disease, smoking and salivary hypofunction.</p></div></div>
]]></content:encoded><description>


Background
A study undertaken in 1992–1993 identified that HIV-infected dental patients were substantially disadvantaged with regard to the social impact of their oral disease. The oral pain experienced by HIV-positive patients prior to the introduction of combination antiretroviral therapy (cART) was attributable to specific features of HIV-related periodontal disease and other oral manifestations of HIV such as candida infections and xerostomia. A repeat of this study in 2009–2010 provided additional information in the post-cART era.


Methods
Data were collected from three sources: the 2009–2010 HIV-positive sample, the National Survey of Adult Oral Health (NSAOH) and the original 1992–1993 study. Collation of data was by clinical and radiographic oral examination. Information about the social impact of oral conditions was obtained from the Oral Health Impact Profile.


Results
The caries experience of the 2009–2010 HIV-positive sample was improved with statistical significance for both mean DMFT and mean DT, while the presence of HIV-related periodontal disease still occurs. Statistically significant improvements were achieved for prevalence and severity of oral health related quality of life.


Conclusions
The need for timely access to oral health care with a focus on prevention is essential for HIV-positive individuals whose health is impacted by chronic disease, smoking and salivary hypofunction.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12024" xmlns="http://purl.org/rss/1.0/"><title>The oral health status, practices and knowledge of pregnant women in south-western Sydney</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12024</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">The oral health status, practices and knowledge of pregnant women in south-western Sydney</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">A George, M Johnson, A Blinkhorn, S Ajwani, S Bhole, AE Yeo, S Ellis</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T19:56:05.969577-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12024</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12024</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12024</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">26</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">33</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12024-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Current evidence highlights the importance of oral health during pregnancy. However, little is known about the oral health of pregnant women in Australia. The aim of this study was to report the oral health status, knowledge and practices of pregnant women in south-western Sydney.</p></div></div>
<div class="section" id="adj12024-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>A cross-sectional survey of 241 pregnant women attending a large hospital in south-western Sydney.</p></div></div>
<div class="section" id="adj12024-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>More than half (59.3%) reported dental problems during pregnancy, less than a third (30.5%) saw a dentist in the last six months, only 10% had received any information about perinatal oral health and many (&gt;50%) were unaware of the potential impact of poor maternal oral health on pregnancy and infant outcomes. Analysis revealed a significant difference (&lt;0.05) in the uptake of dental services among pregnant women who had higher household incomes, private health insurance, received information about perinatal oral health and knowledge about maternal oral health.</p></div></div>
<div class="section" id="adj12024-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>The participants reported significant barriers to obtaining dental care including limited access to affordable dental services and lack of awareness about the importance of maternal oral health. The findings suggest the need for preventive strategies involving dentists and antenatal providers to improve maternal oral health in Australia.</p></div></div>
]]></content:encoded><description>


Background
Current evidence highlights the importance of oral health during pregnancy. However, little is known about the oral health of pregnant women in Australia. The aim of this study was to report the oral health status, knowledge and practices of pregnant women in south-western Sydney.


Methods
A cross-sectional survey of 241 pregnant women attending a large hospital in south-western Sydney.


Results
More than half (59.3%) reported dental problems during pregnancy, less than a third (30.5%) saw a dentist in the last six months, only 10% had received any information about perinatal oral health and many (&gt;50%) were unaware of the potential impact of poor maternal oral health on pregnancy and infant outcomes. Analysis revealed a significant difference (&lt;0.05) in the uptake of dental services among pregnant women who had higher household incomes, private health insurance, received information about perinatal oral health and knowledge about maternal oral health.


Conclusions
The participants reported significant barriers to obtaining dental care including limited access to affordable dental services and lack of awareness about the importance of maternal oral health. The findings suggest the need for preventive strategies involving dentists and antenatal providers to improve maternal oral health in Australia.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12019" xmlns="http://purl.org/rss/1.0/"><title>Local drug delivery of 0.5% azithromycin in the treatment of chronic periodontitis among smokers</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12019</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Local drug delivery of 0.5% azithromycin in the treatment of chronic periodontitis among smokers</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">AR Pradeep, P Bajaj, E Agarwal, NS Rao, SB Naik, N Kalra, N Priyanaka</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T02:42:32.623288-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12019</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12019</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12019</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">34</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">40</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12019-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Along with conventional periodontal surgical therapy, local delivery of antibiotics may provide more effective treatment in smokers by targeting tissue-invasive bacteria. The aim of this randomized, placebo-controlled, double-masked clinical trial was to evaluate the adjunctive effects of subgingivally delivered 0.5% azithromycin (AZM) as an adjunct to scaling and root planing (SRP) in the treatment of chronic periodontitis in smokers.</p></div></div>
<div class="section" id="adj12019-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Fifty-four patients were randomized and categorized into two treatment groups: Group 1 – 26 subjects who received (SRP) plus placebo gel and Group 2 – 28 subjects who received (SRP) plus 0.5% azithromycin. Clinical parameters were recorded at baseline, 3, 6 and 9 months. They included Plaque Index (PI), modified Sulcus Bleeding Index (mSBI), probing depth (PD) and clinical attachment level (CAL).</p></div></div>
<div class="section" id="adj12019-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Azithromycin resulted in significant improvements. A single application of AZM resulted in attachment gain at 9 months of 2.44 ± 0.64 mm as compared to 0.18 ± 0.68 mm for the placebo. Similarly, pocket depth and PI were significantly reduced but no change in the mSBI was noted.</p></div></div>
<div class="section" id="adj12019-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>When compared to the placebo, the adjunctive use of 0.5% AZM resulted in significant improvement in clinical outcome in the treatment of chronic periodontitis among smokers.</p></div></div>
]]></content:encoded><description>


Background
Along with conventional periodontal surgical therapy, local delivery of antibiotics may provide more effective treatment in smokers by targeting tissue-invasive bacteria. The aim of this randomized, placebo-controlled, double-masked clinical trial was to evaluate the adjunctive effects of subgingivally delivered 0.5% azithromycin (AZM) as an adjunct to scaling and root planing (SRP) in the treatment of chronic periodontitis in smokers.


Methods
Fifty-four patients were randomized and categorized into two treatment groups: Group 1 – 26 subjects who received (SRP) plus placebo gel and Group 2 – 28 subjects who received (SRP) plus 0.5% azithromycin. Clinical parameters were recorded at baseline, 3, 6 and 9 months. They included Plaque Index (PI), modified Sulcus Bleeding Index (mSBI), probing depth (PD) and clinical attachment level (CAL).


Results
Azithromycin resulted in significant improvements. A single application of AZM resulted in attachment gain at 9 months of 2.44 ± 0.64 mm as compared to 0.18 ± 0.68 mm for the placebo. Similarly, pocket depth and PI were significantly reduced but no change in the mSBI was noted.


Conclusions
When compared to the placebo, the adjunctive use of 0.5% AZM resulted in significant improvement in clinical outcome in the treatment of chronic periodontitis among smokers.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12021" xmlns="http://purl.org/rss/1.0/"><title>Comparison of periodontal open flap debridement versus closed debridement with Er,Cr:YSGG laser</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12021</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Comparison of periodontal open flap debridement versus closed debridement with Er,Cr:YSGG laser</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">M Gupta, AK Lamba, M Verma, F Faraz, S Tandon, K Chawla, DK Koli</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12021</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12021</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12021</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">41</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">49</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12021-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Traditional periodontal open flap debridement (OFD) results in reduced pocket depth (PD), clinical attachment loss (CAL), gingival recession (GR) and postoperative pain and discomfort. The quest to overcome these shortcomings has led to research into Er,Cr:YSGG laser assisted pocket therapy (ELAPT). This study was designed to compare the clinical outcomes of ELAPT versus OFD.</p></div></div>
<div class="section" id="adj12021-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Fifteen patients with a PD of ≥5 mm and ≤8 mm at two sites were selected. Test sites (Group 1) were treated by ELAPT and the control (Group 2) by OFD. Clinical parameters were recorded at baseline, 3 and 6 months and included Plaque Index (PI), Gingival Index (GI), modified Sulcular Bleeding Index (mSBI), PD, CAL and GR.</p></div></div>
<div class="section" id="adj12021-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Both treatments produced a reduction in PI, GI, mSBI and PD, an increase in GR, and a gain in CAL at 3 and 6 months. The mean gain of CAL in Group 1 at 3 and 6 months (1.60 ± 0.78 and 1.80 ± 0.63) was similar (p &gt; 0.05) to the value of Group 2 (1.93 ± 0.88 and 2.00 ± 0.54). GR increased significantly (p &lt; 0.05) only in Group 2 at 3 and 6 months (1.80 ± 0.56 and 1.87 ± 0.64) compared to Group 1 (0.50 ± 0.68 and 0.60 ± 0.74).</p></div></div>
<div class="section" id="adj12021-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>ELAPT compared with OFD results in similar CAL gains with less GR and significant reductions in PD, GI and mSBI, and may be considered as an alternative to surgical therapy.</p></div></div>
]]></content:encoded><description>


Background
Traditional periodontal open flap debridement (OFD) results in reduced pocket depth (PD), clinical attachment loss (CAL), gingival recession (GR) and postoperative pain and discomfort. The quest to overcome these shortcomings has led to research into Er,Cr:YSGG laser assisted pocket therapy (ELAPT). This study was designed to compare the clinical outcomes of ELAPT versus OFD.


Methods
Fifteen patients with a PD of ≥5 mm and ≤8 mm at two sites were selected. Test sites (Group 1) were treated by ELAPT and the control (Group 2) by OFD. Clinical parameters were recorded at baseline, 3 and 6 months and included Plaque Index (PI), Gingival Index (GI), modified Sulcular Bleeding Index (mSBI), PD, CAL and GR.


Results
Both treatments produced a reduction in PI, GI, mSBI and PD, an increase in GR, and a gain in CAL at 3 and 6 months. The mean gain of CAL in Group 1 at 3 and 6 months (1.60 ± 0.78 and 1.80 ± 0.63) was similar (p &gt; 0.05) to the value of Group 2 (1.93 ± 0.88 and 2.00 ± 0.54). GR increased significantly (p &lt; 0.05) only in Group 2 at 3 and 6 months (1.80 ± 0.56 and 1.87 ± 0.64) compared to Group 1 (0.50 ± 0.68 and 0.60 ± 0.74).


Conclusions
ELAPT compared with OFD results in similar CAL gains with less GR and significant reductions in PD, GI and mSBI, and may be considered as an alternative to surgical therapy.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12033" xmlns="http://purl.org/rss/1.0/"><title>An in vitro study of silver and fluoride ions on remineralization of demineralized enamel and dentine</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12033</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">An in vitro study of silver and fluoride ions on remineralization of demineralized enamel and dentine</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">QH Zhi, ECM Lo, ACY Kwok</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T04:04:01.509353-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12033</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12033</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12033</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">50</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">56</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12033-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The purpose of this study was to compare the effect of silver fluoride, silver nitrate and potassium fluoride on remineralization of demineralized enamel and dentine <em>in vitro</em>.</p></div></div>
<div class="section" id="adj12033-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Forty premolars were cut into cuboidal blocks. Acid-resistant varnish was painted onto each block to cover all surfaces, except two windows, one in enamel and one in dentine. The tooth blocks were placed in demineralizing solution for 96 hours. They were then randomly divided into four groups of 10 blocks each and immersed in solutions of AgF, AgNO<sub>3</sub>, KF or water for 3 minutes. Afterwards, they were immersed in a remineralizing solution for 108 hours. Micro CT scanning was conducted before and after remineralization.</p></div></div>
<div class="section" id="adj12033-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The increase in linear attentuation coefficient (LAC) for the enamel lesions after remineralization was 1.08/cm, 0.95/cm, 0.86/cm and 0.60/cm in the AgF, AgNO<sub>3</sub>, KF and control groups, respectively (ANOVA, p &lt; 0.001; AgF, AgNO<sub>3</sub>, KF &gt; control; AgF &gt; KF). The increase in LAC for the dentine lesions was 1.01/cm, 0.92/cm, 0.88/cm and 0.53/cm, respectively (ANOVA, p &lt; 0.001; AgF, AgNO<sub>3</sub>, KF &gt; control).</p></div></div>
<div class="section" id="adj12033-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Topical application of silver or fluoride ions can increase the mineral density of demineralized enamel and dentine lesions during remineralization. The synergistic effect of silver and fluoride ions is relatively small.</p></div></div>
]]></content:encoded><description>


Background
The purpose of this study was to compare the effect of silver fluoride, silver nitrate and potassium fluoride on remineralization of demineralized enamel and dentine in vitro.


Methods
Forty premolars were cut into cuboidal blocks. Acid-resistant varnish was painted onto each block to cover all surfaces, except two windows, one in enamel and one in dentine. The tooth blocks were placed in demineralizing solution for 96 hours. They were then randomly divided into four groups of 10 blocks each and immersed in solutions of AgF, AgNO3, KF or water for 3 minutes. Afterwards, they were immersed in a remineralizing solution for 108 hours. Micro CT scanning was conducted before and after remineralization.


Results
The increase in linear attentuation coefficient (LAC) for the enamel lesions after remineralization was 1.08/cm, 0.95/cm, 0.86/cm and 0.60/cm in the AgF, AgNO3, KF and control groups, respectively (ANOVA, p &lt; 0.001; AgF, AgNO3, KF &gt; control; AgF &gt; KF). The increase in LAC for the dentine lesions was 1.01/cm, 0.92/cm, 0.88/cm and 0.53/cm, respectively (ANOVA, p &lt; 0.001; AgF, AgNO3, KF &gt; control).


Conclusions
Topical application of silver or fluoride ions can increase the mineral density of demineralized enamel and dentine lesions during remineralization. The synergistic effect of silver and fluoride ions is relatively small.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12032" xmlns="http://purl.org/rss/1.0/"><title>Healing properties of implants inserted concomitantly with anorganic bovine bone. A histomorphometric human study</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12032</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Healing properties of implants inserted concomitantly with anorganic bovine bone. A histomorphometric human study</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">G Menicucci, F Mussano, G Schierano, A Rizzati, M Aimetti, G Gassino, T Traini, S Carossa</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-05T00:43:51.318197-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12032</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12032</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12032</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">57</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">66</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12032-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The present prospective, randomized, double-blind study evaluated the bone-forming process around implants inserted simultaneously with anorganic bovine bone (ABB) in sinus grafting.</p></div></div>
<div class="section" id="adj12032-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>A total of 18 threaded mini-implants with Osseotite (<em>O</em>) and Nanotite <em>(N</em>) surfaces were placed in seven patients (nine sites). After 12 months, the implants were retrieved and processed for histological analysis. A total of 18 cutting and grinding sections were investigated with bright-field light microscopy, circularly polarized light microscopy (CPLM), confocal scanning laser microscope (CSLM), and scanning electron microscope (SEM) with energy dispersive spectrometer (EDS).</p></div></div>
<div class="section" id="adj12032-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The bone-to-implant contact rate in native crestal bone was 62.6 ± 0.4% for <em>N</em> implants and 54.3 ± 0.5% for the <em>O</em> implants (p = 0.001). The collagen fibre density, as assessed by CPLM, was 79.8 ± 6.0 nm for the <em>N</em> group and 74.6 ± 4.6 nm for the <em>O</em> group (p &lt; 0.05). Line scan EDS starting from ABB to newly formed bone showed a decrease in calcium content and an increase of carbon while phosphorus content was constant.</p></div></div>
<div class="section" id="adj12032-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>While the <em>N</em> surface improved the peri-implant endosseous healing properties in the native bone, when compared to the <em>O</em> surface, it did not improve the healing properties in the bone-graft area.</p></div></div>
]]></content:encoded><description>


Background
The present prospective, randomized, double-blind study evaluated the bone-forming process around implants inserted simultaneously with anorganic bovine bone (ABB) in sinus grafting.


Methods
A total of 18 threaded mini-implants with Osseotite (O) and Nanotite (N) surfaces were placed in seven patients (nine sites). After 12 months, the implants were retrieved and processed for histological analysis. A total of 18 cutting and grinding sections were investigated with bright-field light microscopy, circularly polarized light microscopy (CPLM), confocal scanning laser microscope (CSLM), and scanning electron microscope (SEM) with energy dispersive spectrometer (EDS).


Results
The bone-to-implant contact rate in native crestal bone was 62.6 ± 0.4% for N implants and 54.3 ± 0.5% for the O implants (p = 0.001). The collagen fibre density, as assessed by CPLM, was 79.8 ± 6.0 nm for the N group and 74.6 ± 4.6 nm for the O group (p &lt; 0.05). Line scan EDS starting from ABB to newly formed bone showed a decrease in calcium content and an increase of carbon while phosphorus content was constant.


Conclusions
While the N surface improved the peri-implant endosseous healing properties in the native bone, when compared to the O surface, it did not improve the healing properties in the bone-graft area.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12018" xmlns="http://purl.org/rss/1.0/"><title>Age-related changes in maxillary and mandibular cortical bone thickness in relation to temporary anchorage device placement</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12018</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Age-related changes in maxillary and mandibular cortical bone thickness in relation to temporary anchorage device placement</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">S Sathapana, A Forrest, P Monsour, S Naser-ud-Din</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12018</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12018</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12018</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">67</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">74</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12018-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The aim of this study was to investigate the correlation between alveolar bone cortical thickness (ABCT) and age in the maxillae and mandible in humans. This information could then be translated into clinical application with temporary anchorage devices (TADs) in orthdontics.</p></div></div>
<div class="section" id="adj12018-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Samples comprised 82 post-mortem CT datasets (41 males and 41 females) aged between 11 to 50 years, and were divided into five different age groups and gender. Alveolar ABCT was measured in the labial/buccal and palatal/lingual sides of the incisor, canine, premolar, molar and tuberosity/retromolar regions of the maxillae and mandible. Correlations between ABCT and age and gender were analysed with linear regression analysis.</p></div></div>
<div class="section" id="adj12018-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Strong correlation between ABCT and age was found for the maxilla on the labial side of the maxillary incisor region (p &lt; 0.001). On the palatal aspect, significant correlations between ABCT and age were found in the maxillary incisor and maxillary premolar regions (p = 0.01 and p = 0.047 respectively). Significant correlation between ABCT and gender was found only at the buccal aspect of the maxillary molar region (p = 0.022). In the mandible, a statistically significant correlation between ABCT and age was found in the cortical bone of the labial side of the mandibular incisor region (p = 0.017). However, statistically significant negative correlation between ABCT and age was found in the mandibular canine region (p = 0.033). The only site to demonstrate a significant difference in change in ABCT with age between males and females was the lingual side of the retromolar region, in which female ABCT increased more than in males (slope = 0.015).</p></div></div>
<div class="section" id="adj12018-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>There is minimal clinically significant correlation between ABCT and age at the alveolar bone level. Although investigations show statistically significant correlations, these may not be clinically significant as those regions are not ideal for anchorage reinforcement with TADs in orthodontic practice.</p></div></div>
]]></content:encoded><description>


Background
The aim of this study was to investigate the correlation between alveolar bone cortical thickness (ABCT) and age in the maxillae and mandible in humans. This information could then be translated into clinical application with temporary anchorage devices (TADs) in orthdontics.


Methods
Samples comprised 82 post-mortem CT datasets (41 males and 41 females) aged between 11 to 50 years, and were divided into five different age groups and gender. Alveolar ABCT was measured in the labial/buccal and palatal/lingual sides of the incisor, canine, premolar, molar and tuberosity/retromolar regions of the maxillae and mandible. Correlations between ABCT and age and gender were analysed with linear regression analysis.


Results
Strong correlation between ABCT and age was found for the maxilla on the labial side of the maxillary incisor region (p &lt; 0.001). On the palatal aspect, significant correlations between ABCT and age were found in the maxillary incisor and maxillary premolar regions (p = 0.01 and p = 0.047 respectively). Significant correlation between ABCT and gender was found only at the buccal aspect of the maxillary molar region (p = 0.022). In the mandible, a statistically significant correlation between ABCT and age was found in the cortical bone of the labial side of the mandibular incisor region (p = 0.017). However, statistically significant negative correlation between ABCT and age was found in the mandibular canine region (p = 0.033). The only site to demonstrate a significant difference in change in ABCT with age between males and females was the lingual side of the retromolar region, in which female ABCT increased more than in males (slope = 0.015).


Conclusions
There is minimal clinically significant correlation between ABCT and age at the alveolar bone level. Although investigations show statistically significant correlations, these may not be clinically significant as those regions are not ideal for anchorage reinforcement with TADs in orthodontic practice.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12017" xmlns="http://purl.org/rss/1.0/"><title>Associations between area-level disadvantage and DMFT among a birth cohort of Indigenous Australians</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12017</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Associations between area-level disadvantage and DMFT among a birth cohort of Indigenous Australians</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">LM Jamieson, LG Do, RS Bailie, SM Sayers, G Turrell</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T04:05:00.193339-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12017</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12017</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12017</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">75</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">81</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12017-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>Individual-level factors influence DMFT, but little is known about the influence of community environment. This study examined associations between community-level influences and DMFT among a birth cohort of Indigenous Australians aged 16–20 years.</p></div></div>
<div class="section" id="adj12017-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Data were collected as part of Wave 3 of the Aboriginal Birth Cohort study. Fifteen community areas were established and the sample comprised 442 individuals. The outcome variable was mean DMFT with explanatory variables including diet and community disadvantage (access to services, infrastructure and communications). Data were analysed using multilevel regression modelling.</p></div></div>
<div class="section" id="adj12017-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>In a null model, 13.8% of the total variance in mean DMFT was between community areas, which increased to 14.3% after adjusting for gender, age and diet. Addition of the community disadvantage variable decreased the variance between areas by 4.8%, indicating that community disadvantage explained one-third of the area-level variance. Residents of under-resourced communities had significantly higher mean DMFT (β  =  3.86, 95% CI 0.02, 7.70) after adjusting for gender, age and diet.</p></div></div>
<div class="section" id="adj12017-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Living in under-resourced communities was associated with greater DMFT among this disadvantaged population, indicating that policies aiming to reduce oral health-related inequalities among vulnerable groups may benefit from taking into account factors external to individual-level influences.</p></div></div>
]]></content:encoded><description>


Background
Individual-level factors influence DMFT, but little is known about the influence of community environment. This study examined associations between community-level influences and DMFT among a birth cohort of Indigenous Australians aged 16–20 years.


Methods
Data were collected as part of Wave 3 of the Aboriginal Birth Cohort study. Fifteen community areas were established and the sample comprised 442 individuals. The outcome variable was mean DMFT with explanatory variables including diet and community disadvantage (access to services, infrastructure and communications). Data were analysed using multilevel regression modelling.


Results
In a null model, 13.8% of the total variance in mean DMFT was between community areas, which increased to 14.3% after adjusting for gender, age and diet. Addition of the community disadvantage variable decreased the variance between areas by 4.8%, indicating that community disadvantage explained one-third of the area-level variance. Residents of under-resourced communities had significantly higher mean DMFT (β  =  3.86, 95% CI 0.02, 7.70) after adjusting for gender, age and diet.


Conclusions
Living in under-resourced communities was associated with greater DMFT among this disadvantaged population, indicating that policies aiming to reduce oral health-related inequalities among vulnerable groups may benefit from taking into account factors external to individual-level influences.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12030" xmlns="http://purl.org/rss/1.0/"><title>Stress analysis of different post-luting systems: a three-dimensional finite element analysis</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12030</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Stress analysis of different post-luting systems: a three-dimensional finite element analysis</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">SA Romeed, SM Dunne</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T04:03:54.129695-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12030</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12030</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12030</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">82</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">88</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12030-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The longevity of endodontically treated teeth is usually determined by the adequacy of root canal treatments, coronal seal and favourable stress distribution within the remaining tooth tissues. The aim of this study was to investigate the influence of post material and luting cement on the biomechanics of endodontically treated teeth using three-dimensional finite element analysis (3-D FEA).</p></div></div>
<div class="section" id="adj12030-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>A 3 mm section of endodontically treated canine tooth was scanned and reconstructed for 3-D modelling and FE analyses. A metal post (MP) and a glass fibre post (GFP) were tested individually with four luting cements [zinc phosphate (ZPH), glass ionomer (GI), resin modified glass ionomer (RMGI) and resin based cements (RC)]. A push-out test was conducted by subjecting all models to 100 N perpendicular loading at the post.</p></div></div>
<div class="section" id="adj12030-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The maximum stresses generated along the MP-cement interface were significantly higher than corresponding stresses in the GFP-cement interface regardless of the cement type. GFP generated seven times higher stresses within the root dentine than metal posts when ZPH and GI were used, and three times higher when RMGI and RC were used. The displacement of GFP was double (50 μ) the displacement of MP (20 μ) in all groups.</p></div></div>
<div class="section" id="adj12030-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>The low elastic modulus of GFP generated lower stresses along its interface and higher stresses within the root dentine, therefore the probability of debonding and root fracture in the GFP group was lower.</p></div></div>
]]></content:encoded><description>


Background
The longevity of endodontically treated teeth is usually determined by the adequacy of root canal treatments, coronal seal and favourable stress distribution within the remaining tooth tissues. The aim of this study was to investigate the influence of post material and luting cement on the biomechanics of endodontically treated teeth using three-dimensional finite element analysis (3-D FEA).


Methods
A 3 mm section of endodontically treated canine tooth was scanned and reconstructed for 3-D modelling and FE analyses. A metal post (MP) and a glass fibre post (GFP) were tested individually with four luting cements [zinc phosphate (ZPH), glass ionomer (GI), resin modified glass ionomer (RMGI) and resin based cements (RC)]. A push-out test was conducted by subjecting all models to 100 N perpendicular loading at the post.


Results
The maximum stresses generated along the MP-cement interface were significantly higher than corresponding stresses in the GFP-cement interface regardless of the cement type. GFP generated seven times higher stresses within the root dentine than metal posts when ZPH and GI were used, and three times higher when RMGI and RC were used. The displacement of GFP was double (50 μ) the displacement of MP (20 μ) in all groups.


Conclusions
The low elastic modulus of GFP generated lower stresses along its interface and higher stresses within the root dentine, therefore the probability of debonding and root fracture in the GFP group was lower.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12029" xmlns="http://purl.org/rss/1.0/"><title>The healing of dental extraction sockets in patients with Type 2 diabetes on oral hypoglycaemics: a prospective cohort</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12029</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">The healing of dental extraction sockets in patients with Type 2 diabetes on oral hypoglycaemics: a prospective cohort</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">S Huang, H Dang, W Huynh, PJ Sambrook, AN Goss</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-28T03:20:29.845785-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12029</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12029</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12029</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">89</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">93</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12029-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The aim of this study was to determine whether there is a difference in delayed healing following dental extractions for Type 2 diabetics on oral hypoglycaemics and non-diabetic patients.</p></div></div>
<div class="section" id="adj12029-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Prospective patients referred for dental extractions were recruited into two groups: known diabetics and non-diabetics with no conditions associated with poor healing. All had a random blood glucose level (BGL). Extractions were performed using local anaesthesia. Delayed healing cases were identified and statistical evaluation performed to identify risk factors.</p></div></div>
<div class="section" id="adj12029-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>There were 224 Type 2 diabetics on oral hypoglycaemics (BGL 7.51, range 4.1–17.4) and 232 non-diabetics. The diabetic group were older, more males and less smokers than the control group. Twenty-eight patients, 12 (5%) diabetic and 16 (7%) control group, had socket healing delayed for more than one week but all healed in four weeks. There were no statistical differences between delayed healing and age, gender, diabetic state, BGL or smoking. The younger control group had more healing problems.</p></div></div>
<div class="section" id="adj12029-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>The traditional view that diabetics have increased delayed healing was not supported. Type 2 diabetics on oral hypoglycaemics should be treated the same as non-diabetic patients for extractions.</p></div></div>
]]></content:encoded><description>


Background
The aim of this study was to determine whether there is a difference in delayed healing following dental extractions for Type 2 diabetics on oral hypoglycaemics and non-diabetic patients.


Methods
Prospective patients referred for dental extractions were recruited into two groups: known diabetics and non-diabetics with no conditions associated with poor healing. All had a random blood glucose level (BGL). Extractions were performed using local anaesthesia. Delayed healing cases were identified and statistical evaluation performed to identify risk factors.


Results
There were 224 Type 2 diabetics on oral hypoglycaemics (BGL 7.51, range 4.1–17.4) and 232 non-diabetics. The diabetic group were older, more males and less smokers than the control group. Twenty-eight patients, 12 (5%) diabetic and 16 (7%) control group, had socket healing delayed for more than one week but all healed in four weeks. There were no statistical differences between delayed healing and age, gender, diabetic state, BGL or smoking. The younger control group had more healing problems.


Conclusions
The traditional view that diabetics have increased delayed healing was not supported. Type 2 diabetics on oral hypoglycaemics should be treated the same as non-diabetic patients for extractions.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12028" xmlns="http://purl.org/rss/1.0/"><title>Clinical evaluation of different adhesives used in the restoration of non-carious cervical lesions: 24-month results</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12028</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Clinical evaluation of different adhesives used in the restoration of non-carious cervical lesions: 24-month results</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">D Tuncer, AR Yazici, G Özgünaltay, B Dayangac</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-31T00:12:26.114759-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12028</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12028</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12028</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">94</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">100</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12028-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>The aim of this study was to compare after 24 months the clinical performance of cervical restorations placed with the use of an etch-and-rinse and an all-in-one adhesive.</p></div></div>
<div class="section" id="adj12028-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Twenty-four patients with at least one pair of non-carious cervical lesions participated in this study. One hundred and twenty-three cervical lesions were restored (62 etch-and-rinse adhesive, Solobond M; 61 all-in-one adhesive, Futurabond NR). A nanohybrid resin composite, Grandio, was used as the restorative material. Restorations were evaluated according to modified USPHS criteria by two independent examiners. The survival rates of the restorations were calculated by the Kaplan-Meier estimator and log-rank test. The restorations in each category were compared using the Pearson chi-square test, while the performance of restorations at the baseline and at each recall time was evaluated by McNemar's test (p &lt; 0.05).</p></div></div>
<div class="section" id="adj12028-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>The recall rate of patients was 100%. The retention rates were 82% and 75% at 6 months and 77% and 62% at 12 months for Solobond M and Futurabond NR, respectively. At the 24-month recall, the retention rate was 69% for Solobond M and 49% for Futurabond NR, and this difference was statistically significant (p &lt; 0.05). No statistically significant difference was found for colour match, marginal staining, or marginal adaptation between the adhesives (p &gt; 0.05). None of the restorations had secondary caries, loss of anatomical form, or surface texture changes.</p></div></div>
<div class="section" id="adj12028-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Cervical restorations placed with an etch-and-rinse adhesive showed higher retention than an all-in-one adhesive.</p></div></div>
]]></content:encoded><description>


Background
The aim of this study was to compare after 24 months the clinical performance of cervical restorations placed with the use of an etch-and-rinse and an all-in-one adhesive.


Methods
Twenty-four patients with at least one pair of non-carious cervical lesions participated in this study. One hundred and twenty-three cervical lesions were restored (62 etch-and-rinse adhesive, Solobond M; 61 all-in-one adhesive, Futurabond NR). A nanohybrid resin composite, Grandio, was used as the restorative material. Restorations were evaluated according to modified USPHS criteria by two independent examiners. The survival rates of the restorations were calculated by the Kaplan-Meier estimator and log-rank test. The restorations in each category were compared using the Pearson chi-square test, while the performance of restorations at the baseline and at each recall time was evaluated by McNemar's test (p &lt; 0.05).


Results
The recall rate of patients was 100%. The retention rates were 82% and 75% at 6 months and 77% and 62% at 12 months for Solobond M and Futurabond NR, respectively. At the 24-month recall, the retention rate was 69% for Solobond M and 49% for Futurabond NR, and this difference was statistically significant (p &lt; 0.05). No statistically significant difference was found for colour match, marginal staining, or marginal adaptation between the adhesives (p &gt; 0.05). None of the restorations had secondary caries, loss of anatomical form, or surface texture changes.


Conclusions
Cervical restorations placed with an etch-and-rinse adhesive showed higher retention than an all-in-one adhesive.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12027" xmlns="http://purl.org/rss/1.0/"><title>Adhesion of Streptococci to various orthodontic composite resins</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12027</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Adhesion of Streptococci to various orthodontic composite resins</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">U Velazquez-Enriquez, RJ Scougall-Vilchis, R Contreras-Bulnes, J Flores-Estrada,  S Uematsu, R Yamaguchi</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-05T04:25:17.71447-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12027</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12027</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12027</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">101</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">105</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12027-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>This investigation aimed to determine quantitatively the adhesion of <em>Streptococcus mutans</em> and <em>Streptococcus sobrinus</em> to orthodontic composite resins that were tested simultaneously using radio-markers.</p></div></div>
<div class="section" id="adj12027-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Seven orthodontic composite resins were classified into seven groups: BeautyOrtho Bond (GI), Blugloo (GII), Enlight (GIII), Grengloo (GIV), Kurasper F (GV), Transbond CC (GVI) and Turbo Bond II (GVII). Thirty 4 x 4 x 1 mm blocks of each orthodontic composite resin were made (a total of 210 blocks). Both <em>Streptococcus</em> species were cultivated independently. For the quantitative analysis, radioactive markers were used to codify the bacteria (<sup>3</sup>H for <em>Streptococcus mutans</em> and <sup>14</sup>C for <em>Streptococcus sobrinus</em>). The blocks were submerged in a solution with microorganisms previously radiolabelled for 2 hours at 37 °C in constant movement. The blocks were placed in a combustion system to quantify the <em>Streptococcus</em> adhering to the surface of the materials by capturing the residues and measuring the radiation.</p></div></div>
<div class="section" id="adj12027-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>Significant differences in bacterial adhesion were found among the groups. The lowest significant scores for both microorganisms were observed in GIII.</p></div></div>
<div class="section" id="adj12027-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>The orthodontic composite resin evaluated in GIII exhibited the lowest adhesion of <em>Streptococcus mutans</em> and <em>Streptococcus sobrinus</em>, which may reduce enamel demineralization and the risk of white spot lesion formation.</p></div></div>
]]></content:encoded><description>


Background
This investigation aimed to determine quantitatively the adhesion of Streptococcus mutans and Streptococcus sobrinus to orthodontic composite resins that were tested simultaneously using radio-markers.


Methods
Seven orthodontic composite resins were classified into seven groups: BeautyOrtho Bond (GI), Blugloo (GII), Enlight (GIII), Grengloo (GIV), Kurasper F (GV), Transbond CC (GVI) and Turbo Bond II (GVII). Thirty 4 x 4 x 1 mm blocks of each orthodontic composite resin were made (a total of 210 blocks). Both Streptococcus species were cultivated independently. For the quantitative analysis, radioactive markers were used to codify the bacteria (3H for Streptococcus mutans and 14C for Streptococcus sobrinus). The blocks were submerged in a solution with microorganisms previously radiolabelled for 2 hours at 37 °C in constant movement. The blocks were placed in a combustion system to quantify the Streptococcus adhering to the surface of the materials by capturing the residues and measuring the radiation.


Results
Significant differences in bacterial adhesion were found among the groups. The lowest significant scores for both microorganisms were observed in GIII.


Conclusions
The orthodontic composite resin evaluated in GIII exhibited the lowest adhesion of Streptococcus mutans and Streptococcus sobrinus, which may reduce enamel demineralization and the risk of white spot lesion formation.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12016" xmlns="http://purl.org/rss/1.0/"><title>Impact of different fluoride concentrations and pH of dentifrices on tooth erosion/abrasion in vitro</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12016</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Impact of different fluoride concentrations and pH of dentifrices on tooth erosion/abrasion in vitro</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">BM Moron, SSH Miyazaki, N Ito, A Wiegand, F Vilhena, MAR Buzalaf, AC Magalhães</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-01-30T02:42:37.26905-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12016</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12016</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12016</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Scientific Article</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">106</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">111</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="section" id="adj12016-sec-0001" xmlns="http://www.w3.org/1999/xhtml"><h4>Background</h4><div class="para"><p>This <em>in vitro</em> study analysed the effect of different fluoride concentrations in acidic or neutral liquid dentifrices in protecting enamel and dentine from erosive and abrasive wear.</p></div></div>
<div class="section" id="adj12016-sec-0002" xmlns="http://www.w3.org/1999/xhtml"><h4>Methods</h4><div class="para"><p>Bovine enamel and dentine specimens (n = 132) were randomly allocated to 11 groups (each n = 12): experimental liquid dentifrices with 550 ppm F, 1100 ppm F, 5000 ppm F or 0 ppm F/placebo (each at pH 4.5 and pH 7.0); and commercial dentifrices with 550 ppm F (Colgate Baby, pH 7.0), 1100 ppm F (Crest, pH 7.0) and 5000 ppm F (Duraphat, pH 7.0). The specimens were subjected to erosion for 90 seconds, 4 times/day, over 7 days. Immediately after the first and last erosion, the specimens were brushed for 15 seconds using one of the dentifrices. Tooth wear was measured profilometrically (μm) and analysed by ANOVA (p &lt; 0.05).</p></div></div>
<div class="section" id="adj12016-sec-0003" xmlns="http://www.w3.org/1999/xhtml"><h4>Results</h4><div class="para"><p>All fluoridated liquid dentifrices significantly reduced enamel wear compared to the placebo and commercial dentifrices. Only liquid dentifrices with 1100 and 5000 ppm F significantly reduced dentine wear compared to placebo dentifrice. The pH had no effect, but the consistency had a significant impact on the effect of dentifrices.</p></div></div>
<div class="section" id="adj12016-sec-0004" xmlns="http://www.w3.org/1999/xhtml"><h4>Conclusions</h4><div class="para"><p>Liquid dentifrices with high F concentration appear to be a good option to prevent tooth wear.</p></div></div>
]]></content:encoded><description>


Background
This in vitro study analysed the effect of different fluoride concentrations in acidic or neutral liquid dentifrices in protecting enamel and dentine from erosive and abrasive wear.


Methods
Bovine enamel and dentine specimens (n = 132) were randomly allocated to 11 groups (each n = 12): experimental liquid dentifrices with 550 ppm F, 1100 ppm F, 5000 ppm F or 0 ppm F/placebo (each at pH 4.5 and pH 7.0); and commercial dentifrices with 550 ppm F (Colgate Baby, pH 7.0), 1100 ppm F (Crest, pH 7.0) and 5000 ppm F (Duraphat, pH 7.0). The specimens were subjected to erosion for 90 seconds, 4 times/day, over 7 days. Immediately after the first and last erosion, the specimens were brushed for 15 seconds using one of the dentifrices. Tooth wear was measured profilometrically (μm) and analysed by ANOVA (p &lt; 0.05).


Results
All fluoridated liquid dentifrices significantly reduced enamel wear compared to the placebo and commercial dentifrices. Only liquid dentifrices with 1100 and 5000 ppm F significantly reduced dentine wear compared to placebo dentifrice. The pH had no effect, but the consistency had a significant impact on the effect of dentifrices.


Conclusions
Liquid dentifrices with high F concentration appear to be a good option to prevent tooth wear.

</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12026" xmlns="http://purl.org/rss/1.0/"><title>The management of benign salivary disease: a case series</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12026</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">The management of benign salivary disease: a case series</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">RHB Jones, GJ Findlay</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-03T22:00:19.662404-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12026</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12026</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12026</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Case Report</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">112</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">116</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>There are many causes for benign salivary gland disease but the most common relate to inflammation and infection. This usually revolves around duct obstruction and a reduction in the normal salivary flow from the gland into the mouth. This leads to retention of saliva, proximal to the obstruction and ascending infection from the mouth, usually because of the decrease in salivary flow. The increase in tension behind the obstruction causes significant pain and swelling, along with the inevitable infection if the obstruction is not relieved. This paper discusses the various treatments available for benign salivary gland disease, the traditional methods of treatment through to the use of endoscopic techniques which are currently available, including a discussion about the use of sialoendoscopy.</p></div>
]]></content:encoded><description>

There are many causes for benign salivary gland disease but the most common relate to inflammation and infection. This usually revolves around duct obstruction and a reduction in the normal salivary flow from the gland into the mouth. This leads to retention of saliva, proximal to the obstruction and ascending infection from the mouth, usually because of the decrease in salivary flow. The increase in tension behind the obstruction causes significant pain and swelling, along with the inevitable infection if the obstruction is not relieved. This paper discusses the various treatments available for benign salivary gland disease, the traditional methods of treatment through to the use of endoscopic techniques which are currently available, including a discussion about the use of sialoendoscopy.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12025" xmlns="http://purl.org/rss/1.0/"><title>Acute fetal distress following tooth extraction and abscess drainage in a pregnant patient with maxillofacial infection</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12025</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Acute fetal distress following tooth extraction and abscess drainage in a pregnant patient with maxillofacial infection</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/">N Çelebi, MS Kütük, M Taş, E Soylu, OA Etöz, A Alkan</dc:creator><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12025</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12025</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12025</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Case Report</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">117</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">119</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[
<h3 xhtml="http://www.w3.org/1999/xhtml" xmlns:ol="http://www.wiley.com/namespaces/ol/xsl-lib">Abstract</h3>
<div class="para" xmlns="http://www.w3.org/1999/xhtml"><p>Oral infections have been implicated in adverse pregnancy outcomes such as pre-eclampsia, premature delivery and growth retardation. A 28-year-old and 9 months pregnant otherwise healthy woman with a complaint of facial swelling and dental pain was referred to the Department of Oral and Maxillofacial Surgery. Oral examination revealed perimandibular and masticator space infection related to the left mandibular third molar tooth. Eight hours after surgical intervention, fetal distress developed. The patient was immediately taken into surgery and a male baby delivered by Caesarean section. The baby was then admitted to the intensive care unit. On the twelfth day of his admission, the baby was discharged in good health. Severe maxillofacial infection in pregnancy is a medically complicated situation which should be treated by an oral and maxillofacial surgeon in consultation with an obstetric and gynaecology service.</p></div>
]]></content:encoded><description>

Oral infections have been implicated in adverse pregnancy outcomes such as pre-eclampsia, premature delivery and growth retardation. A 28-year-old and 9 months pregnant otherwise healthy woman with a complaint of facial swelling and dental pain was referred to the Department of Oral and Maxillofacial Surgery. Oral examination revealed perimandibular and masticator space infection related to the left mandibular third molar tooth. Eight hours after surgical intervention, fetal distress developed. The patient was immediately taken into surgery and a male baby delivered by Caesarean section. The baby was then admitted to the intensive care unit. On the twelfth day of his admission, the baby was discharged in good health. Severe maxillofacial infection in pregnancy is a medically complicated situation which should be treated by an oral and maxillofacial surgeon in consultation with an obstetric and gynaecology service.
</description></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12034" xmlns="http://purl.org/rss/1.0/"><title>Oral health and other characteristics of pregnant Aboriginal women compared with general population estimates</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12034</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Oral health and other characteristics of pregnant Aboriginal women compared with general population estimates</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12034</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12034</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12034</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Data Watch</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">120</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">124</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[]]></content:encoded><description/></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12035" xmlns="http://purl.org/rss/1.0/"><title>Human papillomavirus and oral disease – emerging evidence: a review</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12035</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Human papillomavirus and oral disease – emerging evidence: a review</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12035</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12035</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12035</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Continuing Professional Development Quiz</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">125</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">125</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[]]></content:encoded><description/></item><item rdf:about="http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12036" xmlns="http://purl.org/rss/1.0/"><title>Continuing Professional Development Calendar</title><link>http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12036</link><dc:title xmlns:dc="http://purl.org/dc/elements/1.1/">Continuing Professional Development Calendar</dc:title><dc:creator xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:date xmlns:dc="http://purl.org/dc/elements/1.1/">2013-02-27T04:52:14.921378-05:00</dc:date><dc:identifier xmlns:dc="http://purl.org/dc/elements/1.1/">doi:10.1111/adj.12036</dc:identifier><dc:rights xmlns:dc="http://purl.org/dc/elements/1.1/"/><dc:publisher xmlns:dc="http://purl.org/dc/elements/1.1/">John Wiley &amp; Sons, Inc.</dc:publisher><prism:doi xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">10.1111/adj.12036</prism:doi><prism:url xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fadj.12036</prism:url><prism:section xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">Continuing Professional Development Calendar</prism:section><prism:startingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">126</prism:startingPage><prism:endingPage xmlns:prism="http://prismstandard.org/namespaces/1.2/basic/">129</prism:endingPage><content:encoded xmlns:content="http://purl.org/rss/1.0/modules/content/"><![CDATA[]]></content:encoded><description/></item></rdf:RDF>